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  • 1
    Electronic Resource
    Electronic Resource
    Self-similar electron distribution, inverse bremsstrahlung, and heat flux inhibition in high-Z nonuniform plasmas (1995)
    [S.l.] : American Institute of Physics (AIP)
    Physics of Plasmas 2 (1995), S. 3100-3105 
    Details
    ISSN: 1089-7674
    Source: AIP Digital Archive
    Topics: Physics
    Notes: The self-similar distribution of electrons is found for a nonuniform underdense plasma that is heated by an intensive laser field. The distribution function is flat-topped for the low-energy electrons. And in the high-energy region, it has a well-pronounced high-energy tail. It is also found how the electron heat flux and the absorption coefficient depend upon both the ratios of electron mean-free path to the inhomogeneity scale of effective temperature and of the oscillation velocity to the thermal velocity. The actual shapes of electron energy distribution, the heat flux limitation, and the electromagnetic radiation absorption rate are given, both for a currentless plasma and for a plasma with a finite electric current. © 1995 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.871141
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  • 2
    Electronic Resource
    Electronic Resource
    The apple mitochondrial atp9 gene: RNA editing and co-transcription with exons a and b of the nad5 gene (1995)
    Oxford, UK : Blackwell Publishing Ltd
    Physiologia plantarum 93 (1995), S. 0 
    Details
    ISSN: 1399-3054
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology
    Notes: We have isolated and characterized a transcribed segment of mitochondrial DNA (mtDNA) from apple (cv. ‘Delicious’) that contains the atp9 gene, and exons a and b of the nad5 gene. Analysis of the cDNA of the atp9 revealed 8 C to U transcript editing events resulting in 7 amino acid changes, and a new stop codon that eliminates 8 carboxy-terminal residues, compared to the genomic sequence. The transcription patterns of atp9, and exons a and b of nad5 share RNA species, indicating that these sequences are part of the same transcriptional unit.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1399-3054.1995.tb06860.x
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  • 3
    Electronic Resource
    Electronic Resource
    Structure of17Ne by the three-neutron pick-up (3He,6He) reaction (1995)
    Springer
    The European physical journal 353 (1995), S. 117-118 
    Details
    ISSN: 1434-601X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract The nuclear structure of17Ne has been studied by the20Ne(3He,6He)17Ne reaction at 70 MeV. Fifteen levels were identified, and the angular distributions have been measured for 9 levels. The observed transferred angular momentum dependence of the angular distributions enabled clear spin-parity assignment. The extensive completion of T=3/2 quartet in mass A =17 have been obtained for the first time. The present data will provide a chance to study the structure of A=17 system in detail.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01295886
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  • 4
    Electronic Resource
    Electronic Resource
    Stress-response (heat-shock) protein 90 expression in tumors of the central nervous system: an immunohistochemical study (1995)
    Springer
    Acta neuropathologica 89 (1995), S. 184-188 
    Details
    ISSN: 1432-0533
    Keywords: Key words Stress-response protein 90 ; Heat-shock ; protein 90 ; Brain tumors ; Meningiomas ; Breast tumor metastases
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract This retrospective study deals with the expression of stress-response (heat-shock) protein 90 (srp 90) in a series of 148 human brain tumors. Immunohistochemical procedures were employed; cells of the human breast cancer line MCF 7 exposed to hyperosmolar stress served as positive controls. Deposits of reaction products were found in the cytoplasm and they displayed a granular pattern. srp 90 was detected in 14/31 meningiomas and 5/10 breast cancer metastases to the brain. The protein was also present in 6/13 glioblastomas and 7/18 astrocytomas. In addition, a positive reaction was found in 2/10 medulloblastomas, 2/14 primitive neuroectodermal tumors, 1/11 pituitary tumor, 2/21 schwannomas and 2/11 lung tumor metastases; however, oligodendrogliomas and primary malignant lymphomas were not stained. The srp 90 was detected in Western blots of meningioma tissue homogenates. No significant immunohistochemical reaction was seen with sections of normal human cerebra, brain stem, cerebella, pituitary glands and spinal cords. These results document the expression of srp 90 by a variety of primary and metastatic intracranial tumors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00296364
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  • 5
    Electronic Resource
    Electronic Resource
    Stress-response (heat-shock) protein 90 expression in tumors of the central nervous system: an immunohistochemical study (1995)
    Springer
    Acta neuropathologica 89 (1995), S. 184-188 
    Details
    ISSN: 1432-0533
    Keywords: Stress-response protein 90 ; Heat-shock protein 90 ; Brain tumors ; Meningiomas ; Breast tumor metastases
