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  • 1
    Electronic Resource
    Electronic Resource
    Synthesis and Processing of Improved Organic Second-Order Nonlinear Optical Materials for Applications in Photonics (1995)
    s.l. : American Chemical Society
    Chemistry of materials 7 (1995), S. 1060-1081 
    Details
    ISSN: 1520-5002
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology , Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/cm00054a006
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Thermosetting polyurethanes with stable and large second-order optical nonlinearity (1992)
    s.l. : American Chemical Society
    Macromolecules 25 (1992), S. 4032-4035 
    Details
    ISSN: 1520-5835
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology , Physics
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ma00041a027
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Optical Studies of Infrared Active Defects in Irradiated Si After Annealing at 450°C (1995)
    s.l. ; Stafa-Zurich, Switzerland
    Materials science forum Vol. 196-201 (Nov. 1995), p. 157-162 
    Details
    ISSN: 1662-9752
    Source: Scientific.Net: Materials Science & Technology / Trans Tech Publications Archiv 1984-2008
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Type of Medium: Electronic Resource
    URL: http://www.tib-hannover.de/fulltexts/2011/0528/02/01/transtech_doi~10.4028%252Fwww.scientific.net%252FMSF.196-201.157.pdf
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  • 4
    Electronic Resource
    Electronic Resource
    Effects of Al-C Ion-Implantation and Annealing in Epitaxial 6H-SiC Studied by Structural and Optical Techniques (1998)
    s.l. ; Stafa-Zurich, Switzerland
    Materials science forum Vol. 264-268 (Feb. 1998), p. 693-696 
    Details
    ISSN: 1662-9752
    Source: Scientific.Net: Materials Science & Technology / Trans Tech Publications Archiv 1984-2008
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Type of Medium: Electronic Resource
    URL: http://www.tib-hannover.de/fulltexts/2011/0528/02/03/transtech_doi~10.4028%252Fwww.scientific.net%252FMSF.264-268.693.pdf
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Coupled Stress Release Model for Time-dependent Seismicity (1999)
    Springer
    Pure and applied geophysics 155 (1999), S. 649-667 
    Details
    ISSN: 1420-9136
    Keywords: Key words: Coupled stress release model, AIC criterion, historical earthquake catalog, synthetic catalog.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Geosciences , Physics
    Notes: Abstract —Based on the original stress release model of seismicity proposed by Vere-Jones (1978), this paper has developed a stochastic coupled stress release model of time-dependent seismicity, which considers the earthquake interaction and stress transfer between different seismic subregions. As an example, the model is applied to a statistical analysis of the historical earthquake catalog with magnitude M ≥ 6.0 during the period from 1480 to 1996 in North China. According to the Akaike information criterion (AIC), the results show that the coupled stress release model is better than the original model, which demonstrates the existence of long-range correlations between different seismic subregions. We also apply both the stochastic (original and developed coupled) models to analyze the synthetic catalog produced by a cellular automata model, which is based on mechanics of a slide-spring-damper system to model the fault network. The stress release model provides a good fit to the synthetic regional stress, and the coupled stress release model provides an improvement in fit to the synthetic catalog over the original model.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s000240050281
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  • 6
    Electronic Resource
    Electronic Resource
    Value of antibodies to GAD65 combined with islet cell cytoplasmic antibodies for predicting IDDM in a childhood population (1994)
    Springer
    Diabetologia 37 (1994), S. 917-924 
    Details
    ISSN: 1432-0428
