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  • 1
    Electronic Resource
    Electronic Resource
    Annulate lamellae in prolactin-secreting pituitary adenomas (1983)
    Springer
    Acta neuropathologica 61 (1983), S. 10-14 
    Details
    ISSN: 1432-0533
    Keywords: Annulate lamellae ; Prolactin ; Prolactinoma ; Pituitary neoplasma ; Pituitary gland
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Electron-microscopic examination revealed the presence of annulate lamellae in four of five prolactinomas. All the patients examined were 24–30-year-old women who had suffered from amenorrhea and galactorrhea for 3–5 years. Serum levels of prolactin were 114–4,700 ng/ml. The tumors removed by a transsphenoidal approach were 4–23 mm in diameter and showed the ultrastructural features of sparsely granulated prolactinoma. Analysis in correlation with clinical and laboratory data revealed that the annulate lamellae were found in rapidly growing prolactinomas. This is the fifth report on pituitary adenomas with annulate lamellae.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688380
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    An interaction between glucose and estrogen in gastric acid secretion in the lateral hypothalamic area of female rats (1994)
    Springer
    Experimental brain research 102 (1994), S. 171-174 
    Details
    ISSN: 1432-1106
    Keywords: Estrogen ; Gastric acid secretion ; Glucose ; Hypothalamus ; Rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Gastric acid outputs caused by glucose injection into the lateral hypothalamic area (LHA) were examined in insulin hypoglycemia with or without estradiol-17β (EST) administration in bilaterally ovariectomized (OV) female rats. The basal level of acid output was higher in OV rats without EST than in OV rats with EST. When acid response was expressed as the percentage change, glucose injection into the LHA decreased acid output in a dose-dependent fashion in OV rats, while, in OV rats with EST, glucose injection into the LHA also reduced acid output without dose dependency. It was also noted that the threshold concentration of glucose that induced an acid response was lower in OV rats without EST than in OV rats with EST. These findings suggest that glucose-sensitive neurons responsible for gastric acid secretion can be modulated by estrogen at the LHA level.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00232450
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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