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  • 1
    Electronic Resource
    Electronic Resource
    s.l. ; Stafa-Zurich, Switzerland
    Materials science forum Vol. 255-257 (Sept. 1997), p. 342-344 
    ISSN: 1662-9752
    Source: Scientific.Net: Materials Science & Technology / Trans Tech Publications Archiv 1984-2008
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    s.l. ; Stafa-Zurich, Switzerland
    Materials science forum Vol. 105-110 (Jan. 1992), p. 1177-1180 
    ISSN: 1662-9752
    Source: Scientific.Net: Materials Science & Technology / Trans Tech Publications Archiv 1984-2008
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Type of Medium: Electronic Resource
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  • 3
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Chemical Physics Letters 173 (1990), S. 513-520 
    ISSN: 0009-2614
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Chemistry and Pharmacology , Physics
    Type of Medium: Electronic Resource
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  • 4
    Electronic Resource
    Electronic Resource
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 88 (2000), S. 4376-4379 
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: The growth of microcrystalline silicon, μc-Si, films has been studied by infrared spectroscopy and x-ray diffraction. Thin films of various thickness have been prepared from SiH4–H2 mixtures by electron-cyclotron resonance chemical vapor deposition. Two structural transitions were observed during film growth. The first transition at a critical thickness of dac=9 nm manifested itself by a change from an initially amorphous growth to polycrystalline growth. The second structural transition was related to an increasing amount of silicon grains of preferred orientation with (110) lattice planes parallel to the substrate. The population of such (110)-oriented grains N110 was found to become dominant at about d110=310 nm, which may be considered as a second critical thickness above which the film exhibits a (110) fiber texture. The increase of N110 with increasing thickness follows a d1/6 dependence. The effect is understood in terms of an interplay between etching and deposition during growth. © 2000 American Institute of Physics.
    Type of Medium: Electronic Resource
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  • 5
    ISSN: 1432-0533
    Keywords: Key words Drug addiction ; Brain ; Hippocampus ; Nerve cell damage ; Hypoxia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Neuropathological studies were carried out on 180 human immunodeficiency virus-seronegative intravenous drug addicts. The findings in victims of acute heroin intoxication (n = 116) were congestion (99.1%), capillary engorgement (68.1%), and/or perivascular bleeding (68.1%) – hemodynamic processes attributable to toxic primary respiratory failure. In a high percentage of these cases (88%), cerebral edema was also present. In 18 cases of acute heroin intoxication who survived for periods of hours or days, the sole postmortem finding was ischemic nerve cell damage, resembling that typically seen in systemic hypoxia. Semiquantitative analysis revealed nerve cell loss in the hippocampal formation and/ or Purkinje cell layer in 26% of the 162 chronic drug abusers. By contrast, in nearly 80% of these cases, the hippocampus showed enhanced expression of glial fibrillary acid protein by astrocytes and/or a proliferation of microglia, demonstrated by CD68 expression. Since such reactive processes are produced by primary neuronal damage, it can be assumed that chronic intravenous drug abuse results in obviously ischemic nerve cell loss. This could be demonstrated in the hippocampus, but it must also occur throughout the whole brain. The demonstration of ischemic nerve cell damage and neuronal loss or secondary reactive alterations has not been described previously.
    Type of Medium: Electronic Resource
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  • 6
    ISSN: 1434-5196
    Keywords: Key words Internal cerebral vein thrombosis ; Sinovenous thrombosis ; Colitis ulcerosa ; Trauma ; Schlüsselwörter Hirnvenenthrombose ; Sinusthrombose ; Trauma ; Colitis ulcerosa
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine , Law
    Description / Table of Contents: Zusammenfassung Venöse Abflußstörungen des Gehirns werden mit unterschiedlicher Häufigkeit im Obduktionsgut aufgeführt. Neben entzündlichen Prozessen, hämorheologischen Störungen und Neoplasien kommen bei der forensischen Begutachtung traumatische Ursachen für die Genese von Thrombosen in Frage. Berichtet wird über eine 14 Jahre alt gewordene Schülerin, die 2 1/2 Tage nach einer tätlichen Auseinandersetzung verstarb. Anamnestisch wurde von einer Colitis ulcerosa berichtet. Initial waren weder äußere Verletzungen noch neurologische Auffälligkeiten erkennbar. In der Nacht wurde sie komatös in der Klinik aufgenommen. Die kranielle computertomographische Untersuchung zeigte eine zunehmende intraventrikuläre und intrazerebrale Blutung, Zeichen der venösen Stauung und ein Hirnödem. Die Patientin verstarb im dissoziierten Hirntod. Neuropathologisch fand sich eine in Organisation stehende Thrombose des Sinus sagittalis superior, die sich in die inneren Hirnvenen fortsetzte und die durch die entzündliche Darmerkrankung verursacht war. Eine traumatische Genese war somit auszuschließen.
    Notes: Abstract Thromboses of the cerebral veins which occur at a frequency of 3.75 to 9.3% in autopsy material are caused by local or systemic inflammatory processes, hematological disorders, neoplasms or traumatic brain damage. We report on a 14-year-old girl who suffered from colitis ulcerosa during the last 4 years of life. Immediately after a brawl with a schoolboy she complained of headache and vertigo, but no physical abnormality was detected by the physician. She was admitted to the hospital after becoming comatose during the following night. On neurological examination she was comatous with a decerebrate state, the pupils were small and non-reactive to light, divergent ocular bulbi and bilateral extensor plantar responses. A cerebral computer tomogramm showed a cerebral edema and a hemorrhage in the left basal ganglia and both cerebral ventricles which subsequently increased to the right cerebral ganglia, the corpus callosum and the right cerebral hemisphere. Despite antiedematous therapy the girl died on the second day after the brawl. A neuropathology examination revealed thrombosis of the superior sagittal sinus extending to the internal cerebral veins and a bilateral hemorrhagic infarction of the thalamus and the basal ganglia with intraventricular hemorrhage. As the thrombus in the sagittal sinus showed signs of organisation, a postraumatic origin alone is improbable and a hemorrheological disorder due to colitis ulcerosa seemed to be the main cause.
    Type of Medium: Electronic Resource
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