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  • 1
    Electronic Resource
    Electronic Resource
    Influence of collateral blood flow and of variations in MVO"2 on tissue-ATP content in ischemic and infarcted myocardium
    Amsterdam : Elsevier
    Journal of Molecular and Cellular Cardiology 19 (1987), S. 19-37 
    Details
    ISSN: 0022-2828
    Keywords: Adenine nucleotides ; Collateral flow ; Infarct size ; Ischemia ; MVO"2
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/S0022-2828(87)80542-4
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Effect of intraaortic balloon counterpulsation (IABP) on myocardial infarct size and collateral flow in an experimental dog model (1982)
    Springer
    Intensive care medicine 8 (1982), S. 131-137 
    Details
    ISSN: 1432-1238
    Keywords: Infarct size ; Intraaortic balloon pumping ; Collateral flow
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract To determine the influence of IABP on infarct size and collateral blood flow in each of 12 openchest anaesthetised mongrel dogs two small branches of the left coronary artery were occluded consecutively. The perfusion areas of both branches were comparable in size. IABP was started immediately before ligation of the first branch for a 90-min period followed by a reperfusion period of 90 min. Subsequently the second vessel was also occluded for 90 min as a control without IABP while myocardial oxygen consumption remained constant and was then reperfused. Infarct size was expressed as a percentage of the perfusion area. A difference in infarct size with and without IABP (18±17, 18±10% respectively) could not be observed. However a significant increase of collateral blood flow due to IABP in the subendocardial layer from 8.9±4.8 to 14.9±4.6 ml/100 g/min (p〈0.05) was prevalent. In the subepicardial layer the augmentation from 23.7±19.9 to 26.9±15.2 was not significant. Thus, in spite of a small increase of collateral blood flow in the subendocardial layer of the ischemic myocardium the infarct size was not reduced by IABP in our dog model.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01693432
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Ischemic preconditioning, remembrances of things past and future (1996)
    Springer
    Basic research in cardiology 91 (1996), S. 8-11 
    Details
    ISSN: 1435-1803
    Keywords: Adenosine ; Insulin-like growth factor ; heatshock proteins ; K+-Channel
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Conclusion Myocardial protection by ischemic preconditioning is probably caused by more than one mechanism, one of which involves most certainly an inhibitory G-protein and opening of a K+-channel. Additionally, a wide-ranging reprograming of the cellmembrane involving ion-channels, receptors, ligands and the translocation of cytosolic enzyme systems is probably necessary to achieve myocardial protection.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00788852
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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