    Keywords: Key words GAD65 antibodies ; islet cell cytoplasmic antibodies ; predictive value ; IDDM in childhood.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The value of a test for islet cell cytoplasmic antibodies together with a test for GAD65 antibodies to predict the subsequent development of diabetes over a period of 11.5 years was assessed in an open childhood population comprising 2,805 individuals. A single serum sample was obtained from each individual between 1975 and 1977 and screened for islet cell cytoplasmic antibodies for which eight individuals were positive (0.29 %). During the average follow-up period of 11.5 years, four of eight islet cell antibody positive and three islet cell antibody negative individuals developed clinical diabetes. Sera from all individuals, who were islet cell antibody positive and/or developed diabetes (total of 11) and from 100 randomly selected control subjects were analysed for GAD65 antibodies. Six of eight islet cell antibody positive individuals were GAD65 antibody positive including all four who subsequently developed IDDM. Furthermore, one of the three islet cell antibody negative individuals who developed IDDM was GAD65 antibody positive both in 1976 and in 1989. Thus, a positive test for GAD65 antibodies alone correctly predicted diabetes in five of seven children, who developed the disease. Only one of the children, who developed diabetes was positive for insulin autoantibodies and this individual was also positive for islet cell cytoplasmic antibodies and GAD65 antibodies. One of the 100 control subjects was positive for GAD65 antibodies (1 %). The results suggest that a single GAD65 antibody test may have a higher sensitivity for predicting IDDM than a test for islet cell cytoplasmic antibodies, but that a combined positive test for both antibodies increases the specificity for predicting IDDM over a period of 11.5 years. [Diabetologia (1994) 37: 917–924]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00400948
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    Paper (German National Licenses)
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  • 7
    Electronic Resource
    Electronic Resource
    DNA fragmentation is an early event in cytokine-induced islet beta-cell destruction (1994)
    Springer
    Diabetologia 37 (1994), S. 733-738 
    Details
    ISSN: 1432-0428
    Keywords: Islet beta cells ; cytokines ; interleukin 1 ; tumour necrosis factor ; interferon gamma ; DNA damage
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The cytokines, interleukin 1, tumour necrosis factor, and interferon gamma are cytotoxic to islet beta cells, however, their mechanisms of beta-cell killing are not fully characterized. Since DNA damage is a mechanism of cytokine-induced cell death in some cell types, we sought evidence for cytotoxic effects of cytokines at a nuclear level in islet beta cells by measuring DNA fragmentation in rat islets and islet beta-cell lines. The individual cytokines, interleukin 1 (10 U/ml), tumour recrosis factor (103 U/ml) and interferon gamma (103 U/ml) inhibited insulin release from rat islets, but did not cause DNA fragmentation or destroy islet cells; by contrast, combination of the three cytokines induced DNA fragmentation and islet-cell death. Cytokine-induced DNA fragmentation preceded cell lysis in islet beta-cell lines (RINm5F, rat insulinoma cells; and NIT-1, NOD/Lt mouse transgenic beta cells), whereas in non-islet cell lines (GH-3, rat pituitary; and PC-12, rat adrenal) the cytokines induced cell lysis and no or late DNA fragmentation. Nicotinamide prevented both DNA fragmentation and destruction of RINm5F islet cells by the cytokines. These findings identify DNA as an early target of cytokine action in islet beta cells, and implicate DNA fragmentation as a mechanism of cytokine-induced beta-cell destruction.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00404328
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    Paper (German National Licenses)
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  • 8
    Electronic Resource
    Electronic Resource
    Value of antibodies to GAD65 combined with islet cell cytoplasmic antibodies for predicting IDDM in a childhood population (1994)
    Springer
    Diabetologia 37 (1994), S. 917-924 
    Details
    ISSN: 1432-0428
    Keywords: GAD65 antibodies ; islet cell cytoplasmic antibodies ; predictive value ; IDDM in childhood
