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  • 1985-1989  (2)
  • 1980-1984  (1)
  • Brain infarction  (1)
  • Cyclosporin A  (1)
  • EPSP  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Cyclosporin A enhances Streptozocin-induced diabetes in CD-1 mice (1987)
    Springer
    Cellular and molecular life sciences 43 (1987), S. 324-327 
    Details
    ISSN: 1420-9071
    Keywords: Cyclosporin A ; cellular immunity ; Streptozocin ; insulitis ; diabetes mellitus
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Cyclosporin A (CYA), when administered to CD-1 mice treated with a subdiabetogenic dose of Streptozocin (STZ), exacerbated the STZ-induced insulitis and elevated the plasma glucose levels, parallel to a reduction of the insulin content of the pancreas. The possible mechanisms of CYA-mediated aggravation of STZ-induced diabetes are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01945570
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Effects of amino acids on cat red nucleus neurons in vitro (1984)
    Springer
    Experimental brain research 54 (1984), S. 150-156 
    Details
    ISSN: 1432-1106
    Keywords: EPSP ; IPSP ; Amino acids ; Red nucleus ; Brain slice
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Intracellular records were obtained from neurons in the region of the red nucleus (RN) of cat brain slices. Both EPSPs and IPSPs were recorded in response to local electrical stimulation and these resembled similar electrophysiological responses observed in experiments conducted in vivo. Monosynaptic and polysynaptic IPSPs were observed, suggesting the existence of inhibitory interneurons near or within the RN region. When added to the bathing solution, L-glutamate and L-aspartate depolarized RN neurons with a decrease in input resistance. γ-Aminobutyric acid (GABA) and glycine hyperpolarized the cells with a decrease in input resistance. GABA also elicited a depolarizing response. These amino acid actions had direct postsynaptic effects, since the experiments were conducted in a low Ca2+/high Mg2+ medium which blocked synaptic transmission.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00235826
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Magnetic resonance imaging of cerebral infarction: Time course of Gd-DTPA enhancement and CT comparison (1988)
    Springer
    Neuroradiology 30 (1988), S. 372-378 
    Details
    ISSN: 1432-1920
    Keywords: Magnetic resonance (MR) imaging ; Gd-DTPA ; Brain infarction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Thirty-five patients (7 females and 28 males) with cerebral infarction and suspicion of cerebral infarction of 4 h to 27 months duration were studied 45 times with magnetic resonance (MR) imaging using Bd-DTPA. Spin echo (SE) images were obtained before and after the administration of Gd-DTPA (0.1 or 0.15 mmol/kg) and compared with the enhanced CT. MR imaging using Gd-DTPA was more sensitive than enhanced CT and very useful for detecting a new focus of cerebral infarction, especially in the cases with multiple infarcted areas and for showing the extent of cortical and subcortical infarction. In most cases the MR enhancement was obvious in the subacute stage, especially after cerebral embolism, and the signal intensity of the lesion tended to show a gradual increase. The diagnosis of embolism was accepted on the basis of acute onset without prior TIA, coupled with angiography showing the embolus itself and/or a capillary blush and a wide area of infarction.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00404100
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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