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  • 1
    Electronic Resource
    Electronic Resource
    Die Wirkung von Histamin, Carbachol, Pentagastrin und Hühnergastrinextrakten auf die Magensekretion von nicht narkotisierten Hühnern mit einer Magenfistel (1970)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 267 (1970), S. 170-176 
    Details
    ISSN: 1432-1912
    Keywords: Chicken ; Acid Secretion ; Histamine ; Gastrin ; Carbachol ; Huhn ; Säuresekretion ; Histamin ; Gastrin ; Carbachol
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. The effect of histamine, carbachol, pentagastrin and gastrin extracts of different organs (glandular stomach, gizzard, duodenum) on gastric acid secretion was investigated in unanaesthetized chickens with a gastric fistula. 2. On a molar basis, pentagastrin was three times as potent as carbachol and 12 times as potent as histamine. The dose of pentagastrin required for stimulation of gastric secretion in chickens was very much higher than in mammals. 3. Extracts from the glandular stomach, the gizzard and the duodenum were free from acid secretion stimulating activity. 4. It was concluded that gastric acid secretion in chickens is controlled by a mechanism other than gastrin.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00999398
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  • 2
    Electronic Resource
    Electronic Resource
    Does burimamide inhibit gastric acid secretion by a release of catecholamines? (1973)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 279 (1973), S. 417-420 
    Details
    ISSN: 1432-1912
    Keywords: Cats ; Histamine ; Burimamide ; Gastric Acid Secretion ; Gastric Mucosal Blood Flow
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In anaesthetized cats burimamide inhibits gastric acid secretion, reduces gastric mucosal blood flow, rises blood pressure and increases cardiac frequency. The effects or burimamide on blood pressure, cardiac frequency and on gastric acid secretion are antagonized by reduction of adrenergic influences by adrenalectomy of adrenergic blocking agents. The results indicate that burimamide releases catecholamines which are at least in part responsible for the effects of burimamide.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00500807
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Do chickens have gastrin-like compounds? (1973)
    Springer
    Cellular and molecular life sciences 29 (1973), S. 1096-1096 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Zusammenfassung Bei Hühnern wurden in Extrakten der Schleimhaut des Duodenums geringe Mengen einer Substanz nachgewiesen, die radioimmunologisch mit Antikörpern gegen synthetisches Humangastrin 2–17 reagiert. Serum, Oesophagus, Kropf, Drüsenmagen, Hornmagen und Pankreas enthielten keinerlei Aktivität. Die gastrinähnliche Substanz spielt vermutlich für die Steuerung der Magensekretion bei Hühnern keine wesentliche Rolle.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01946740
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  • 4
    Electronic Resource
    Electronic Resource
    Histamin, Histidindecarboxylase und Gastrin im oberen Verdauungstrakt des Huhns (1970)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 265 (1970), S. 301-309 
    Details
    ISSN: 1432-1912
    Keywords: Chicken ; Gastrointestinal Tract ; Histamine ; Histidine Decarboxylase ; Gastrin ; Huhn ; Gastrointestinaltrakt ; Histamin ; Histidin-decarboxylase ; Gastrin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. The histamine, histidine decarboxylase and gastrin content of the oesophagus, crop, glandular stomach, gizzard and duodenum was determined in chickens. 2. Only minute amounts of histamine were found in the oesophagus (0.8 μg/g) and crop (0.5 μg/g). In the glandular stomach (7.7 μg/g), however, the gizzard (4.8 μg/g) and the duodenum (12.9 μg/g) the histamine content was considerably higher. 3. Measurable amounts of the specific histidine decarboxylase activity were found only in the glandular stomach (25.3 nMole newly formed histamine/g · hr−1). 4. In none of the organs investigated, could any gastrin activity be definitely detected. 5. From the experiments it was concluded that gastric acid secretion in chickens may be controlled by systems other than gastrin. The role of histamine in this process is uncertain.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00997188
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  • 5
    Electronic Resource
    Electronic Resource
    Die Rolle des Kropfs bei der Steuerung der Magensaftsekretion von Hühnern (1971)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 271 (1971), S. 142-148 
    Details
    ISSN: 1432-1912
    Keywords: Chicken ; Crop ; Acid Secretion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. The contribution of the crop to gastric acid secretion was investigated in unanaesthetized chickens with a gastric fistula. 2. Basal secretion was enhanced by removal or distension of the crop and reduced by irrigating the crop with food suspension, acetylcholine, glycine or propyl alcohol as compared with phosphate buffer. 3. It was concluded that the crop is able to release stimulating and inhibiting impulses towards the glandular stomach. The stimulating impulses were elicited by mechanical, the inhibiting ones by chemical excitation.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00998575
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  • 6
    Electronic Resource
    Electronic Resource
    Gastric acid secretion and the expiration of radioactive carbon dioxide of carboxyl-14C-labelled histidine-loaded rats (1971)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 271 (1971), S. 149-156 
    Details
    ISSN: 1432-1912
