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  • 2000-2004  (2)
  • Cyclosporin A  (1)
  • N2O decomposition  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Isotopic study of nitrous oxide decomposition on an oxidized Rh catalyst: mechanism of oxygen desorption (2000)
    Springer
    Catalysis letters 66 (2000), S. 87-90 
    Details
    ISSN: 1572-879X
    Keywords: 18O isotope ; N2O decomposition ; oxidized Rh catalyst ; reaction mechanism
    Source: Springer Online Journal Archives 1860-2000
    Topics: Chemistry and Pharmacology
    Notes: Abstract N2O decomposition on an oxidized Rh catalyst (unsupported) has been studied using a tracer technique in order to reveal the reaction mechanism. N2 16O was pulsed onto an 18O/oxidized Rh catalyst at 493 K and desorbed O2 molecules were monitored. The 18O fraction in the desorbed oxygen had the same value as that on the surface oxygen. The result shows that the oxygen molecules do not desorb via the Eley–Rideal mechanism, but via the Langmuir–Hinshelwood mechanism. On the other hand, desorption of oxygen from Rh surfaces (in vacuum or in He) occurs at higher temperatures, which suggests reaction-assisted desorption of oxygen during the N2O decomposition reaction at low temperature.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1019066732528
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  • 2
    Electronic Resource
    Electronic Resource
    Skeletal effects of low-dose cyclosporin A therapy in children with nephrotic syndrome (2000)
    Springer
    Clinical and experimental nephrology 4 (2000), S. 313-317 
    Details
    ISSN: 1437-7799
    Keywords: Key words Bone mineral density ; Cyclosporin A ; Glucocorticoid ; Nephrotic syndrome ; Osteopenia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Background. High doses of cyclosporin A (CsA) produce high-turnover osteopenia in rats. The aim of this study was to determine whether low-dose CsA affects the skeleton in children with nephrotic syndrome. Methods. Biochemical parameters of mineral and skeletal homeostasis, and bone mineral density (BMD) in eight boys with steroid-dependent, frequently relapsing minimal change nephrotic syndrome who had received low-dose CsA (between 1.6 and 3.1 mg/kg per day) for 2 years were compared with measurements in the same patients before CsA therapy and who had received glucocorticoids for long periods, and with measurements in age-matched controls. Results. It was possible to discontinue glucocorticoid therapy within 4 months after the start of CsA therapy. There was a significant increase in the mean serum alka-line phosphatase concentration in CsA therapy patients compared with the same patients before CsA therapy and the controls. Serum osteocalcin and tartrate-resistant acid phosphatase, and urinary deoxypyridinoline concentrations in CsA therapy patients did not differ from those in the controls. BMD in CsA therapy patients was increased significantly compared with values in the same patients before CsA therapy. BMD in CsA therapy patients was lower than that in the controls, but remained within 80% of the overall mean BMD value. Conclusions. Two years of low-dose CsA therapy without glucocorticoids does not appear to induce high-turnover osteopenia in children with steroid-dependent nephrotic syndrome.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s101570070007
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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