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  • 2000-2004  (3)
  • 1945-1949
  • 2000  (3)
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  • 2000-2004  (3)
  • 1945-1949
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  • 1
    Electronic Resource
    Electronic Resource
    Single-crystalline silicon lift-off films for metal–oxide–semiconductor devices on arbitrary substrates (2000)
    Woodbury, NY : American Institute of Physics (AIP)
    Applied Physics Letters 77 (2000), S. 558-560 
    Details
    ISSN: 1077-3118
    Source: AIP Digital Archive
    Topics: Physics
    Notes: We present a technique to mount single-crystalline silicon thin films on arbitrary substrates. We demonstrate in detail the preparation of a 190-nm-thin silicon metal–oxide–semiconductor field-effect transistor (MOSFET) on a silicon-on-insulator film lifted from its substrate and bonded to quartz. Functioning of this hybrid MOSFET on a rigid surface at room temperature is demonstrated. © 2000 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.127043
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Single-electron tunneling in silicon nanostructures (2000)
    Springer
    Applied physics 71 (2000), S. 357-365 
    Details
    ISSN: 1432-0630
    Keywords: PACS: 85.30.Vw; 85.30.Yy; 07.10.Cm
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Notes: Abstract. We present a brief overview on different realizations of single-electron devices fabricated in silicon-on-insulator films. Lateral structuring of highly doped silicon films allows us to observe quasi-metallic Coulomb blockade oscillations in shrunken wires where no quantum dot structure is geometrically defined. Embedding quantum dot structures into the inversion channel of a silicon-on-insulator field-effect transistor Coulomb blockade up to 300 K is observed. In contrast to the quasi-metallic structures, in these devices the influence of the quantum mechanical level spacing inside the dot becomes visible. Suspending highly doped silicon nanostructures leads to a novel kind of Coulomb blockade devices allowing both high-power application as well as the study of electron–phonon interaction.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003390000549
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Role of apoptosis in autoimmunity (2000)
    Springer
    Apoptosis 5 (2000), S. 443-449 
    Details
    ISSN: 1573-675X
    Keywords: apoptosis ; autoimmunity
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Apoptosis is a physiological form of cell death required to ensure that the rate of cell division is balanced by the rate of cell death in multicellular organisms. Dysregulation of apoptosis is associated with the pathogenesis of a wide array of diseases: cancer, neurodegeneration, autoimmunity, heart disease and others. In this review we collect arguments supporting a hypothesis of a dysregulated apoptosis leading to development of autoimmunity like systemic lupus erythematosus (SLE). This notion is supported by occurence of known autoantigens in apoptotic blebs, in vitro findings of an increased rate of apoptotic lymphoblasts despite optimal cytokine stimulation combined with a defective in vitro clearance of apoptotic bodies by SLE phagocytes. Moreover, we and others could generate histone-specific lymphocytic cell lines from cells after activation with autologous apoptotic material. These lymphocytes could stimulate autologous B-lymphocytes to produce of anti-dsDNA antibodies, a diagnostic hallmark for SLE. Finally, antibodies against phospholipids like phosphatidylserine are often associated with systemic autoimmunopathies like SLE and others. Phosphatidylserine is exposed on apoptotic cells as early sign of programmed cell death and serves as phagocyte recognition molecule for apoptotic cells. Formation of immune complexes and deposition in tissues might lead to organ damage and disease. This scenario will be discussed in this review in detail.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1009692902805
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    Paper (German National Licenses)
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