Bibliothek

feed icon rss

Ihre E-Mail wurde erfolgreich gesendet. Bitte prüfen Sie Ihren Maileingang.

Leider ist ein Fehler beim E-Mail-Versand aufgetreten. Bitte versuchen Sie es erneut.

Vorgang fortführen?

Exportieren
  • 1
    ISSN: 1432-0630
    Schlagwort(e): 74.70.Vy ; 74.30.Ci ; 74.60.Jg
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Maschinenbau , Physik
    Notizen: Abstract Finely ground powders ofRBa2Cu3O7 − δ (R=Y, Nd, Sm, Eu, Dy, Ho, Er, Tm, Yb) have been mixed at dilute 3%-by-volume concentrations into epoxy matrices which were then allowed to harden in applied magnetic fieldsH A=18 kOe. X-ray diffractometry studies and 4.3 K measurements of supercurrent-induced magnetization hysteresisΔM are interpreted as indicating at least partial alignment of single-crystal-grain c-axes (1) parallel toH A forR=Y, Nd, Sm, Dy, Ho (as earlier found by Farrell et al. forR=Y), and (2) perpendicular toH A forR=Eu, Er, Tm, Yb. With a few exceptions (Y, Sm, Eu) the alignment direction correlates with the sign of the second-order Stevens factorα J of the crystalline electric field Hamiltonian in the manner suggested by Livingston et al. For the best aligned specimens (Ho, Dy) critical current densitiesJ c (4.3 K, 5 kOe) for individual grains are estimated fromΔM and the Bean model to be of order 107 A/cm2 for the measuring fieldH parallel to the original alignment fieldH A, and of order 106 A/cm2 forH perpendicular toH A.
    Materialart: Digitale Medien
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
    BibTip Andere fanden auch interessant ...
  • 2
    Digitale Medien
    Digitale Medien
    Springer
    Clinical and experimental nephrology 4 (2000), S. 24-28 
    ISSN: 1437-7799
    Schlagwort(e): Key words Uranyl acetate ; ARF ; Glycine ; Apoptosis ; Tubular damage
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract Background. Although uranyl acetate (UA) is known to induce apoptosis in renal tubular cells, the pathophysiological role of apoptotic cell death in UA-induced acute renal failure (ARF) is not clear. In this study, we examined whether glycine, which is known to provide protection against nephrotoxic acute renal failure, attenuated tubular damage in UA-induced ARF in rats, and, if so, whether the attenuation of tubular damage was associated with reduced apoptotic cell death. Methods. Sprague-Dawley rats were allocated to three groups; normal controls, UA-treated, and UA plus glycine-treated. Acute renal failure was induced by the intravenous injection of UA (5 mg/kg). UA plus glycine-treated rats were given glycine at 1 g/kg, i.v. over 3 min at the same time as the UA injection. Serum creatinine concentration (Scr) and tubular damage score were examined 5 days after UA administration. Apoptosis was evaluated by counting the number of terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling (TUNEL)-positive cells in the outer stripe of the outer medulla. Results. Glycine significantly decreased the UA-induced increases in Scr (3.73 ± 0.31 vs 2.74 ± 0.11 mg/dl; P 〈 0.05) and the tubular damage score (3.83 ± 0.13 vs 2.58 ± 0.01; P 〈 0.01). UA significantly increased the number of TUNEL-positive cells in the outer stripe of the outer medulla (0.16 ± 0.04 vs 7.45 ± 0.46/high power field at ×400 magnification; P 〈 0.01 vs normal control value). Glycine infusion significantly lessened the number of TUNEL-positive cells (5.84 ± 0.31/ high power field at ×400 magnification; P 〈 0.01 vs UA-treated rats). A significant correlation was found between the number of TUNEL-positive cells and the tubular damage score (r = 0.93; P 〈 0.01). Conclusion. Glycine ameliorated the severity of UA-induced ARF and the degree of apoptotic cell death. This finding suggested that the protective effect of glycine in UA-induced ARF may be mediated, at least in part, through a reduction of apoptosis.
    Materialart: Digitale Medien
    Bibliothek Standort Signatur Band/Heft/Jahr Verfügbarkeit
    BibTip Andere fanden auch interessant ...
Schließen ⊗
Diese Webseite nutzt Cookies und das Analyse-Tool Matomo. Weitere Informationen finden Sie hier...