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  • 1990-1994  (3)
  • 1980-1984  (2)
  • 1975-1979
  • 1965-1969
  • 1960-1964
  • 1990  (3)
  • 1982  (2)
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Years
  • 1990-1994  (3)
  • 1980-1984  (2)
  • 1975-1979
  • 1965-1969
  • 1960-1964
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  • 1
    Electronic Resource
    Electronic Resource
    Distribution of glycosylglycerols and oligosaccharides in the marine environment and their ecological significance in the deep sea (1990)
    Springer
    Marine biology 106 (1990), S. 309-313 
    Details
    ISSN: 1432-1793
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract Determination of low molecular weight carbohydrates in marine environments indicated that 1-O-β-D-galactosylglycerol, 6-O-α-D-galactosyl-1-O-β-D-galacto-sylglycerol, sucrose, laminaribiose and laminaritriose are widely distributed in seawaters, suspended and sinking particles, and sediments in coastal as well as in deep-sea waters [e.g. Mikawa Bay and Sagami Bay, Kumano Nada (offshore Japan), and northwest North Pacific Ocean, Bering Sea and Antarctic Ocean: collections during 1978–1984]. Identification of these glycosylglycerols and oligosaccharides in algal cells such as a flagellate (Olisthodisus luteus), blue-green algae (e.g.Trichodesmium sp.) and a diatom (Reptocylindrus denicus) strongly suggests that these sugars are photosynthetically produced by algae in the euphotic zone and are then rapidly transported to the deep sea as sinking particles which can be collected by sediment-trap experiments. The rapid decay rate of low molecular weight carbohydrates by microorganisms suggests that the transported sugars provide energy substrates for microorganisms living in the deep sea.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01314815
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Amygdalar inputs to ADH-secreting supraoptic neurones in rats (1982)
    Springer
    Experimental brain research 48 (1982), S. 420-428 
    Details
    ISSN: 1432-1106
    Keywords: ADH-neurone ; Amygdala ; Rat ; Supraoptic nucleus
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Effects of amygdala stimulation on the discharge activity of antidromically identified supraoptic neurosecretory neurones were studied in male rats anaesthetized with urethane. Stimulation of the medial and the basal amygdala produced excitation or inhibition of discharge activity both in phasically firing (“phasic”) and in continuously firing (”continuous”) neurones. More “phasic” neurones were excited than were inhibited after medial amygdala stimulation. On the other hand, fewer “continuous” neurones were excited by stimulation of the either amygdala area than were inhibited. This difference of responsiveness between “phasic” and “continuous” neurones is statistically significant. Synaptic inputs to supraoptic neurosecretory neurones after amygdala stimulation were also observed in rats with a lesion of the stria terminalis. Supraoptic nucleus stimulation activated antidromically 14 of the 336 amygdala neurones tested. Since “phasic” neurones have been identified as ADH-secreting neurones, it is concluded that ADH-secreting neurones in the rat supraoptic nucleus receive predominantly excitatory synaptic inputs from the medial amygdala and these amygdalar synaptic inputs are mediated by pathways which are at least in part monosynaptic and are not included in the stria terminalis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00238618
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Degeneration of retinal neuroblasts by chinoform-ferric chelate (1982)
    Springer
    Cellular and molecular life sciences 38 (1982), S. 120-122 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The possible mechanism of neuropathic effect of chinoform was investigated using cultured retinal neuroblasts from chick embryos. Retinal neuroblasts completely degenerated by chinoform-ferric chelate within a day. This change, however, was not observed with free chinoform or ferric ion.a-Tocopherol had a potent protective effect on the toxicity of the chelate. From these results, it was concluded that the lipid peroxidation due to ferric ion chelated with chinoform incorporated into the membrane of nerve tissues is the most important step in induction of the neuropathy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01944564
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Differential effects of naloxone on neuroendocrine responses to fear-related emotional stress (1990)
    Springer
    Experimental brain research 81 (1990), S. 53-58 
