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  • 1980-1984  (2)
  • collagen  (1)
  • subarachnoid haemorrhage (SAH)  (1)
  • hydrocephalus
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  • 1
    Electronic Resource
    Electronic Resource
    Springer
    Acta neurochirurgica 53 (1980), S. 237-245 
    ISSN: 0942-0940
    Keywords: Subarachnoid haemorrhage (SAH) ; subarachnoid fibrosis (SF) ; polarization microscope ; collagen
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The author used the polarization microscope and topo-optical reactions such as phenol reaction, collagen-specific sulphation, and picrosirius red F 3 BA staining to investigate the nature of subarachnoid fibrosis after experimental subarachnoid haemorrhage. It is strongly suggested that the subarachnoid fibrotic deposits—which here analysed in a previous study by scanning electronmicroscopy—contain a high amount of collagen as a main component both three weeks and three months after the SAH.
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 0942-0940
    Keywords: Subarachnoid fibrosis ; subarachnoid haemorrhage (SAH) ; tranexamic acid ; antifibrinolytic therapy ; scanning electron microscopy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The effect of antifibrinolytic therapy on posthaemorrhagic subarachnoid fibrosis was observed experimentally in dogs with the scanning electron microscope (SEM). The subchronic subjects, given intravenous injections of tranexamic acid (1 mg/day) for 12 days and sacrificed 3 weeks after cisternal blood injection, showed residual clot with thick fibrosis, especially around the haemorrhage. The chronic subjects, to which the same procedure was applied and which were sacrificed three months after cisternal blood injection, showed significant increases in the subarachnoid fibrosis, most remarkably in the parasagittal region. Tranexamic acid is widely used for preventing the recurrence of subarachnoid haemorrhage. However, it was revealed in this study that antifibrinolytic therapy might increase chronic posthaemorrhagic subarachnoid fibrosis, which is considered to be responsible for communicating hydrocephalus by disturbing epicortical CSF flow4.
    Type of Medium: Electronic Resource
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