Library

X
feed icon rss

Your email was sent successfully. Check your inbox.

An error occurred while sending the email. Please try again.

Proceed reservation?

Export
  • 1
    Electronic Resource
    Electronic Resource
    New α-decaying neutron deficient isotopes197Rn and200Fr (1995)
    Springer
    The European physical journal 352 (1995), S. 7-8 
    Details
    ISSN: 1434-601X
    Keywords: 23.60.+e ; 25.70.−z ; 27.80.+w
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract New neutron-deficient isotopes,197Rn,197mRn, and200Fr have been produced and identified on the basis of genetic correlations in the166Er(36Ar,5n)197Rn (Elab=186, 200 MeV), and169Tm(36Ar,5n)200Fr (Elab=186 MeV) reactions. The evaporation residues were separated from the beam by using a gas-filled recoil separator and implanted onto a position-sensitive solid-state detector. The α-decay energies (half-lives) of197Rn,197mRn and200Fr have been determined to be 7261±30 keV (51 −15 +35 ms), 7370±30 keV (18 −5 +9 ms), and 7500±3O keV, (570 −140 +270 ms), respectively.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01292753
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Identification of an abdominal ganglion neuron antagonizing L7-driven gill contraction in Aplysia (1999)
    Springer
    Journal of comparative physiology 185 (1999), S. 11-19 
    Details
    ISSN: 1432-1351
    Keywords: Key words Aplysia ; Gill ; Motor neuron ; L7 ; Abdominal ganglion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Gill motor neuron L7-induced longitudinal shortening of the gill in Aplysia kurodai and A. juliana was suppressed when extracellular stimuli were applied to a restricted dorsal central region of the abdominal ganglion. We found a neuron there which antagonized the L7-driven contraction. Since the contraction was suppressed when the identified neuron was activated simultaneously with L7, we refer to the newly found neuron as “Anti-L7”. Anti-L7 did not change the L7 impulse generation in the abdominal ganglion. No direct synaptic connection from L7 to Anti-L7 was detected. A fluorescent dye injected into the soma of Anti-L7 revealed that the neuron sent axonal branches to the branchial nerve. These results may show that Anti-L7 antagonizes L7 at the periphery inside the gill, rather than in the abdominal ganglion. EJPs induced by L7 were unaffected by Anti-L7. Activation of Anti-L7 alone did not induce any change in tone or membrane potential of the gill musculature. The suppressive effect of Anti-L7 lasts many seconds after the cessation of a train of Anti-L7 impulses. The results may suggest that the suppression is mediated through an inhibitory neuromodulatory mechanism without inhibition of L7 itself.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003590050361
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
    Paper (German National Licenses)
    Fulltext
Close ⊗
This website uses cookies and the analysis tool Matomo. More information can be found here...