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  • 1
    Electronic Resource
    Electronic Resource
    Tris(trimethylphosphine)cobalt(I) halides. Preparation and properties (1975)
    s.l. : American Chemical Society
    Inorganic chemistry 14 (1975), S. 473-477 
    Details
    ISSN: 1520-510X
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ic50145a004
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  • 2
    Electronic Resource
    Electronic Resource
    Nickel phosphine complexes. Crystal and molecular structure of a new nickel(I) complex: [Ni(P(CH3)3)4]B(C6H5)4 (1977)
    s.l. : American Chemical Society
    Journal of the American Chemical Society 99 (1977), S. 5187-5189 
    Details
    ISSN: 1520-5126
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ja00457a053
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  • 3
    Electronic Resource
    Electronic Resource
    Putrescine and Spermidine in Peas: Effeets on Nitrogen Source and Potassium Supply (1979)
    Oxford, UK : Blackwell Publishing Ltd
    Physiologia plantarum 45 (1979), S. 0 
    Details
    ISSN: 1399-3054
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology
    Notes: Free and total putrescine and, to a lesser extent, spemidine accumulate in both roots and shoots of peas in response to potas stum deficiency and ammomnium supply. Free putrescine responds more sensitively than total putreseine to variatioin of nutrients. Accumulation of putrescine is more pronounced in the roots than in the shoots.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1399-3054.1979.tb02621.x
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  • 4
    Electronic Resource
    Electronic Resource
    Zur Feinstruktur der Basalzelladenome der Speicheldrüsen (1971)
    Springer
    Virchows Archiv 353 (1971), S. 333-346 
    Details
    ISSN: 1432-2307
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Die feinstrukturelle Untersuchung von 7 Basalzelladenomen der Glandula parotis ergibt, daß diese gutartigen Speicheldrüsentumoren aus epithelial differenzierten Zellen aufgebaut sind. Die Geschwulstzellen enthalten außer einem spärlichen Inventar von Zellorganellen zahlreiche ungeordnet verlaufende feine Fibrillen, wie sie auch in anderen Tumoren der Speicheldrüsen und der Brustdrüse vorkommen. Herkunft und Funktion dieser Fasern sind nicht bekannt. Die epithelial differenzierten Geschwulstzellen wirken offensichtlich mit an der Bildung von interstitiellen Fibrillen und basalmembranartiger Substanz, wobei in einzelnen Basalzelladenomen der Eindruck einer ödematösen Durchtränkung des Geschwulststromas entsteht. Eine Besonderheit der Basalzelladenome stellt darüber hinaus das Vorkommen von Elastica-Material zwischen den Geschwulstepithelien dar. Da in den Basalzelladenomen Myoepithelzellen am Geschwulstaufbau nicht beteiligt sind, können sie von den Myoepithel führenden Speicheldrüsentumoren, wie Mischtumoren, Cylindromen und Speichelgangcarcinomen eindeutig abgegrenzt werden. Sie stellen eine eigenständige Geschwulstart dar und sind aufgrund der feinstrukturellen Organisation in die Gruppe der epithelialen Geschwülste der Speicheldrüsen einzuordnen.
    Notes: Summary A fine structural investigation of seven basal-cell adenomas of the parotid gland reveals their cells undergo epithelial differentiation. They contain few cell organelles and many fine filaments, a state that has also been observed in many other salivary gland tumors. The origin and function of these fibrils is not known. The cells of these epithelial tumors obviously take part in the formation of interstitial fibrils and basement membrane-like material. In some basal-cell adenomas the stroma becomes edematous. A peculiarity of these tumors is the occurrence of elastica-like material among the tumor cells. Since myoepithelial cells do not occur, the basal-cell adenoma can easily be separated from salivary gland tumors containing myoepithelial cells, i.e., from the pleomorphic adenoma (socalled mixed tumor), the adenoid-cystic carcinoma (cylindroma), and the salivary duct carcinoma. The basal-cell adenoma is a special type of salivary gland tumor and because of its fine structure has to be included with the epithelial neoplasms of the salivary glands.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00549046
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  • 5
    Electronic Resource
    Electronic Resource
    Renal effects of furosemide in glycerol induced acute renal failure of the rat (1976)
    Springer
    Pflügers Archiv 365 (1976), S. 81-87 
    Details
    ISSN: 1432-2013
    Keywords: Renal failure ; Micropuncture ; Diuretics ; Glomerular filtration rate ; Effective filtration pressure ; Renal blood flow
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The renal effects of furosemide in acute renal failure of the rat were studied using clearance and micropuncture techniques. Acute renal failure was induced by an intramuscular injection of 50% glycerol (10 ml/kg). Functional impairement of the glycerol treated animals consisted of a decrease in urinary sodium excretion, renal blood flow, total kidney GFR and effective filtration pressure of superficial nephrons. Effective filtration pressure was calculated from proximal free flow and stop flow pressure measurements. In contrast to control animals furosemide did not increase urine volume during acute renal failure due to a marked fall in GFR. Renal blood flow, as measured by an electromagnetic flowmeter, also decreased after furosemide in glycerol treated rats and increased in control animals. Furosemide reduced effective filtration pressure during acute renal failure to almost zero, whereas in control animals effective filtration pressure virtually remained constant.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00583631
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  • 6
    Electronic Resource
    Electronic Resource
    Zur Pathogenese stressbedingter Magenulcera (1971)
    Springer
