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  • 1995-1999  (3)
  • Conditioned reward  (2)
  • Ionosphere (Auroral ionosphere)  (1)
  • 1
    Electronic Resource
    Electronic Resource
    SuperDARN studies of the ionospheric convection response to a northward turning of the interplanetary magnetic field (1998)
    Springer
    Annales geophysicae 16 (1998), S. 549-565 
    Details
    ISSN: 0992-7689
    Keywords: Ionosphere (Auroral ionosphere) ; Magnetospheric physics (Magnetopause, cusp, and boundary layers; Magnetosphere-ionosphere interaction)
    Source: Springer Online Journal Archives 1860-2000
    Topics: Geosciences , Physics
    Notes: Abstract The response of the dayside ionospheric flow to a sharp change in the direction of the interplanetary magnetic field (IMF) measured by the WIND spacecraft from negative Bz and positive By, to positive Bz and small By, has been studied using SuperDARN radar, DMSP satellite, and ground magnetometer data. In response to the IMF change, the flow underwent a transition from a distorted twin-cell flow involving antisunward flow over the polar cap, to a multi-cell flow involving a region of sunward flow at high latitudes near noon. The radar data have been studied at the highest time resolution available (∼2 min) to determine how this transition took place. It is found that the dayside flow responded promptly to the change in the IMF, with changes in radar and magnetic data starting within a few minutes of the estimated time at which the effects could first have reached the dayside ionosphere. The data also indicate that sunward flows appeared promptly at the start of the flow change (within ∼2 min), localised initially in a small region near noon at the equatorward edge of the radar backscatter band. Subsequently the region occupied by these flows expanded rapidly east-west and poleward, over intervals of ∼7 and ∼14 min respectively, to cover a region at least 2 h wide in local time and 5° in latitude, before rapid evolution ceased in the noon sector. In the lower latitude dusk sector the evolution extended for a further ∼6 min before quasi-steady conditions again prevailed within the field-of-view. Overall, these observations are shown to be in close conformity with expectations based on prior theoretical discussion, except for the very prompt appearance of sunward flows after the onset of the flow change.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s00585-998-0549-0
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Enhanced responding for conditioned reward produced by intra-accumbens amphetamine is potentiated after cocaine sensitization (1999)
    Springer
    Psychopharmacology 142 (1999), S. 31-40 
    Details
    ISSN: 1432-2072
    Keywords: Key words Locomotor activity ; Nucleus accumbens ; d-Amphetamine ; Cocaine ; Sensitization ; Conditioned reward
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract  The mesolimbic dopamine (DA) system has been implicated in conditioned reward (CR), locomotor sensitization, and the reinforcing properties of psychomotor stimulants. Stimuli with formerly motivationally neutral properties that gain incentive properties by their predictive association with primary reinforcers are termed conditioned, or secondary, reinforcers. In these experiments, we investigated whether cocaine sensitization could potentiate augmented responding for CR produced by intra-accumbens amphetamine. After subjects were trained on the CR paradigm for 14 days, they received a regimen of cocaine sensitization or saline injections. On 2 test days, 8–10 days later, subjects were given amphetamine (6 µg/0.5 µl) or saline infusions into the nucleus accumbens (NAc) and responding for CR was measured using the ”acquisition of a new response” paradigm. Responding on one novel lever resulted in the delivery of the conditioned stimulus (conditioned reinforcer, or CR lever), whereas responding on the other lever resulted in no CR stimulus presentation (NCR lever). Animals sensitized to cocaine showed increased responding on the CR lever after intra-NAc saline and potentiated CR lever responding after intra-NAc amphetamine. No differences in responding between the cocaine- and saline-treated groups on the NCR lever after the challenge were found. Locomotor sensitization under these conditions was confirmed in a separate group of subjects. These findings show that prior exposures to cocaine results in changes that potentiate the ability of intra-NAc amphetamine to enhance CR. Repeated stimulant drug use may induce long-term neuronal adaptations that result in increased sensitivity to the behavioral, or incentive motivational, effects of stimulant drugs.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s002130050859
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Impulsivity resulting from frontostriatal dysfunction in drug abuse: implications for the control of behavior by reward-related stimuli (1999)
    Springer
    Psychopharmacology 146 (1999), S. 373-390 
    Details
    ISSN: 1432-2072
    Keywords: Key words Compulsion ; Addiction ; Cocaine ; Amphetamine ; Cannabis ; Phencyclidine ; Nucleus accumbens ; Amygdala ; Frontal cortex ; Limbic ; Stimulus-reward association ; Conditioned reward ; Sensitization ; Drug-seeking ; Inhibitory control ; Cognition ; Conditioned stimulus ; Incentive motivational ; Dopamine ; Rat ; Monkey
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract  Drug abuse and dependence define behavioral states involving increased allocation of behavior towards drug seeking and taking at the expense of more appropriate behavioral patterns. As such, addiction can be viewed as increased control of behavior by the desired drug (due to its unconditioned, rewarding properties). It is also clear that drug-associated (conditioned) stimuli acquire heightened abilities to control behaviors. These phenomena have been linked with dopamine function within the ventral striatum and amygdala and have been described specifically in terms of motivational and incentive learning processes. New data are emerging that suggest that regions of the frontal cortex involved in inhibitory response control are directly affected by long-term exposure to drugs of abuse. The result of chronic drug use may be frontal cortical cognitive dysfunction, resulting in an inability to inhibit inappropriate unconditioned or conditioned responses elicited by drugs, by related stimuli or by internal drive states. Drug-seeking behavior may thus be due to two related phenomena: (1) augmented incentive motivational qualities of the drug and associated stimuli (due to limbic/amygdalar dysfunction) and (2) impaired inhibitory control (due to frontal cortical dysfunction). In this review, we consider the neuro-anatomical and neurochemical substrates subserving inhibitory control and motivational processes in the rodent and primate brain and their putative impact on drug seeking. The evidence for cognitive impulsivity in drug abuse associated with dysfunction of the frontostriatal system will be discussed, and an integrative hypothesis for compulsive reward-seeking in drug abuse will be presented.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/PL00005483
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    Paper (German National Licenses)
    Fulltext
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