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  • 1995-1999  (5)
  • Advance directives  (2)
  • C3-C4 intermediate photosynthesis  (1)
  • Cell & Developmental Biology  (1)
  • Hordeum vulgare  (1)
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Materialart
Erscheinungszeitraum
  • 1995-1999  (5)
Jahr
Schlagwörter
  • 1
    Digitale Medien
    Digitale Medien
    Limitation of life support: Frequency and practice in a London and a Cape Town intensive care unit (1996)
    Springer
    Intensive care medicine 22 (1996), S. 1020-1025 
    Details
    ISSN: 1432-1238
    Schlagwort(e): Critical care ; Ethics ; Resuscitation orders ; Advance directives ; Life support withdrawal ; Prognosis ; Severity of illness index
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract Objectives To examine the frequency of limiting (withdrawing and withholding) therapy in the intensive care unit (ICU), the grounds for limiting therapy, the people involved in the decisions, the way the decisions are implemented and the patient outcome. Design Prospective survey. Ethical approval was obtained. Setting ICUs in tertiary centres in London and Cape Town. Patients All patients who died or had life support limited. Interventions Data collection only. Results There were 65 deaths out of 945 ICU discharges in London and 45 deaths out of 354 ICU discharges in Cape Town. Therapy was limited in 81.5% and 86.7% respectively (p=0.6) of patients who died. The mean ages of patients whose therapy was limited were 60.2 years and 51.9 years (p=0.014) and mean APACHE II scores 18.5 and 22.6 (p=0.19) respectively. The most common reason for limiting therapy in both centres was multiple organ failure. Both medical and nursing staff were involved in most decisions, which were only implemented once wide consensus had been reached and the families had accepted the situation. Inotropes, ventilation, blood products, and antibiotics were most commonly withdrawn. The mean time from admission to the decision to limit therapy was 11.2 days in London and 9.6 days in Cape Twon. The times to outcome (death in all patients) were 13.2 h and 8.1 h respectively. Conclusions Withdrawal of therapy occurred commonly, most often because of multiple organ failure. Wide consensus was reached before a decision was made, and the time to death was generally short.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF01699222
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  • 2
    Digitale Medien
    Digitale Medien
    Limitation of life support: frequency and practice in a London and a Cape Town intensive care unit (1996)
    Springer
    Intensive care medicine 22 (1996), S. 1020-1025 
    Details
    ISSN: 1432-1238
    Schlagwort(e): Key words Critical care ; Ethics ; Resuscitation orders ; Advance directives ; Life support withdrawal ; Prognosis ; Severity of illness index
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract Objectives: To examine the frequency of limiting (withdrawing and withholding) therapy in the intensive care unit (ICU), the grounds for limiting therapy, the people involved in the decisions, the way the decisions are implemented and the patient outcome. Design: Prospective survey. Ethical approval was obtained. Setting: ICUs in tertiary centres in London and Cape Town. Patients: All patients who died or had life support limited. Interventions: Data collection only. Results: There were 65 deaths out of 945 ICU discharges in London and 45 deaths out of 354 ICU discharges in Cape Town. Therapy was limited in 81.5% and 86.7% respectively (p=0.6) of patients who died. The mean ages of patients whose therapy was limited were 60.2 years and 51.9 years (p=0.014) and mean APACHE II scores 18.5 and 22.6 (p=0.19) respectively. The most common reason for limiting therapy in both centres was multiple organ failure. Both medical and nursing staff were involved in most decisions, which were only implemented once wide consensus had been reached and the families had accepted the situation. Inotropes, ventilation, blood products, and antibiotics were most commonly withdrawn. The mean time from admission to the decision to limit therapy was 11.2 days in London and 9.6 days in Cape Town. The times to outcome (death in all patients) were 13.2 h and 8.1 h respectively. Conclusions: Withdrawal of therapy occurred commonly, most often because of multiple organ failure. Wide consensus was reached before a decision was made, and the time to death was generally short.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF01699222
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
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  • 3
    Digitale Medien
    Digitale Medien
    Cellular expression pattern of the glycine decarboxylase P protein in leaves of an intergeneric hybrid between the C3-C4 intermediate species Moricandia nitens and the C3 species Brassica napus (1998)
    Springer
    Theoretical and applied genetics 96 (1998), S. 922-927 
    Details
    ISSN: 1432-2242
    Schlagwort(e): Key words Brassica napus ; C3-C4 intermediate photosynthesis ; Glycine decarboxylase ; Intergeneric hybrid ; Moricandia arvensis
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Biologie