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The value of a test for islet cell cytoplasmic antibodies together with a test for GAD65 antibodies to predict the subsequent development of diabetes over a period of 11.5 years was assessed in an open childhood population comprising 2,805 individuals. A single serum sample was obtained from each individual between 1975 and 1977 and screened for islet cell cytoplasmic antibodies for which eight individuals were positive (0.29%). During the average follow-up period of 11.5 years, four of eight islet cell antibody positive and three islet cell antibody negative individuals developed clinical diabetes. Sera from all individuals, who were islet cell antibody positive and/ or developed diabetes (total of 11) and from 100 randomly selected control subjects were analysed for GAD65 antibodies. Six of eight islet cell antibody positive individuals were GAD65 antibody positive including all four who subsequently developed IDDM. Furthermore, one of the three islet cell antibody negative individuals who developed IDDM was GAD65 antibody positive both in 1976 and in 1989. Thus, a positive test for GAD65 antibodies alone correctly predicted diabetes in five of seven children, who developed the disease. Only one of the children, who developed diabetes was positive for insulin autoantibodies and this individual was also positive for islet cell cytoplasmic antibodies and GAD65 antibodies. One of the 100 control subjects was positive for GAD65 antibodies (1%). The results suggest that a single GAD65 antibody test may have a higher sensitivity for predicting IDDM than a test for islet cell cytoplasmic antibodies, but that a combined positive test for both antibodies increases the specificity for predicting IDDM over a period of 11.5 years.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00400948
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 9
    Electronic Resource
    Electronic Resource
    DNA fragmentation is an early event in cytokine-induced islet beta-cell destruction (1994)
    Springer
    Diabetologia 37 (1994), S. 733-738 
    Details
    ISSN: 1432-0428
    Keywords: Key words Islet beta cells, cytokines, interleukin 1, tumour necrosis factor, interferon gamma, DNA damage.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The cytokines, interleukin 1, tumour necrosis factor, and interferon gamma are cytotoxic to islet beta cells, however, their mechanisms of beta-cell killing are not fully characterized. Since DNA damage is a mechanism of cytokine-induced cell death in some cell types, we sought evidence for cytotoxic effects of cytokines at a nuclear level in islet beta cells by measuring DNA fragmentation in rat islets and islet beta-cell lines. The individual cytokines, interleukin 1 (10 U/ml), tumour recrosis factor (103 U/ml) and interferon gamma (103 U/ml) inhibited insulin release from rat islets, but did not cause DNA fragmentation or destroy islet cells; by contrast, combination of the three cytokines induced DNA fragmentation and islet-cell death. Cytokine-induced DNA fragmentation preceded cell lysis in islet beta-cell lines (RINm5F, rat insulinoma cells; and NIT-1, NOD/Lt mouse transgenic beta cells), whereas in non-islet cell lines (GH-3, rat pituitary; and PC-12, rat adrenal) the cytokines induced cell lysis and no or late DNA fragmentation. Nicotinamide prevented both DNA fragmentation and destruction of RINm5F islet cells by the cytokines. These findings identify DNA as an early target of cytokine action in islet beta cells, and implicate DNA fragmentation as a mechanism of cytokine-induced beta-cell destruction. [Diabetologia (1994) 37: 733–738]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001250050172
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    Paper (German National Licenses)
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  • 10
    Electronic Resource
    Electronic Resource
    Evaluation of Biaxial Forming Limits by Bulge Testing (2006)
    s.l. ; Stafa-Zurich, Switzerland
    Materials science forum Vol. 519-521 (July 2006), p. 117-124 
    Details
    ISSN: 1662-9752
    Source: Scientific.Net: Materials Science & Technology / Trans Tech Publications Archiv 1984-2008
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Finite element modelling of sheet-forming operations, such as pressure-ram-forming,(PRF™) requires knowledge of forming limits under biaxial strain conditions. In this work,elliptical bulge tests have been used to evaluate the forming limits of an aluminum bodystock alloy,X309, that is used for PRF™ applications. Limiting dome heights have been determined as afunction of pressure-rate and temperature. All tests have been done with the rolling direction, RD,of the sheet aligned with the major axis of the bulge
    Type of Medium: Electronic Resource
    URL: http://www.tib-hannover.de/fulltexts/2011/0528/02/13/transtech_doi~10.4028%252Fwww.scientific.net%252FMSF.519-521.117.pdf
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