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  • 1
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Applied Animal Behaviour Science 31 (1991), S. 147-156 
    ISSN: 0168-1591
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Applied Animal Behaviour Science 21 (1988), S. 307-313 
    ISSN: 0168-1591
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
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  • 3
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Applied Animal Behaviour Science 33 (1992), S. 149-163 
    ISSN: 0168-1591
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology
    Type of Medium: Electronic Resource
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  • 4
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Applied Animal Behaviour Science 33 (1992), S. 149-163 
    ISSN: 0168-1591
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
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  • 5
    Electronic Resource
    Electronic Resource
    Springer
    Archives of virology 39 (1972), S. 248-256 
    ISSN: 1432-8798
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Domestic fowl were exposed to an aerosol of infectious bronchitis virus. Birds were killed at intervals after infection and sections of trachea were examined in the electron microscope. The virus replicated in superficial cells,i.e., ciliated epithelial cells and mucus cells. Both these cell types were desquamated over large areas but the underlying basal cell layer was not significantly damaged. At an early stage of replication, virus particles were confined to a small vacuolated region of cytoplasm but in desquamated cells the whole cytoplasm was vacuolated and the vacuoles contained virus particles. Virus was not found in the nucleus and all cytoplasmic particles were within vacuoles. Virus formed by budding at vacuole membranes and the vacuoles involved were derived from both Golgi vesicles and endoplasmic reticulum. The virus probably has a linear internal component and is limited by a trilaminar membrane bearing projections which tend to stain lightly. Regeneration of morphologically normal tracheal epithelium occurred between 6 and 7 days after inoculation but a second wave of virus replication in the trachea was not detected although the virus could be recovered from inoculated birds for up to 14 days following inoculation.
    Type of Medium: Electronic Resource
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  • 6
    Electronic Resource
    Electronic Resource
    Springer
    Archives of virology 39 (1972), S. 369-375 
    ISSN: 1432-8798
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The replication of myxoma virus was examined using thin sections of rabbit skin. The virus is assembled in the cytoplasm as crescent shaped structures which eventually form complete immature particles. These are roughly spherical, contain granular material and are limited by a unit membrane bearing spicules on its outer surface. The contents of the immature particles condense and most of the granular material is incorporated into a dumbell shaped core which has 3 electron dense layers. The residual material is compressed by the collapse of the limiting layers and this forms the lateral bodies of mature virus particles. The cytoplasm of infected cells is frequently interspersed with small vacuoles which do not contain virus precursor material but do contain one or a few virus particles. Particles enter the vacuoles by budding and thus gain an extra envelope. The budding process may be initiated by cytoplasmic particles whether or not they are mature. Although virus could, in principle, be released from the cell by fusion of virus containing vacuoles with the plasmic membrane, we found no evidence for this and conclude that most virus is released following cell disruption. Conventional microscopy of rabbit epithelial cells showed that myxoma virus forms only a few. poorly developed cytoplasmic inclusions. This is apparently in marked contrast to the classical inclusions formedin vivo following infection with pigeonpox virus, however in thin section the basic processes of virus morphogenesis and budding are similar if not identical.
    Type of Medium: Electronic Resource
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