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  • Electronic Resource  (2)
  • 1995-1999  (2)
  • 1955-1959
  • 1998  (2)
  • Key Words: Neutrophils—Apoptosis—Macrophages—Ozone—TUNEL.  (1)
  • PACS: 61.48.+c; 68.35.-p; 68.35.Bs  (1)
  • 1
    Electronic Resource
    Electronic Resource
    AFM observation of surface reconstruction of C60 single crystals (1998)
    Springer
    Applied physics 66 (1998), S. S763 
    Details
    ISSN: 1432-0630
    Keywords: PACS: 61.48.+c; 68.35.-p; 68.35.Bs
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Notes: 60 single crystals. The (111) and the (001) faces of C60 single crystals were illuminated with photons with energies below the band gap (generation of Frenkel-type excitons) or above the band gap. In the case of sub-band-gap illumination, reconstructed surfaces formed on the crystals. The reconstruction was represented by fringes running in the [11 ] and [0 0] directions on the (111) and (001) surfaces, respectively. As a result of supra-band-gap illumination, however, there appeared only cracks, without any fringes, which were oriented in directions characteristic of each face. The results suggest that the photo-induced surface reconstruction of C60 single crystals is induced via the recombination of Frenkel-type excitons.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003390051238
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  • 2
    Electronic Resource
    Electronic Resource
    Elimination of Neutrophils by Apoptosis During the Resolution of Acute Pulmonary Inflammation in Rats (1998)
    Springer
    Lung 176 (1998), S. 89-98 
    Details
    ISSN: 1432-1750
    Keywords: Key Words: Neutrophils—Apoptosis—Macrophages—Ozone—TUNEL.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract. We evaluated the apoptosis of neutrophils during the resolution of acute pulmonary inflammation induced by exposure to ozone. The inflammatory response was assessed in rat lungs 0, 1, 3, and 7 days after 4-h exposure to air or 2 ppm ozone. Analysis of bronchoalveolar lavage fluid demonstrated significant increases in albumin concentrations on days 0 and 1 and in the number of lavageable neutrophils on days 0, 1, and 3, indicating the presence of acute pulmonary inflammation. These parameters returned to control values on day 7, which suggests that the acute pulmonary inflammation induced by ozone was reversible. On days 1 and 3, but not on day 0, the neutrophils showed morphologic evidence of apoptosis. Based on morphologic analysis, the proportion of apoptotic neutrophils was 23.3 ± 2.2% on day 1 and 55.7 ± 3.2% on day 3. Terminal deoxynucleotidyl transferase-mediated dUTP end labeling (TUNEL), in contrast, revealed that the proportion of apoptotic cells was 59.7 ± 9.1% on day 1 and 68.0 ± 4.3% on day 3. On day 3, light microscopy and electron microscopy demonstrated engulfment of the neutrophils by macrophages. These findings indicate that the apoptosis of neutrophils followed by their engulfment by macrophages contributes to the clearance of neutrophils from the sites of inflammation. Moreover, TUNEL detected apoptotic neutrophils with greater sensitivity compared with morphologic assessment.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/PL00007597
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    Paper (German National Licenses)
    Fulltext
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