Library

feed icon rss

Your email was sent successfully. Check your inbox.

An error occurred while sending the email. Please try again.

Proceed reservation?

Export
Filter
  • 1995-1999  (3)
  • 21.60.Cs  (1)
  • Imaging  (1)
  • Key words Biomechanics • Resultant hip force • Acetabular labrum • Acetabular dysplasia • Osteoarthritis  (1)
  • 1
    ISSN: 1434-601X
    Keywords: 23.20.Lv ; 21.60.Cs ; 27.70.+q
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract In-beamγγ- andγγn-measurements have for the first time identified excited states in the 72 157 Hf85 nucleus and have established its yrast levels up to 6.5 MeV and I=(51/2). The results of parameter-free 11-particle recoupling calculations in the framework of the shell model for the configurationsπh 11 2/8 νf 7 2/3 andπh 11 2/8 νf 7 2/2 h9/2 are in excellent agreement with the observed levels up to 39/2 at 4.758 MeV. A weakly populated 52 ns 29/2+ yrast isomer at 2.876 MeV is assigned as the three-neutron configurationνf7/2h9/2i13/2.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 2
    ISSN: 1433-0431
    Keywords: Key words Biomechanics • Resultant hip force • Acetabular labrum • Acetabular dysplasia • Osteoarthritis ; Schlüsselwörter Biomechanik • Hüftgelenksresultierende • Labrum acetabulare • Hüftdysplasie • Koxarthrose
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Die Labrumläsion beim biomechanischen Fehlbau des Hüftgelenks ist Symptom der chronischen Gelenkinstabilität und Fehlbelastung. Durch die schräg und exzentrisch überdachende Facies lunata wird der Mechanismus der kongruenten Inkongruenz und das Prinzip des elastischen Hüftgelenks gestört. Der Hüftkopf wird gegen den ventrokraniolateralen Erker gedrängt und verursacht eine Streßkonzentration im dort gelegenen Labrum-Kapsel-Komplex. Dieser kompensiert vorübergehend als sekundärer Stabilisator die latente Instabilität solange bis früher oder später die Ruptur als Ausdruck der mechanischen Dekompensation eintritt. Dadurch verschlimmert sich die vorbestehende Instabilität und der Arthroseprozeß kann lawinenartig fortschreiten. Biomechanisch betrachtet ist die Labrumläsion sowohl „Symptom“ einer latenten Instabilität als auch „Auslöser“ und „Schrittmacher“ einer rasch fortschreitenden „sekundären“ Koxarthrose. Therapeutische Konsequenz ist deshalb die rechtzeitige Normalisierung des zugrundeliegenden Fehlbaus durch korrektive Osteotomien.
    Notes: Summary The capsular-labrum-complex consists of the triangular fibrocartilaginous labrum, which is fixed at the bony acetabular ring and supported by the ligamentum transversum actetabuli in the caudal part. In a normally developed and correctly orientated acetabulum, the femoral head is symmetrically covered by the contact area of the lunate surface. The vertical component of the resultant hip joint force concentrically meets the horizontal weight bearing surface: Evenly distributed compressive forces are transmitted; the capsular-labrum-complex does not have to compensate excessive shear or tension. In contrast, in a “dysplastic” acetabulum the femoral head is poorly covered by the pathologically orientated lunate surface: The lunate surface is sloping in an anterolateral direction, leading to tension and shear on the superolateral capsular-labrum-complex, which becomes a secondary stabilizer (“guide rail”) against the decentering femoral head. The vertical component of the resultant hip force eccentrically meets the oblique weight bearing surface, causing extra stress in the labrum: The capsular-labrum-complex is trying to compensate this biomechanical stress with hypertrophy (Type IB). Progressive mechanical decompensation of the capsular-labral-complex might lead to tears (Type IIB) or complete avulsions (Type IIIB) of the labrum. In conclusion labral lesions result from pathobiomechanics caused by residual hip dysplasia (RHD). Logically, the basic therapeutic principle can only be corrective osteotomy.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 3
    Electronic Resource
    Electronic Resource
    Springer
    European radiology 7 (1997), S. 180-186 
    ISSN: 1432-1084
    Keywords: Key words: AVN ; Pathophysiology ; Imaging
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract. The etiology of avascular necrosis (AVN) is multifactorial. Independent of its etiology and localization it shows typical pathologies and radiological images. In the early stages localized subchondral edema is characteristic. In 50 % of all cases accompanying joint effusion may be found. Due to necrosis of the cells of bone marrow and bone fibrovascular, reactions with hyperemia can be delineated. These reactions allow us to visualize necrosis indirectly. The best imaging methods are MRI and, to a lesser extent, bone scintigraphy. In later stages calcification as well as new bone formation and microfractures are typically demonstrated and visualized best with plain X-rays and CT. Why reparations in many cases, particularly in the hip, are incomplete and may stop in any stage is unknown. Over years clinically complete silent AVNs are not an uncommon finding. Prognosis depends on the localization and size of the AVN. The number of repair mechanisms is best outlined with contrast-enhanced MRI and return of fatty marrow.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
Close ⊗
This website uses cookies and the analysis tool Matomo. More information can be found here...