    Keywords: Gastric Acid Stimulants ; Carboxyl-labelled Histidine ; 14C-Co2-Expiration ; Gastric Secretion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. The effects of gastrin I, bethanechol, histamine and reserpine on gastric acid secretion and the expiration of radioactive carbon dioxide have been studied in anaesthetized carboxyl-14C-l-histidine-loaded rats. 2. All compounds tested stimulated gastric acid secretion. In unstimulated animals the radioactive carbon dioxide in the expired air dropped exponentially with time; it was increased by reserpine and histamine, but not by gastrin and bethanechol. 3. In gastrin- and histamine-treated animals acetazolamide suppressed gastric acid secretion as well as the expiration of radioactive carbon dioxide. 4. The effect of histamine on the expiration of radioactive carbon dioxide can be attributed to effects other than the enhanced decarboxylation of histidine. The response to reserpine can be explained as an activation of histidine decarboxylase. Whether the failure of gastrin I and bethanechol to increase the expiration of radioactive carbon dioxide documents their inability to stimulate histidine decarboxylase in vivo, cannot be decided.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00998576
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  • 7
    Electronic Resource
    Electronic Resource
    The effect of phloxin on bethanechol and histamine stimulated gastric acid secretion in rats (1972)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 272 (1972), S. 117-120 
    Details
    ISSN: 1432-1912
    Keywords: Gastric Secretion ; Bethanechol ; Histamine ; Phloxin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The effect of phloxin (Na salt of tetrabromo-tetrachlorofluorescein) on histamine- and bethanechol-stimulated gastric acid secretion was studied in anaesthetized rats. Concentrations of phloxin, equimolar with those of the stimulants, depressed the secretory response to histamine and had no influence on the bethanechol effect. The results suggest that histamine is not involved in the events triggered by bethanechol which stimulate gastric acid secretion in rats.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00498798
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  • 8
    Electronic Resource
    Electronic Resource
    Gastric acid secretion in conscious miniature pigs (1974)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 283 (1974), S. 83-92 
    Details
    ISSN: 1432-1912
    Keywords: Pig ; Gastric Secretion ; Histamine ; Carbachol ; Pentagastrin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Gastric acid secretion was studied in 5 miniature pigs provided with a chronic gastric fistula. Basal secretion did not change during the growth of the animals. Histamine, carbachol, and pentagastrin produced a dose-dependent volume and acid response. The highest peak acid output (PAO) was achieved with histamine. The maximal peak volume output after histamine was only exceeded by carbachol which was due to an excessive salivation. The maximal PAO after carbachol and pentagastrin as well as the maximal peak volume output after pentagastrin were very low. The poor secretory responses to carbachol and pentagastrin are discussed in the light of possible inhibitory mechanisms or of drug-receptor-interactions.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00500147
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  • 9
    Electronic Resource
    Electronic Resource
    Acid base balance in relation to gastric acid secretion stimulated by histamine and pentagastrin in cats (1974)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 285 (1974), S. 325-335 
    Details
    ISSN: 1432-1912
    Keywords: Cats ; Gastric Acid Secretion ; Acid Base Balance ; Carbonic Anhydrase
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. In anaesthetized gastric fistula cats gastric acid secretion and the following blood gases and acid base parameters were determined in the arterial blood and in the gastric venous drainage: pH, HCO 3 − concentration, pCO2 and pO2. 2. During pentagastrin-stimulated gastric acid secretion HCO 3 − concentration and pH rose in the gastric venous effluent and to a small extent in the arterial blood. The venous pO2 dropped. 3. During histamine-stimulated gastric acid secretion, in addition to the changes occurring during pentagastrin-stimulation, an initial drop in arterial pO2, HCO 3 − concentration and pH was observed. 4. After pretreatment of the animals with phenoxybenzamine or mepyramine the effects of histamine in the arterial blood were abolished suggesting that a histamine-mediated release of catecholamines is responsible for these effects of histamine. 5. Inhibition of carbonic anhydrase by acetazolamide inhibited gastric acid secretion and produced a rise in pCO2 with a concomitant drop in pH.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00501462
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  • 10
    Electronic Resource
    Electronic Resource
    The effect of histamine-H2-receptor-antagonists on gastric acid secretion, gastric mucosal blood flow and acid base balance in cats (1974)
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 285 (1974), S. 337-353 
    Details
    ISSN: 1432-1912
    Keywords: Cats ; H2-Receptor-Antagonists ; Gastric Acid Secretion ; Gastric Mucosal Blood Flow ; Acid Base Balance
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. In anaesthetized acute gastric fistula cats the effect was studied of the histamine-H2-receptor-antagonists burimamide and metiamide on histamine-stimulated gastric acid secretion, gastric mucosal blood flow, and acid base balance of the arterial blood and the gastric venous drainage. 2. Both burimamide and metiamide inhibit gastric secretion and gastric mucosal blood flow. The effects of metiamide are more pronounced than those of burimamide. On acid base balance both histamine-H2-receptor-antagonists have essentially the same effects which reflect the reduced loss of H+ from the stomach. 3. The inhibitory effects of burimamide on the stomach are at least in part antagonized by the histamine-H1-receptor-antagonist mepyramine (which presumably prevents burimamide from releasing catecholamines) and by the α-adrenergic blocking agent phenoxybenzamine, providing indirect evidence for the view that these effects of burimamide are mediated by catecholamines. 4. Phenoxybenzamine and mepyramine do not antagonize the gastric inhibitory effects of metiamide indicating that metiamide is void of catecholamine releasing properties. 5. Phenoxybenzamine and mepyramine influence acid base balance only when the inhibition of gastric acid secretion by burimamide was diminished. They prevented the histamine-mediated drop in arterial pO2, indicating that this histamine effect can be attributed to catecholamines, too.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00501463
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