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  • Electronic Resource  (2)
  • 1,25 Dihydroxycholecalciferol  (1)
  • Collagen  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Further data on the effect of galactose on bone tissue (1974)
    Springer
    Calcified tissue international 16 (1974), S. 153-155 
    Details
    ISSN: 1432-0827
    Keywords: Galactose ; Glucose-6-phosphate dehydrogenase ; Collagen ; Galactose-1-phosphate
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine , Physics
    Notes: Abstract The bone tissue levels of galactose-1-P have been found to be a function of the concentration of galactose in blood.In vitro, the glucose-6-phosphate dehydrogenase (bone isoenzyme) was competitively inhibited by galactose-1-P. Galactose or galactitol were found ineffective. Therefore, the previously reported depressed activity of bone glucose-6-phosphate dehydrogenase in galactose-fed rats should be interpreted as mediated by galactose-1-P. When compared with their controls, galactose-fed rats showed a significantly lower growth rate which was reflected in a lower bone collagen content and in a lower percentage of salt soluble collagen.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02008221
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Respiratory alkalosis and reduced plasmatic concentration of ionized calcium in rats treated with 1,25 dihydroxycholecalciferol (1984)
    Springer
    Calcified tissue international 36 (1984), S. 604-607 
    Details
    ISSN: 1432-0827
    Keywords: Respiratory alkalosis ; Ionized calcium ; 1,25 Dihydroxycholecalciferol ; Parathyroid hormone
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine , Physics
    Notes: Summary The daily administration of supraphysiological doses of 1,25 dihydroxycholecalciferol (0.1–2.5 µg/d/100 g body weight) to rats, produced respiratory alkalosis. With the doses of 0.1–0.2 µg/d/100 g and feeding a diet with 0.7% of calcium, calcemias did not exceed 2.75 mM, and significantly reduced plasma ionized calcium levels were measured. The latter phenomenon was found associated with increased urinary excretion of cAMP, soft tissue calcium content, and polyuria with hypostenuria, all known effects of parathyroid hormone. These effects were absent in thyroparathyroidectomized rats treated in the same fashion. Present results suggest that the stimulus of low levels of plasma ionized calcium overcomes the probably inhibitory effect of the steroid on parathyroid hormone secretion.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02405374
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    Paper (German National Licenses)
    Fulltext
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