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  • 1985-1989  (5)
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Material
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Year
  • 1
    Electronic Resource
    Electronic Resource
    Interleukin-1β as a potent hyperalgesic agent antagonized by a tripeptide analogue (1988)
    [s.l.] : Nature Publishing Group
    Nature 334 (1988), S. 698-700 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] Human recombinant (h.r.) IL-1a, h.r. IL-1Β and peptide analogues of IL-1Β (Table 1) were tested for hyperalgesic/analgesic activities in a modified Randall-Sellito rat-paw pressure test9. Three of the peptides (£121-134-Tyr, p 140-153 and Tyr-Β190-202) had been synthesized ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/334698a0
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Inhibition of the release of a neutrophil chemotactic factor from macrophages partially explains the anti-inflammatory action of glucocorticoids (1986)
    Springer
    Inflammation research 17 (1986), S. 314-317 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01982632
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Neutrophil migration induced by inflammatory stimuli is reduced by macrophage depletion (1988)
    Springer
    Inflammation research 24 (1988), S. 377-380 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Previous experiments of our group have shown that neutrophil migration induced by inflammatory stimuli is reduced by agents which block the release from macrophages of a specific factor for neutrophil migration (MNCF, [1, 2]). The present paper evaluated the influence of macrophage depletion induced by lavage of the peritoneal cavity on neutrophil migration. In both normal and thioglycollatestimuled peritoneal cavities, lavage with saline reduced the resident macrophage population by about 80% and significantly blocked neutrophil migration induced by inflammatory stimuli such as carrageenin, zymosan andE. coli endotoxin. Peritoneal lavage, however, did not affect neutrophil migration induced by MNCF. Thus, these results support the suggestion that macrophages participate in the control of neutrophil migration induced by acute inflammatory stimuli.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02028296
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    Paper (German National Licenses)
    Fulltext
  • 4
    Electronic Resource
    Electronic Resource
    Blockade by antimacrophage serum of the migration of PMN neutrophils into the inflamed peritoneal cavity (1985)
    Springer
    Inflammation research 17 (1985), S. 97-103 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The effect of rat antimacrophage serum (rAMS) was tested on the influence of normal or thioglycollate-stimulated macrophage populations of the rat peritoneal cavity on the migration of polymorphonuclear neutrophils (PMN) induced by carrageenin, heterologous serum (rabbit) and sheep red blood cells. The rAMS used did not cross-react with PMN or lymphocytes nor did it affect circulating white cells, complement levels or lysed PMN present in the inflammatory exudate. It did, however, give a positive immunofluorescence reaction with resident and stimulated macrophages. The rAMS inhibited macrophage function as tested by sheep red blood cell phagocytosisin vivo and release of a PMN chemotactic factor(s)in vitro. Thioglycollate-stimuated peritoneal cavities showed an increased macrophage population and responded with increased PMN migration when challenged with heterologous serum or carrageenin, as compared with control rats. The presence of rat antimacrophage antibodies inhibited PMN migration induced by heterologous serum, sheep red blood cells and carrageenin. It is concluded that resident macrophages participate in the control of PMN migration to the site of an acute inflammation by acting as ‘alarm cells’ and triggering several defence mechanisms which ultimately protect the host from injurious stimuli.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01966691
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Failure of PAF-acether to inducein vivo neutrophil migration (1988)
    Springer
    Inflammation research 25 (1988), S. 191-194 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The ability of PAF-acether (platelet activating factor, 1-alkyl-2-acetyl-sn-glycero-3-phosphocholine) to induce neutrophil migration was tested in the peritoneal cavities of rats and guinea-pigs and in rat skin air pouches. PAF-acether, at doses of 2 and 5 μg per animal, did not promote significant neutrophil migration. The ability of PAF-acether antagonists to inhibit carrageenin induced neutrophil migration in rat abdominal cavities was also investigated. Carrageenin is a substance described to cause the release of endogenous PAF-acether. Neutrophil migration was not affected by PAF-antagonists. These observations challenge the possible role of PAF-acether as a neutrophil chemotactic factorin vivo.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01969111
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    Paper (German National Licenses)
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