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  • 65F  (1)
  • DNA damage  (1)
  • Finite-element analysis  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Sensors and Actuators A: Physical 44 (1994), S. 145-149 
    ISSN: 0924-4247
    Keywords: Die-mount materials ; Finite-element analysis ; Silicon pressure sensors ; Thermal stresses
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Electrical Engineering, Measurement and Control Technology
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Springer
    Computing 57 (1996), S. 245-253 
    ISSN: 1436-5057
    Keywords: 68Q20 ; 90C39 ; 65F ; Semigroup computation ; broadcasting ; mesh-connected computers with segmented buses ; reconfigurable buses ; parametric parallel algorithm
    Source: Springer Online Journal Archives 1860-2000
    Topics: Computer Science
    Description / Table of Contents: Zusammenfassung Wir stellen einen Algorithmus für die Halbgruppenberechnung auf Gittern mit rekonfigurierbaren Bussen (MRB) vor. Mitn Operanden kann unser Algorithmus in $$O(2^{(2c^2 + 3c)/(4c + 1)} n^{1/(8c + 2)} )$$ Zeit ausgeführt werden, wennn Prozessoren in einem $$2^{(c^2 - c)/(8c + 2)} n^{1/(5c + 1)/(8c + 2)} \times 2^{(c - c^2 )/(8c + 2)} n^{(3e + 1)/(8c + 2)} $$ MRB-Gitter zur Verfügung stehen. Dabei gilt $$0 \leqslant c \leqslant O(\sqrt {\log _2 n} )$$ . Fürc=0 bedeutet dies $$O(\sqrt n )$$ Zeit für den $$\sqrt n \times \sqrt n $$ MRB und stimmt mit dem Wert für Gitter ohne Busse überein; fürc=1 ist nurO(n 1/10) Zeit für einenn 3/5×n 2/5 MRB erforderlich, was mit einem früheren Ergebnis für Berechnung auf Gittern mit mehreren segmentierten Bussen übereinstimmt; fürc=2 istO(n 1/18) Zeit für einen 21/9 n 11/18×2−1/9 n 7/18 MRB erforderlich; für $$c = O(\sqrt {\log _2 n} )$$ istO(log2 n) Zeit erforderlich, was auch mit dem früheren Ergebnis über MRB übereinstimmt. Unsere Resultate können also als vereinheitlichte Darstellung der bekanntesten Ergebnisse bei verschiedenen Modellen des Parallel-Computing angesehen werden.
    Notes: Abstract In this paper, we present a new parametric parallel algorithm for semigroup computation on mesh with reconfigurable buses (MRB). Givenn operands, our parallel algorithm can be performed in $$O(2^{(2c^2 + 3c)/(4c + 1)} n^{1/(8c + 2)} )$$ , time on a $$2^{(c^2 - c)/(8c + 2)} n^{(5c + 1)/(8c + 2)} \times 2^{(c - c^2 )/(8c + 2)} n^{(3c + 1)/(8c + 2)} $$ MRB ofn processors, where $$0 \leqslant c \leqslant O(\sqrt {\log _2 n} )$$ . Specifically, whenc=0, it takes $$O(\sqrt n )$$ time on the $$\sqrt n \times \sqrt n $$ MRB and is equal to the result on the mesh-connected computers; whenc=1, it takesO(n 1/10) time on then 3/5×n 2/5 MRB and is equal to the previous result on the mesh-connected computers with segmented multiple buses; whenc=2, it takesO(n 1/18) time on the 21/9 n 11/18×2(−1/9) n 7/18 MRB; when $$O(\sqrt {\log _2 n} )$$ , it takesO(log2 n) time and is equal to the previous result on the MRB. Consequently, our results can be viewed as a unification of some best known results on different parallel computational models.
    Type of Medium: Electronic Resource
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  • 3
    ISSN: 1617-4623
    Keywords: Key words Yeast ; Cell Cycle ; Checkpoints ; DNA damage
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract Chk1 is an evolutionarily conserved protein kinase that plays an essential role in mediating G2 arrest in response to DNA damage in Schizosaccharomyces pombe and human cells. It functions by maintaining the inhibition (by phosphorylation of a specific tyrosine residue) of the cyclin-dependent kinase Cdc2 that initiates the G2/M transition. Here, we characterize a structural homologue of Chk1 in the budding yeast Saccharomyces cerevisiae. In this organism, G2/M arrest following DNA damage is considered to be independent of tyrosine phosphorylation of the Cdc2 homologue Cdc28. Nevertheless, a partial defect in G2/M-phase arrest following treatment with ionizing radiation, but not UV radiation, is associated with deletion of CHK1. The fact that such an effect remains detectable in cells synchronized with the microtubule inhibitor nocodazole prior to γ irradiation implies the existence of a CHK1-dependent checkpoint in M phase. We conclude from epistasis analysis that Chk1 participates in the Pds1-dependent subpathway of M-phase arrest. In spite of the partial checkpoint defect of the chk1 mutant, the survival of colony-forming cells is not notably decreased following UV and γ irradiation. In two-hybrid screens, we identified a heme-binding stress protein (encoded by the yeast ORF YNL234W), a protein involved in genomic silencing (Sas3) and Chk1 itself as interacting partners of Chk1.
    Type of Medium: Electronic Resource
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