    Virchows Archiv 352 (1971), S. 195-208 
    Details
    ISSN: 1432-2307
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Aufgrund funktioneller, licht- und elektronenmikroskopischer Untersuchungen an 49 Ratten in Zwangshaltung läßt sich ein differenziertes Konzept zum lokalen Entstehungsmechanismus stressbedingter Magenulcera ableiten. Die erste Phase erstreckt sich über die ersten 3 Std und läßt funktionelle Veränderungen insbesondere an den Gefäßen der tieferen Wandschichten des Magens erkennen. Die daraus resultierende Zirkulationsstörung in der Schleimhaut selbst führt in einer weiteren Phase zur Entwicklung einer Hypoxie mit trophischen Gewebeschäden. Der in dieser zweiten Phase unter cholinergischem Einfluß sezernierte hochkonzentrierte hyperacide stagnierende Magensaft wird durch eine gleichzeitige hochgradige Kontraktion der Magenwand in innigen Kontakt mit der Magenschleimhaut gebracht und verursacht etwa eine Stunde nach Überschreitung der Gipfelsekretion in der dritten, läsionellen Phase die organischen Schädigungen in Form von Erosionen und Ulcera.
    Notes: Summary Gastric secretion and the development of gastric lesions were studied in the rat under total restraint. Three phases can be distinguished: The first one lasts about 3 hours and is characterised by functional vascular disturbances in the deeper layers of the gastric wall, especially by a constriction of the arteriolae in the submucosa. It follows a second phase with trophic alteration of the mucosa by hypoxia and stasis in the capillaries of the mucosa itself. At this time (about 5 hours after beginning of the experiment) moderate amounts of gastric juice with a high concentration of hydrochloric acid are secreted. Its action on the altered mucosa is intensified by the contraction of the gastric wall, which guarantees a close contact of the stagnant acid juice with the mucous membrane. About one hour after the peak of acid secretion, in a third phase, irreversible changes in form of erosions and ulcerations of the gastric mucosa can be observed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00600670
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  • 7
    Electronic Resource
    Electronic Resource
    Coronary and endocardial fibroelastosis of the ventricles in the hypoplastic left and right heart syndromes (1975)
    Springer
    Virchows Archiv 368 (1975), S. 87-97 
    Details
    ISSN: 1432-2307
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In an autopsy material of 29 cases of the hypoplastic left heart syndrome coronary fibroelastosis was found in 1 case, endocardial fibroelastosis in 8 eases. Figures for 10 cases of the hypoplastic right heart syndrome were 6 cases of coronary fibroelastosis and 1 ease of endocardial fibroelastosis. Age ranged from stillborn up to 11 1/2 months. Coronary and endocardial fibroelastosis seemed to be mutually exclusive localizations of congenital fibroelastosis since in our material they did not occur together in the same hearts. In hypoplastic right hearts coronary fibroelastosis was either restricted to the right coronary artery (right circumflex and posterior interventricular branch), or it was found also in the left coronary artery (anterior interventricular branch), with the most serve affections always being situated in the right one. In the only case of coronary fibroelastosis among the hypoplastic left hearts the condition was limited to the anterior interventricular branch of the left coronary artery which communicated with the hypoplastic left ventricle by a fistula. Coronary fibroelastosis was exclusively found in branches supplying the hypoplastic right ventricle and/or in a branch connected by a fistula to the hypoplastic left or right ventricle. Endocardial fibroelastosis was generally found in hypoplastic left ventricles with either no outflow or with severe outflow obstruction. A theory concerning the aetiology of both coronary and endocardial fibroelastosis of the hypoplastic ventricles is proposed. It is argued that development of fibroelastosis may in both localizations be caused or favoured by the coincidence of two factors: abnormal haemodynamic conditions and poor oxygenation of blood and tissues. Observations made in a reference material of 35 hypoplastic left and 24 hypoplastic right hearts were in accordance with this view
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00432410
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  • 8
    Electronic Resource
    Electronic Resource
    Der Umbau des Genoms von Pisum sativum durch induzierte Translokationen der Satelliten-Chromosomen (1974)
    Springer
    Theoretical and applied genetics 44 (1974), S. 69-72 
    Details
    ISSN: 1432-2242
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Description / Table of Contents: Zusammenfassung In der vorliegenden Arbeit werden die Karyotypen von zwei röntgeninduzierten Formen von Pisum sativum dargestellt und mit dem Karyotyp der Ausgangsform ‘Dippes gelbe Viktoria’ verglichen. Die zytologische Analyse erbrachte deutliche Unterschiede zwischen der Vergleichsform und diesen beiden Linien, die auf jeweils eine einfache reziproke Translokation zurückgeführt werden konnten. In beiden Fällen waren Satelliten-Chromosomen an dem Austauschprozeß beteiligt. Bei 62B erfolgte eine Translokation zwischen dem langen Arm von Chromosom V und dem satellitentragenden langen Arm von VII. Die Linie 488 entstand durch Austausch zwischen den beiden Satelliten-Chromosomen IV und VII, und zwar in der Weise, daß ein sehr kleines satellitenfreies und ein sehr langes Chromosom gebildet wurde, welches an den Enden seiner etwa gleich langen Arme jeweils einen Satelliten trägt. Die Vereinigung der beiden Satelliten-Regionen in einem Chromosom ist bisher für Pisum noch nicht beschrieben worden. Zum Schluß wurde kurz die Bedeutung diskutiert, die solche drastischen Veränderungen des Karyotyps für die züchterische Praxis und für evolutionistische Fragestellungen haben könnten.