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract This retrospective study deals with the expression of stress-response (heat-shock) protein 90 (srp 90) in a series of 148 human brain tumors. Immunohistochemical procedures were employed; cells of the human breast cancer line MCF 7 exposed to hyperosmolar stress served as positive controls. Deposits of reaction products were found in the cytoplasm and they displayed a granular pattern. srp 90 was detected in 14/31 meningiomas and 5/10 breast cancer metastases to the brain. The protein was also present in 6/13 glioblastomas and 7/18 astrocytomas. In addition, a positive reaction was found in 2/10 medulloblastomas, 2/14 primitive neuroectodermal tumors, 1/11 pituitary tumor, 2/21 schwannomas and 2/11 lung tumor metastases; however, oligodendrogliomas and primary malignant lymphomas were not stained. The srp 90 was detected in Western blots of meiningioma tissue homogenates. No significant immunohistochemical reaction was seen with sections of normal human cerebra, brain stem, cerebella, pituitary glands and spinal cords. These results document the expression of srp 90 by a variety of primary and metastatic intracranial tumors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00296364
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  • 6
    Electronic Resource
    Electronic Resource
    Effects of Troglitazone (CS-045) on insulin secretion in isolated rat pancreatic islets and HIT cells: an insulinotropic mechanism distinct from glibenclamide (1995)
    Springer
    Diabetologia 38 (1995), S. 24-30 
    Details
    ISSN: 1432-0428
    Keywords: Key words Troglitazone (CS-045) ; insulin secretion ; pancreatic islets ; HIT-T15 cells ; glucose transport ; sulphonylurea receptor ; ATP-sensitive K++ channel.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In order to elucidate the direct effects of (±)-5-[4-(6-hydroxy-2, 5, 7, 8-tetramethylchroman-2-yl-methoxy) benzyl]-2,4-thiazolidinedione (Troglitazone), a newly-developed oral hypoglycaemic agent, on pancreatic beta-cell function, in vitro investigation of isolated rat pancreatic islets and a hamster beta-cell line (HIT cell) were performed. Troglitazone stimulates both glucose, and glibenclamide-induced insulin release at a concentration of 10−6 mol/l in these cells but, conversely, inhibits insulin secretion at 10−4 mol/l. Glucose uptake in HIT cells is similarly enhanced by 10−6 mol/l Troglitazone, but is reduced in the presence of 10−4 mol/l Troglitazone. However, a quantitative immunoblot analysis with a specific antibody for GLUT 2 glucose transporter revealed no significant change in GLUT 2 protein in HIT cells with 10−6 mol/l Troglitazone. Specific binding of [3H]-glibenclamide to beta-cell membranes is replaced by Troglitazone in a non-competitive manner, but 10−6 mol/l Troglitazone failed to eliminate ATP-sensitive K++ channel activity. These results suggest that Troglitazone has a putative non-competitive binding site at, or in the vicinity of, the sulphonylurea receptor in rat pancreatic islets and HIT cells and that the dual effect of Troglitazone on insulin secretory capacity is mediated through the modulation of glucose transport activity, possibly due to the modification of intrinsic activity in glucose transporter in pancreatic beta cells by this novel agent. [Diabetologia (1995) 38: 24–30]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001250050249
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  • 7
    Electronic Resource
    Electronic Resource
    Effects of Troglitazone (CS-045) on insulin secretion in isolated rat pancreatic islets and HIT cells: an insulinotropic mechanism distinct from glibenclamide (1995)
    Springer
    Diabetologia 38 (1995), S. 24-30 
    Details
    ISSN: 1432-0428
    Keywords: Troglitazone (CS-045) ; insulin secretion ; pancreatic islets ; HIT-T15 cells ; glucose transport ; sulphonylurea receptor ; ATP-sensitive K++ channel
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In order to elucidate the direct effects of (±)-5-[4-(6-hydroxy-2, 5, 7, 8-tetramethylchroman-2-yl-methoxy) benzyl]-2,4-thiazolidinedione (Troglitazone), a newly-developed oral hypoglycaemic agent, on pancreatic beta-cell function, in vitro investigation of isolated rat pancreatic islets and a hamster beta-cell line (HIT cell) were performed. Troglitazone stimulates both glucose, and glibenclamide-induced insulin release at a concentration of 10−6 mol/l in these cells but, conversely, inhibits insulin secretion at 10−4 mol/l. Glucose uptake in HIT cells is similarly enhanced by 10−6 mol/l Troglitazone, but is reduced in the presence of 10−4 mol/l Troglitazone. However, a quantitative immunoblot analysis with a specific antibody for GLUT 2 glucose transporter revealed no significant change in GLUT 2 protein in HIT cells with 10−6 mol/l Troglitazone. Specific binding of [3H]-glibenclamide to beta-cell membranes is replaced by Troglitazone in a non-competitive manner, but 10−6 mol/l Troglitazone failed to eliminate ATP-sensitive K++ channel activity. These results suggest that Troglitazone has a putative non-competitive binding site at, or in the vicinity of, the sulphonylurea receptor in rat pancreatic islets and HIT cells and that the dual effect of Troglitazone on insulin secretory capacity is mediated through the modulation of glucose transport activity, possibly due to the modification of intrinsic activity in glucose transporter in pancreatic beta cells by this novel agent. [Diabetologia (1995) 38: 24–30]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02369349
