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  • 1
    Electronic Resource
    Electronic Resource
    Teratogenic Risks of Drug Synergism (1964)
    [s.l.] : Nature Publishing Group
    Nature 203 (1964), S. 527-528 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] In the present enquiry we set out to determine if two compounds, which lead to related malformations when administered singly and in the requisite dosages, will interfere with normal development when individually harmless amounts are used in combination. The compounds selected for these experiments ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/203527a0
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Uncouplers of Oxidative Phosphorylation and Teratogenic Activity of Insulin (1964)
    [s.l.] : Nature Publishing Group
    Nature 204 (1964), S. 285-286 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] When we reported these observations, we suggested that uncoupling of oxidative phosphorylation by chlorpromazine might well play an important part in bringing about the teratogenic synergism which follows combined administration of the two compounds. This seemed the more suggestive since Handle and ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/204285a0
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Interaction of Insulin and Chlorpromazine in Teratogenesis (1963)
    [s.l.] : Nature Publishing Group
    Nature 198 (1963), S. 215-216 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] The highly efficient protection which nicotinamide provides in 4- and 5-day embryos against insulin damage suggested that in these developmental stages disruption of oxidative phosphorylation may play an important part in the origin of malformations. This interpretation is supported by evidence ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/198215b0
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Independent segregation of the plastid genome and cytoplasmic male sterility in Petunia somatic hybrids (1985)
    Springer
    Molecular genetics and genomics 199 (1985), S. 440-445 
    Details
    ISSN: 1617-4623
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Summary The chloroplast genomes of three sets of Petunia somatic hybrids were analyzed to examine the relationship between chloroplast DNA (cpDNA) composition and cytoplasmic male sterility (CMS). Chloroplast genomes of somatic hybrid plants were identified either by restriction and electrophoresis of purified cpDNAs or by hybridization of total DNA digests with cloned cpDNA probes that distinguish the parental genomes. The chloroplast genomes of a set of seven somatic hybrids derived from the fusion of Petunia CMS line 2423 and fertile line 3699 were analyzed. All seven plants were fertile, and all exhibited the cpDNA restriction pattern of the sterile cytoplasm. Similarly, four fertile somatic hybrids derived from the fusion of CMS line 3688 and fertile line 3677 were found to contain the CMS chloroplast genome. The cpDNA compositions of four fertile and two sterile somatic hybrids derived from the fusion of CMS line 3688 and fertile line 3704 were determined by restriction analysis of purified cpDNAs; all six plants exhibited the cpDNA restriction pattern of line 3704. Thus the CMS phenotype segregates independently of the chloroplast genome in Petunia somatic hybrids, indicating that CMS in Petunia is not specified by the chloroplast genome.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00330756
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    Paper (German National Licenses)
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