    Details
    ISSN: 1432-1106
    Keywords: Vasopressin ; Oxytocin ; Adrenocorticotrophic hormone ; Emotional stress ; Opioid ; Prolactin ; Rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The effects of an opioid receptor antagonist, naloxone (NAL), were studied on the changes in pituitary hormone secretion induced by emotional stress. Male Wistar rats were trained with tone stimuli paired with electric footshocks and tested with the tone and environmental cue signals for emotional stress of fear acquired by learning as described previously (Onaka et al. 1988). Rats received s.c. injected NAL 30 min before testing at doses of 0, 0.2, 1.0, 5.0 and 25.0 mg/kg b.w. Half the rats were injected with 0.5 M NaCl (20 ml/kg b.w.) together with NAL. In these hypertonic rats plasma vasopressin level was slightly increased after NAL. The increment was statistically significant in control groups but not in experimental groups. However the suppression of vasopressin secretion by emotional stimuli was not changed by NAL. Plasma oxytocin levels were extremely high and not significantly different among experimental, unshocked control and untested control groups. NAL further increased the oxytocin level dose-dependently. NAL did not significantly change plasma adrenocorticotrophic hormone (ACTH) levels and hence did not modify the augmentative response in ACTH secretion to emotional stimuli. Plasma prolactin level was significantly elevated after emotional stimuli and NAL depressed the prolactin level in each of experimental and control groups. After NAL, the magnitude of the facilitatory response in prolactin secretion to emotional stimuli was decreased. Motor activity and its suppressive response to emotional stimuli were not influenced by NAL. In another half of rats under a normal osmotic condition the vasopressin response to emotional stimuli was not affected by NAL. NAL further augmented potentiation of oxytocin secretion after emotional stimuli dose-dependently. Effects of NAL on ACTH level, prolactin level and motor activity were similar to those in rats under hypertonic conditions. These results demonstrate that endogenous opioids are selectively and differentially involved in hypothalamo-hypophysial responses to fear-related emotional stress.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00230100
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    The effects of octopamine on juvenile hormone biosynthesis, electrophysiology, and cAMP content of the corpora allata of the cockroach Diploptera punctata (1990)
    Springer
    Journal of comparative physiology 160 (1990), S. 241-249 
    Details
    ISSN: 1432-136X
    Keywords: Octopamine ; Juvenile hormone ; cAMP ; Cockroach ; Electrophysiology
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Juvenile hormone production by the corpora allata of the adult female cockroach, Diploptera punctata, can be modulated by treatment with the biogenic amine, octopamine. Endogenous octopamine has been identified within the CA, using HPLC and electrochemical detection. Treatment with octopamine results in a sinusoidal, dose-dependent inhibition of JH biosynthesis by CA from day 2 virgin females, with maximal inhibition occurring at 10-10 M and 10-4 M. In day 4 and day 8 mated female corpora allata octopamine inhibited JH biosynthesis at 5·10-5 M. Although the elevation of either cAMP or cGMP within the CA is known to be associated with an inhibition of JH biosynthesis, treatment with high concentrations of octopamine results in an increase in the level of cAMP but not cGMP. This effect is both dose- and time-dependent. Octopamine treatment also initiates changes in the passive membrane responses of the CA. Superfusion of CA with octopamine results in a pronounced hyperpolarization of CA cells and an increase in the electrotonic potential (indicative of the degree of electrical coupling between CA cells). This effect could be blocked by the octopamine receptor blocker phentolamine. Treatment with octopamine or phentolamine also blocked the hyperpolarization of CA cells normally associated with electrical stimulation of the axon tracts innervating the CA. We hypothesize that octopamine may be a natural neuromodulator of JH production by CA, regulating ion channels in CA cells themselves as well as release of the inhibitory neuropeptide, allatostatin, from the terminals within the CA.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00302589
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    Paper (German National Licenses)
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