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  • 8
    Electronic Resource
    Electronic Resource
    Hyperresponse in calcium-induced insulin release from electrically permeabilized pancreatic islets of diabetic GK rats and its defective augmentation by glucose (1995)
    Springer
    Diabetologia 38 (1995), S. 772-778 
    Details
    ISSN: 1432-0428
    Keywords: Key words Insulin release ; intracellular calcium ; exocytosis ; GK rat ; permeabilized islets.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In spontaneously diabetic GK rats, insulin secretion from pancreatic beta cells in response to glucose is selectively impaired, probably due to deficient intracellular metabolism of glucose and impaired closure of KATP channels during glucose stimulation. By using electrically permeabilized islets of GK rats, we explored the functional modulations in exocytotic steps distal to the rise in [Ca2 + ]i in the diabetic condition. At 30 nmol/l Ca2 + (basal conditions) insulin release was similar between GK and non-diabetic control Wistar rats. In response to 3.0 μmol/l Ca2 + (maximum stimulatory conditions), insulin release was significantly augmented in permeabilized GK islets (p 〈 0.01). Raising glucose concentrations from 2.8 to 16.7 mmol/l further augmented insulin release induced by 3.0 μmol/l Ca2 + from permeabilized control islets(p 〈 0.001), but had no effect on that from permeabilized GK islets. The stimulatory effect of glucose on insulin release from permeabilized control islets was partly inhibited by 2,4-dinitrophenol, an inhibitor of mitochondrial oxidative phosphorylation (p 〈 0.01). The hyperresponse to Ca2 + in GK islets may play a physiologically compensatory role on the putative functional impairment both in [Ca2 + ]i rise and energy state in response to glucose in diabetic β cells, and may explain the relative preservation of insulin release induced by non-glucose depolarizing stimuli, such as arginine, from pancreatic islets in non-insulin-dependent diabetes mellitus. [Diabetologia (1995) 38: 772–778]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001250050351
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  • 9
    Electronic Resource
    Electronic Resource
    Endoscopic ultrasonographic-guided punctured pancreatic ductography: an initial and successful trial (1995)
    Springer
    Abdominal imaging 20 (1995), S. 222-224 
    Details
    ISSN: 1432-0509
    Keywords: Pancreatic ductography ; Endoscopic ultrasonography ; Puncture ; Intraductal papillary tumor ; Pancreas
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract A 71-year-old male suffering from an intraductal papillary tumor of the pancreas was admitted to our hospital for further investigation. Diagnostic trials, including endoscopic retrograde pancreatography, did not produce an adequate ductography because of a large amount of mucinous fluid. Therefore, we performed endoscopic ultrasonographic-guided punctured pancreatic ductography (EPPD). This procedure was safely performed without any complications. We report this initial and successful trial of EPPD.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00200400
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  • 10
    Electronic Resource
    Electronic Resource
    Effect of stopping fluoride administration on the distribution profiles of fluoride in three different kinds of rat bones (1995)
    Springer
    Calcified tissue international 56 (1995), S. 292-296 
    Details
    ISSN: 1432-0827
    Keywords: Fluoride ; Bone ; Defluoridation ; Rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine , Physics
    Notes: Abstract The aim of this work was to explore the reduction of fluoride concentrations in the skeleton after stopping experimental fluoride administration. Fluoride was administered to the rats at varying doses (0, 50, 100 ppm in drinking water) and for different lengths of time (4, 13, 25 weeks). A series of fluoride concentrations across the full thickness of humerus, parietal bone, and vertebra arch in rats were measured by means of an abrasive micro-sampling technique. The distribution profiles of fluoride from periosteal to endosteal surfaces, which were apparently related to the histological structure of these bones, were U shaped in the humerus, V shaped in the parietal bone, and W shaped in the vertebra arch. The average fluoride concentrations in the bones increased significantly with each increasing dose and length of fluoride administration. The relative increments were similar between the different regions or the different bones. After stopping fluoride administration, on the other hand, the relative reduction of the average fluoride concentrations in the bones were 30–100%. They were greatly related to the length after stopping fluoride administration and the dose and length of fluoride administration, but also dependent upon the type of bone and the region examined.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00318049
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