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  • 1980-1984  (3)
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Material
Years
Year
  • 1
    Electronic Resource
    Electronic Resource
    Inhibition of brain cholinesterase activity in forest birds and squirrels exposed to aerially applied acephate (1980)
    Springer
    Bulletin of environmental contamination and toxicology 24 (1980), S. 676-683 
    Details
    ISSN: 1432-0800
    Source: Springer Online Journal Archives 1860-2000
    Topics: Energy, Environment Protection, Nuclear Power Engineering , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01608173
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Prey-capturing ability of American kestrels fed DDE and acephate or acephate alone (1984)
    Springer
    Archives of environmental contamination and toxicology 13 (1984), S. 367-372 
    Details
    ISSN: 1432-0703
    Source: Springer Online Journal Archives 1860-2000
    Topics: Energy, Environment Protection, Nuclear Power Engineering , Medicine
    Notes: Abstract American kestrels (Falco sparverius) were given a single acute dose of the insecticide acephate (50 mg/kg), either alone or superimposed on a moderate background level of DDE (35 ppm wet-weight in carcass homogenates). The combined DDE-acephate treatment was chosen to resemble exposure conditions for wild avian predators whose tissues may contain appreciable sublethal accumulations of organochlorine insecticides. Acephate produced similar cholinesterase (ChE) depression in both groups (39% median depression of serum ChE, 25% median brain ChE depression). Predatory vigilance and attack behavior, measured by frequency and speed of responses to a familiar moving prey model, were not altered by acephate administration in either group. Neither DDE nor acephate at these low dosages has appreciable effects on kestrels' responses to a prey stimulus with which they have had extensive prior contact.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01055288
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Toxicity of acephate and methamidophos to dark-eyed juncos (1981)
    Springer
    Archives of environmental contamination and toxicology 10 (1981), S. 185-192 
    Details
    ISSN: 1432-0703
    Source: Springer Online Journal Archives 1860-2000
    Topics: Energy, Environment Protection, Nuclear Power Engineering , Medicine
    Notes: Abstract The calculated, acute oral LD50 of acephate and methamidophos to dark-eyed juncos (Junco hyemalis) was 106 mg/kg and 8 mg/kg, respectively. Brain cholinesterase (ChE) activity in birds that died after acephate poisoning was depressed 80% below that of control birds. Birds that died of acute methamidophos poisoning had brain ChE depression of 60%. The birds killed by acephate had brain acephate residue concentrations greater than 2 mg/kg and methamidophos concentrations usually greater than 0.25 mg/kg. Eighty percent of the birds killed with methamidophos had brain methamidophos concentrations greater than 0.1 mg/kg. The five-day feeding LC50 for acephate was 1485 mg/kg. Brain ChE activities of birds which died early in the study were less depressed (51.5%) than those which died at a later date (69.6%). Brain residues of acephate and methamidophos were lower in these birds than in the birds of the acute oral LD50 studies. Brain ChE activity returned to normal within three days after the birds received a single sublethal dose of acephate. These studies indicate that the amount of acephate needed to produce the ChE depression found in other investigations in most dark-eyed juncos exposed to forest applications of insecticide is about one-fifth of the LD50; however, in a few birds the ChE activity may be depressed to near lethal levels.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01055620
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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