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  • 1
    Electronic Resource
    Electronic Resource
    Theory of local optical spectroscopy of quantum wires with interface fluctuations (2002)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 91 (2002), S. 2302-2307 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: We present a theory of local optical spectroscopy in quantum wires taking into account structural disorder. The calculated spatially resolved spectra show the individual spectral lines due to the exciton states localized by the disordered potential in agreement with experimental findings. We investigate systematically the influence of the potential profile and of the spatial resolution on the local optical spectra. Several line scans along the wire axis are obtained for different spatial correlations and strength of the disorder potential and for different spatial resolutions ranging from the subwavelength to the diffraction limit. Lowering the spatial resolution causes the disappearance of many spectral lines due to destructive spatial interference. However, our results show that information on the individual eigenstates of this quasi one-dimensional quantum system can be obtained at also resolutions significantly lower than the correlation length of interface fluctuations. © 2002 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.1424049
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  • 2
    Electronic Resource
    Electronic Resource
    Imaging spectroscopy of quantum wells with interfacial fluctuations: A theoretical description (2000)
    Woodbury, NY : American Institute of Physics (AIP)
    Applied Physics Letters 77 (2000), S. 2804-2806 
    Details
    ISSN: 1077-3118
    Source: AIP Digital Archive
    Topics: Physics
    Notes: We present a theoretical approach for the simulation of scanning local optical spectroscopy in disordered quantum wells (QWs). After a single realization of the disorder potential, we calculate spectra on a mesh of points on the QW plane, thus obtaining a three-dimensional matrix of data from which we construct two-dimensional spectroscopic images of excitons laterally localized at interface fluctuations. Our simulations are in close agreement with the experimental findings, and contribute to the interpretation of spatially resolved spectra in QWs. © 2000 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.1322057
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  • 3
    Electronic Resource
    Electronic Resource
    Muscle phosphofructokinase deficiency in a myopathic child with severe mental retardation and aplasia of cerebellar vermis (1992)
    Springer
    Child's nervous system 8 (1992), S. 237-241 
    Details
    ISSN: 1433-0350
    Keywords: Skeletal muscle ; Phosphofructokinase deficiency ; Biochemistry ; Morphology ; Enzyme histochemistry
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Muscle phosphofructokinase (PFK) deficiency in man is responsible for at least two forms of myopathy; one is characterized by painful contractures of muscles and typically occurs in adults, whereas the other is often disabling and typically occurs in childhood, with psychomotor and growth retardation. In this investigation, a young myopathic patient with severe mental retardation and aplasia of the cerebellar vermis presented with muscular hypotrophy of the limbs, generalized hypotonia, convergent strabismus and marked pain during passive movement. Biopsy of quadriceps femoris muscle showed variation in the fiber size with sarcoplasmic areas positive for periodic acid-Schiff stain. Histochemical qualitative reaction for PFK showed no staining of muscle fibers; ultrastructural studies showed abnormal accumulation of glycogen granules in both intermyofibrillar and subsarcolemmal areas. While some enzyme activities in the muscular crude extract were significantly lower than in controls, direct assay of PFK revealed no activity, thus demonstrating that the child's myopathy was due to the lack of PFK activity.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00262858
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  • 4
    Electronic Resource
    Electronic Resource
    The motilin gene: subregional localisation, tissue expression, DNA polymorphisms and exclusion as a candidate gene for the HLA-associated immotile cilia syndrome (1994)
    Springer
    Human genetics 〈Berlin〉 94 (1994), S. 671-674 
    Details
    ISSN: 1432-1203
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract The product of the human motilin gene (MLN) has an important role in regulating gastrointestinal motility. The precise chromosomal localisation and expression of this gene are still unresolved. Here, we report a detailed study assigning MLN to 6p21.3; MLN is tightly linked to the HLA-DQalpha locus. Moreover, MLN expression has been evaluated in a large series of tissues. Positive signals have been obtained for brain, bronchi and a gastrointestinal malignancy. Direct sequencing exon by exon of the codifying region, intron/exon boundaries and promoter has allowed the identification of three DNA polymorphisms, one of which corresponds to a common protein variant. The chromosomal localisation of MLN, and its expression in broncoepithelial cells suggests that this gene is involved in immotile-cilia syndrome (ICS) disease. Sequence and segregation analysis of the MLN gene carried out in two families, in which the disease locus was previously assigned to 6p21.3, exclude MLN as a candidate gene for the HLA-associated form of ICS.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00206962
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  • 5
    Electronic Resource
    Electronic Resource
    HLA haplotype segregation and ultrastructural study in familial immotile-cilia syndrome (1992)
    Springer
    Human genetics 〈Berlin〉 89 (1992), S. 270-274 
    Details
    ISSN: 1432-1203
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The immotile-cilia syndrome (ICS) is a congenital disorder characterized by dysmotility or even complete immotility of the cilia in the ciliated epithelia. The most frequent consequences include recurrent air-way infections from early childhood. Neonatal asphyxia often occurs. Males are usually sterile, whereas females may be fertile or infertile. The disease is inherited as an autosomal recessive trait, but previous attempts to localize the ICS susceptibility gene have so far been unsuccessful. Here, we present the case of two sib pairs affected by ICS from two unrelated families. The electron microscopic investigation of nasal biopsies showed structural anomalies of the cilia, characterized by single microtubules or doublets, arranged randomly in the axoneme. Histocompatibility antigen (HLA)-genotyping of all family members revealed: 1) a significant association of ICS with the HLA-DR7; DQW2 haplotype, which is shared by all the affected sibs (P = 0.0099; RR = 25.94); 2) a possible linkage of the ICS susceptibility gene with HLA, both the affected sibs being HLA-identical, the healthy brother in family B being HLA-different (sibpair analysis: P 〈 0.001).
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00220538
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