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  • 1
    Electronic Resource
    Electronic Resource
    Magnetic properties of FeAlMnC steels (1990)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 67 (1990), S. 4835-4837 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: A series of high yielding strength (90–180 ksi) and high-elongation (30%–45%) FeAlMnC steels have been made. The magnetic, microstructural, and thermal expansion properties of these steels have been studied. Basically, their magnetic transitions on cooling can be classified into three groups according to their microstructures: (i) For fully austenitic (γ) steels, the transition is from paramagnetic to antiferromagnetic. TN is lowered with the addition of Al; (ii) for α+γ phase steel (volume fraction of α≤0.3%), the transition is from superparamagnetic to antiferromagnetic, and (iii) for the mixed phase steel, whose α phase has percolated, it is ferromagnetic with TC(approximately-equal-to)200 C. The susceptibility of austenitic steels is low. Their nonmagnetic properties are comparable to commercial 304 or 25/12 stainless steel. An Invar-like property in the thermal expansion was observed around TN. Their volume magnetostriction values are in the range of 10−6–10−5.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.346053
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Critical behavior of amorphous Fe80−xMxB14Si6 alloys with M=Mo, V, Mn, and 2≤x≤14 (1990)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 67 (1990), S. 5439-5441 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: Amorphous Fe80−xMxB14Si6 alloys with M=Mo, V, Mn, and 2≤x≤14 have been made by the rapid quenching method. The critical exponents β, γ, δ, and Tc were determined by two independent methods: the method of Kouvel-Fisher and the scaling plot. With the addition of M, β, and γ deviate more from the values predicted by the three-dimensional Heisenberg model, and are closer to the values proposed by Sobotta and Wagner. δ remains less changed. The magnetic inhomogeneity of these alloys is evidenced by the specific heat, the forced magnetoresistance, and the Mössbauer measurements. By increasing x, the peak of specific heat becomes smeared and less divergent, and forced magnetoresistance changes from negative to positive around Tc.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.346103
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Measurement of temperature gradient in diode-laser-pumped high-power solid-state laser by low-coherence reflectometry (1998)
    Woodbury, NY : American Institute of Physics (AIP)
    Applied Physics Letters 73 (1998), S. 3342-3344 
    Details
    ISSN: 1077-3118
    Source: AIP Digital Archive
    Topics: Physics
    Notes: A noninvasive technique was developed to measure the temperature distribution in laser gain medium. Both axial and radial temperature distributions of a diode-laser-pumped and intracavity frequency-doubled high-power Nd:YVO4/KTP laser were characterized using a high dynamic range, low-coherence reflectometer. This measurement is important to the design of high-power solid-state lasers both in terms of mode matching and laser-rod doping selection. © 1998 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.122763
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  • 4
    Electronic Resource
    Electronic Resource
    Genetic analysis of nitric oxide synthase isoforms: targeted mutation in mice (1996)
    Springer
    Journal of molecular medicine 74 (1996), S. 415-421 
    Details
    ISSN: 1432-1440
    Keywords: Key words Nitric oxide ; Nitric oxide synthase ; Targeted disruption ; Animal models ; Homologous recombination
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract  Since the discovery that nitric oxide is an endogenous vasodilator responsible for endothelium-derived relaxing factor activity, nitric oxide has been found in many different cell types and implicated in many diverse biological processes. Because pharmacological blockade does not distinguish between the three major isoforms of nitric oxide synthase, the tissue and enzyme source of nitric oxide is unclear in many situations. Targeted disruption of the genes for the various isoforms of nitric oxide synthase offers a useful genetic approach to study the roles of each isoform and to examine the effects of their deletion on physiological processes in intact animals. Here we review the phenotypes of the various nitric oxide synthase mutant mice and examine what they reveal about the complexities of the nitric oxide signaling system and about molecular and physiological compensations brought into play in the absence of individual isoforms.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001090050043
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Genetic analysis of nitric oxide synthase isoforms: targeted mutation in mice (1996)
    Springer
    Journal of molecular medicine 74 (1996), S. 415-421 
    Details
    ISSN: 1432-1440
    Keywords: Nitric oxide ; Nitric oxide synthase ; Targeted disruption ; Animal models ; Homologous recombination
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Sine the discovery that nitric oxide is an endogenous vasodilator responsible for endothelium-derived relaxing factor activity, nitric oxide has been found in many different cell types and implicated in many diverse biological processes. Because pharmacological blockade does not distinguish between the three major isoforms of nitric oxide synthase, the tissue and enzyme source of nitric oxide is unclear in many situations. Targeted disruption of the genes for the various isoforms of nitric oxide synthase offers a useful genetic approach to study the roles of each isoform and to examine the effects of their deletion on physiological processes in intact animals. Here we review the phenotypes of the various nitric oxide synthase mutant mice and examine what they reveal about the complexities of the nitric oxide signaling system and about molecular and physiological compensations brought into play in the absence of individual isoforms.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00217517
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    Paper (German National Licenses)
    Fulltext
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