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  • 1
    Electronic Resource
    Electronic Resource
    The lipopigments in human brain tissue necroses (1980)
    Springer
    Acta neuropathologica 52 (1980), S. 141-145 
    Details
    ISSN: 1432-0533
    Keywords: Brain tissue necrosis ; Phagocytic pigments ; Ceroid ; Pigment maturation ; Histochemistry
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The ceroid pigment of macrophages were subdivided into four different types according to their own color and their ability to stain with luxol fast blue. With reference to this mode a series of more than 200 brain infarcts which had happened 1 day to 56 years before death, was examined systematically. According to the results the ceroid variants do not only succeed one another chronologically but they also represent histochemically successive phases of development which correspond to the progressive auto-oxidation of unsaturated lipids. The occurrence of the different stages is chronologically defined. During this development cortical and subcortical macrophages show different speeds in the ageing process of their pigments. There is no evidence for an essential participation of proteins in the maturation of the ceroid. Not during any phase of development is the reaction pattern identical with that of neuronal and glial lipofuscin.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688012
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Later changes in brain death. Signs of partial recirculation (1983)
    Springer
    Acta neuropathologica 62 (1983), S. 15-23 
    Details
    ISSN: 1432-0533
    Keywords: Brain death ; Ischemic neuronal alterations ; Brain stem ; Meningoencephalitic reaction ; Reperfusion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The varying cell picture of the brain in brain death is impressive. Some authors have interpreted this cell picture as a result of intravital autolysis and others as necrosis, at which the maturation time obviously plays an important part. The following time-dependent cerebral changes were established on the basis of an evaluation of 190 brain death cases: (1) neuronal necroses that arise at different rates within the cerebral cortex and the lower brain stem; (2) a hemorrhagic-meningoencephalitic reaction that occurs exclusively at least 4 days after brain death or hemorrhages alone after intervals of at least 48 h; and (3) a washed-out tissue picture. The alterations in the spinal border zone of the total infarction, like in the brain itself, increase rapidly after 48 h. The regular onset of inflammatory alterations after long brain death intervals can only be explained by partial recirculation due to a decline of the high intracranial pressure. The hermorrhages and increasing necroses in some cases with longer intervals therefore are likewise evidence of a not entirely complete cerebral ischemia in spite of an angiographically demonstrable circulatory arrest.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00684915
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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