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  • 1
    ISSN: 1433-0385
    Keywords: Key words: Cholangitis ; Anisoperistaltic Roux reconstruction ; Hepaticojejunostomy. ; Schlüsselwörter: Cholangitis ; anisoperistaltische Roux-Schlinge ; Hepaticojejunostomie.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung. Beim Auftreten einer Cholangitis nach Hepaticojejunostomie ist die häufigste Ursache eine Stenose der biliodigestiven Anastomose. Die vorliegende Kasuistik beschreibt die Krankengeschichte eines Patienten, bei dem eine Hepaticojejunostomie infolge einer Gallengangsverletzung bei Mirizzi-Syndrom angelegt worden war, und der postoperativ unter rezidivierenden Cholangitiden litt. Die präoperative bildgebende Diagnostik zeigte eine Stenose der biliodigestiven Anastomose und eine massiv erweiterte Roux-Schlinge, so daß zusätzlich der Verdacht auf eine Stenose dieser Schlinge im Mesocolonschlitz bestand. Intraoperativ fand sich jedoch eine anisoperistaltisch angeschlossene Roux-Schlinge. Diese Konstellation sollte als seltene Ursache rezidivierender Cholangitiden nach Hepaticojejunostomie differentialdiagnostisch berücksichtigt werden.
    Notes: Summary. Stenosis of the hepatico-intestinal anastomosis after hepaticojejunostomy is a common cause for recurrent cholangitis. In the following report a patient's history of recurrent cholangitis after hepaticojejunostomy performed because of bile duct injury is presented. Preoperative imaging revealed a stricture of the hepatico-intestinal anastomosis and a massive dilatation of the Roux-en-Y loop, so that a draining disorder was assumed. Laparotomy showed an anisoperistaltic Roux-en-Y loop. When assessing the differential diagnosis of recurrent cholangitis after hepaticojejunostomy, anisoperistaltic reconstruction should be considered.
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Springer
    Naunyn-Schmiedeberg's archives of pharmacology 345 (1992), S. 606-609 
    ISSN: 1432-1912
    Keywords: Chick sympathetic neurons ; α2-Adrenoceptors ; Pertussis toxin ; Ca2+ currents ; H-noradrenaline release
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Effects of α2-adrenoceptor agonists on whole-cell Ca2+ currents and 3H-noradrenaline release were investigated by applying the patch-clamp technique and electrical field stimulation to cultured embryonic chick sympathetic neurons. A 24-h exposure of the sympathetic neurons to pertussis toxin (100 ng/ml) abolished both the α2-adrenoceptor-mediated inhibition of Ca 2+ currents and the modulation of noradrenaline release caused by noradrenaline (1 μmol/l; in the presence of 10 μmol/l cocaine) or the α2-adrenoceptor agonists 5-bromo-6-(2imidazolin-2-ylamino)quinoxaline (UK 14,304, 10 μmol/ l) and clonidine (10 μmol/l). These results suggest that the α2-autoreceptor-mediated inhibition of noradrenaline release from chick sympathetic neurons operates through the modulation of Ca2+ channels via pertussistoxin-sensitive GTP-binding-proteins.
    Type of Medium: Electronic Resource
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