    Notizen: Abstract  An intergeneric hybrid plant was produced between the C3-C4 intermediate species Moricandia nitens and the C3 species Brassica napus by sexual hybridization and in vitro embryo rescue. The hybrid nature of the plant was apparent in its morphology and flower pigmentation and was confirmed by leaf isozyme patterns. The overall plant morphology and the shape and thickness of leaves of the hybrid plant were intermediate between those of the parent species. However, the bundle-sheath cells of the hybrid resembled those of the C3 parent and lacked the organelle development of the C3-C4 intermediate parent. Immunogold labelling for the presence of the P subunit of the mitochondrial glycine decarboxylase complex revealed a very similar labelling density on mitochondria in bundle-sheath and mesophyll cells in B. napus, while in  M. nitens the P subunit was only detectable in bundle sheath cells. In the hybrid the labelling density on mesophyll cell mitochondria was almost half of that on the bundle-sheath mitochondria. The CO2 compensation point of the hybrid was significantly less than that of the C3 parent but was not as low, nor as responsive to changes in light intensity, as for the C3-C4 parent.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/s001220050821
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  • 4
    Digitale Medien
    Digitale Medien
    Variation in thylakoid proteins and photosynthesis in Syrian landraces of barley (1995)
    Springer
    Euphytica 82 (1995), S. 213-220 
    Details
    ISSN: 1573-5060
    Schlagwort(e): barley landraces ; drought ; Hordeum vulgare ; leaf colour ; photosynthesis ; photosystem I and II ; thylakoid chlorophyll-proteins
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Land- und Forstwirtschaft, Gartenbau, Fischereiwirtschaft, Hauswirtschaft
    Notizen: Summary Barley breeders at ICARDA have observed that genotypes adapted to dry regions have leaves which are lighter in colour than those of unadapted ones. We measured photosynthesis, chlorophyll content and chlorophyll a:b ratios in two sets of genotypes which had previously been observed to have either light green or dark green leaves when grown in the field. Thylakoid membranes were also extracted and the proteins analysed on SDS-PAGE gels. The light leaf colour was associated with a higher chlorophyll a:b ratio. This was a measure of a reduction in the amount of antenna chlorophyll compared to that in the core complex of PSII. Genotypes with light green leaves had consistently less chlorophyll per unit leaf area and lower photosynthetic rates per unit area than those with dark green leaves. It is suggested that these features of light green leaves may confer the ability to adapt to high levels of irradiance under drought conditions. This ability may result from a high rate of photosynthetic electron transport through each PSII reaction centre, thus reducing the risk of damage from the overexcitation of these centres.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF00029563
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  • 5
    Digitale Medien
    Digitale Medien
    Bone morphogenetic proteins inhibit adipocyte differentiation by bone marrow stromal cells (1995)
    New York, N.Y. : Wiley-Blackwell
    Journal of Cellular Biochemistry 58 (1995), S. 393-402 
    Details
    ISSN: 0730-2312
    Schlagwort(e): adipocytes ; bone morphogenetic proteins ; differentiation ; bone marrow stromal cells ; transforming growth factor β ; Life and Medical Sciences ; Cell & Developmental Biology
    Quelle: Wiley InterScience Backfile Collection 1832-2000
    Thema: Biologie , Chemie und Pharmazie , Medizin
    Notizen: The bone morphogenetic proteins were originally identified based on their ability to induce ectopic bone formation in vivo and have since been identified as members of the transforming growth factor-β gene superfamily. It has been well established that the bone morphogenetic cytokines enhance osteogenic activity in bone marrow stromal cells in vitro. Recent reports have described how bone morphogenetic proteins inhibited myogenic differentiation of bone marrow stromal cells in vitro. In vivo, bone marrow stromal cells differentiate along the related adipogenic pathway with advancing age. The current work reports the inhibitory effects of the bone morphorphogenetic proteins on adipogenesis in a multipotent murine bone marrow stromal cell line, BMS2. When exposed to bone morphogenetic protein-2, the pre-adipocyte BMS2 cells exhibited the expected induction of the osteogenic-related enzyme, alkaline phosphatase. Following induction of the BMS2 cells with adipogenic agonists, adipocyte differentiation was assessed by morphologic, enzymatic, and mRNA markers. Flow cytometric analysis combined with staining by the lipophilic fluorescent dye, Nile red, was used to quantitate the extent of lipid accumulation within the BMS2 cells. By this morphologic criteria, the bone morphogenetic proteins inhibited adipogenesis at concentrations of 50 to 500 ng/ml. This correlated with decreased levels of adipocyte specific enzymes and mRNAs. The BMS2 pre-adipocytes constitutively expressed mRNA encoding bone morphogenetic protein-4 and this was inhibited by adipogenic agonists. Together, these findings demonstrate that bone morphogenetic proteins act as adipogenic antagonists. This supports the hypothesis that adipogenesis and osteogenesis in the bone marrow microenvironment are reciprocally regulated.
    Zusätzliches Material: 4 Ill.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1002/jcb.240580312
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