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  • Contact site  (1)
  • Dermatophytosis  (1)
  • Key words. Transcription; protein degradation; ubiquitin; SUMO-1.  (1)
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  • 1
    Electronic Resource
    Electronic Resource
    Cross-linking analysis of yeast mitochondrial outer membrane
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Biomembranes 860 (1986), S. 690-698 
    Details
    ISSN: 0005-2736
    Keywords: (Yeast) ; Contact site ; Cross-linking ; Glutathione ; Hexokinase ; Oligomerization ; Outer membrane ; Pore protein ; transferase
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine , Physics
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0005-2736(86)90568-7
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Regulation of transcription factors by protein degradation (2000)
    Springer
    Cellular and molecular life sciences 57 (2000), S. 1207-1219 
    Details
    ISSN: 1420-9071
    Keywords: Key words. Transcription; protein degradation; ubiquitin; SUMO-1.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract. The level of transcription factors is tightly controlled by their rates of synthesis and degradation. Many critical factors are maintained at an appropriate level by targeted addition of ubiquitin and degradation via the proteasome. Whereas ubiquitination targets modified proteins for degradation, modification of substrates by the family of ubiquitin-like proteins does not target the proteins for degradation but can alter the stability and other properties of the modified proteins. Here we discuss the elaborate mechanisms that have evolved to allow specific recognition of substrates targeted for modification. Specific examples are discussed to illustrate the different mechanisms involved and the importance of regulated degradation in diseases such as cancer.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/PL00000760
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Genetic susceptibility to dermatophytosis (1992)
    Springer
    European journal of epidemiology 8 (1992), S. 346-349 
    Details
    ISSN: 1573-7284
    Keywords: Epidemiology ; Dermatophytosis ; Genetic susceptibility
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Clustering of cases of dermatophytosis suggests that inherited susceptibility may play a part in determining the epidemiology of some forms of this infection, notably tinea imbricata. Some studies of T. concentricum infection show that autosomal recessive susceptibility may provide an answer although this is not the case in all endemic areas. Further support comes from the association between dermatophytosis in man and inherited conditions such as atopy, chronic mucocutaneous candidosis and tylosis as well as experimental data showing that susceptibility to dermatophytosis in mice varies in different inbred strains. Possible mechanisms are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00158566
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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