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  • 2000-2004  (2)
  • 1965-1969
  • 1960-1964
  • 1945-1949
  • 1925-1929
  • K+ channel  (1)
  • PACS. 25.70.-z Low and intermediate energy heavy-ion reactions – 24.30.Cz Giant resonances – 23.20.-g Electromagnetic transitions – 25.70.Gh Compound nucleus  (1)
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  • 2000-2004  (2)
  • 1965-1969
  • 1960-1964
  • 1945-1949
  • 1925-1929
Year
Keywords
  • 1
    ISSN: 1434-601X
    Keywords: PACS. 25.70.-z Low and intermediate energy heavy-ion reactions – 24.30.Cz Giant resonances – 23.20.-g Electromagnetic transitions – 25.70.Gh Compound nucleus
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract: The 32S + 100Mo and 36S + 96Mo fusionlike reactions were studied at incident energy of E lab = 298 MeV and 320 MeV, respectively, with the aim of probing the influence of the entrance channel charge asymmetry on the dipole γ-ray emission. The excitation energy and spin distribution of the compound nucleus created in these reactions were identical, the only difference being associated with the unequal charge asymmetry of the two entrance channels. High-energy γ-rays were detected in an array of 9 seven-pack BaF2 clusters. Coincidence with fusionlike residues detected in four PPAC ensured the selection of central reaction events. By studying the differential γ-ray multiplicity associated with the two reactions it was shown that the dipole strength excited in the compound nucleus increases with the entrance channel charge asymmetry. From the linearized spectra, the increase of the GDR γ-ray intensity was found to be ∼ 25% for the more charge asymmetric system. The results are discussed and compared with those of previous data obtained at different incident energies.
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 1364-6753
    Keywords: Key words Kcnab2 ; Bis1 ; K+ channel ; Epilepsy ; Mice
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: ABSTRACT¶We tested the hypothesis that Bis1, a gene involved in seizure regulation in mice which has been localized to the distal part of chromosome 4, was the same as the gene Kcnab2, encoding the β2-subunit for voltage-dependent K+ channels. Two facts suggested this hypothesis: Kcnab2 is located in the 3.1-cM confidence interval containing Bis1 and many studies have shown an involvement of K+ channels in the genesis of seizures. DNA sequence analysis of the coding sequence for Kcnab2 from JE/Le mice revealed no structural alterations which might affect Kcnab2 function. However, several nucleotide changes were observed.
    Type of Medium: Electronic Resource
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