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  • Atrophy, neurogenic  (1)
  • Central nervous conduction  (1)
  • Diagnosis  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Choline acetyltransferase activity in human muscular diseases (1976)
    Springer
    Journal of neurology 211 (1976), S. 189-192 
    Details
    ISSN: 1432-1459
    Keywords: Muscle enzymes ; Cholineacetyltransferase ; Muscular dystrophy ; Atrophy, neurogenic
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Zusammenfassung Die Cholinacetyltransferase-Aktivität wurde in Muskeln von Patienten mit Neuromyopathien gemessen. Die Muskelbiopsien wurden bei Patienten mit Myasthenia gravis, mit verschiedenen neurogenen Atrophien der Muskeln und mit Muskeldystrophie vom geschlechtsgebundenen Typus entnommen. Die CAT-Aktivität ist bei den Muskeldystrophien sehr nennenswert und signifikant vermindert (−48%), während keine statistisch signifikante Verminderung bei der Myasthenie nachweisbar ist. Bei den neurogenen Atrophien ist die Verminderung der CAT-Aktivität parallel zum Ausmaß der Erkrankung. Es wird auf die möglichen Zusammenhänge zwischen der CAT-Aktivitätsverminderung im Muskel und dem neurogenen Prozeß hingewiesen.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00313362
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Correlation between multimodal evoked potentials and magnetic resonance imaging in multiple sclerosis (1989)
    Springer
    Journal of neurology 236 (1989), S. 4-8 
    Details
    ISSN: 1432-1459
    Keywords: Multiple sclerosis ; Diagnosis ; Electrophysiological tests ; Magnetic resonance imaging
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Sixty multiple sclerosis (MS) patients (33 definite, 13 probale and 14 suspected were investigated by computed tomography (CT), magnetic resonance imaging (MRI), multimodality evoked potentials (EPs) and cerebrospinal fluid (CSF) electrophoresis. MRI abnormalities were found in 50 cases, while at least one abnormal evoked potential was detected in each of 52 cases. Brain-stem auditory evoked potentials were more sensitive than MRI for the detection of brainstem involvement. All the patients with oligoclonal bands had abnormal MRI and none of the patients with normal MRI had oligoclonal bands in the CSF. The number and the extent of MRI lesions were significantly correlated with the duration of disease and with the degree of disability. Our observations stress the importance of the combined use of MRI and EPs in detecting silent CNS lesions in MS patients.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00314209
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Effects of hyperglycaemia on visual evoked potentials in insulin-dependent diabetic patients (1992)
    Springer
    Acta diabetologica 29 (1992), S. 34-37 
    Details
    ISSN: 1432-5233
    Keywords: Visual evoked potentials ; Hyperglycaemic clamp ; Central nervous conduction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Multimodality evoked potentials frequently reveal subclinical involvement of the central nervous system in patients with insulin-dependent diabetes mellitus. We devised this study to evaluate the possible effects of acute hyperglycaemia on visual evoked potential (VEP) parameters in type 1 diabetic patients. A hyperglycaemic clamp (250 mg/dl for 180 min) was performed in ten patients. Monocular pattern reversal VEPs (check size 15′, contrast 50%) were recorded before, and every 30 min after the start of the clamp. Basal VEP latencies and amplitudes were normal bilaterally in nine patients. No significant changes in pattern reversal and flash VEP parameters were observed after the induction or during the clamp period. None of the neurophysiological parameters evaluated during the test was related to the duration of the disease, the basal VEP latency or amplitude or the presence of retinopathy. Our data suggest that the neurophysiological abnormalities detected in insulin-dependent diabetic patients are due to structural involvement of the central nervous pathways and not to functional damage induced by acute short-term hyperglycaemia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00572827
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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