    Notes: Summary In connection with X-ray experiments on Pisum sativum ‘Dippes gelbe Victoria’, two translocation line showing dramatic deviations from the normal karyotype were selected and cytologically analysed (root tips). In line 62B, an exchange between the long arms of chromosome V and the satellite chromosome VII took place. In line 488, both satellite chromosomes IV and VII were involved in the exchange process, leading to a very short chromosome (VII T!), and a very long chromosome (IV T!) uniting both the satellites in one chromosome. The importance of these lines with regard to mutation breeding and evolution is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00277955
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  • 9
    Electronic Resource
    Electronic Resource
    Action of the competitive angiotensin II antagonist saralasin during the initial phase of glycerol-induced acute renal failure of the rat (1977)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 301 (1977), S. 139-143 
    Details
    ISSN: 1432-1912
    Keywords: Renal function ; Renal blood flow ; Glomerular filtration rate ; Urine volume ; Urinary sodium excretion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The effects of the competitive angiotensin II antagonist saralasin (1-sarcosine-8-alanine-5-isoleucine-angiotensin II) on renal function in healthy rats and in rats with myohemoglobinuric acute renal failure were studied. Acute renal failure was induced by an intramuscular injection of 50% glycerol (10 ml ·kg−1). Functional impairment of the glycerol treated animals consisted in a decrease of renal blood flow (electromagnetic flowmeter) and GFR and in an increase of urine volume and arterial blood pressure. In healthy rats saralasin (6 μg·kg−1·min−1 i.v.) had no renal effects by itself but antagonized the angiotensin II (200 ng·kg−1·min i.v.) induced fall of renal blood flow and GFR and the increase of arterial blood pressure. Given to glycerol treated animals saralasin did not induce any change of arterial blood pressure, renal blood flow, GFR or the urinary excretion of fluid and sodium.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00501429
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  • 10
    Electronic Resource
    Electronic Resource
    Intracerebral cavernous hemangioma with angiographically demonstrated pathologic vasculature (1971)
    Springer
    Neuroradiology 3 (1971), S. 57-63 
    Details
    ISSN: 1432-1920
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Résumé Les hémangiomes cérébraux caverneux sont des malformations vasculaires rares. Dans les dix cas étudiés précédemment par angiographie, les auteurs constatèrent un déplacement artériel, mais aucune vascularisation pathologique n'était visible. Ils décrivent le cas d'un malade porteur d'un cavernome avec néovascularisation, opacification de la tumeur et remplissage veineux précoce à l'angiographie. En raison de la relative facilité de l'ablation chirurgicale des hémangiomes caverneux, les auterus conseillent de les prendre en considération dans la discussion du diagnostic différentiel
    Abstract: Zusammenfassung Die cerebralen kavernösen Hämangiome sind seltene Gefäßmißbildungen Bisher konnten pathologische Vascularisierungen nicht beschrieben werden. Es handelt sich bei der vorliegenden Arbeit um eine Fallbeschreibung eines kavernösen Hämangioms mit dem Nachweis von Gefäßneubildungen und vorzeitigem Venenabfluß.
    Notes: Summary Cerebral cavernous hemangiomas are rare vascular malformations. In the ten cases previously studied by angiography, arterial displacement was demonstrated, but pathologic vasculature has not been seen. The first patient with a cavernoma in which neovasculature, a stain, and early venous filling were discernible at angiography is described. Because of the relative ease of the surgical removal of cavernous hemangiomas, they should be considered in the differential diagnosis of lesions with these angiographic features.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00339895
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