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  • 1
    Electronic Resource
    Electronic Resource
    Interstitial and substitutional Zn in InP and InGaAsP (1989)
    [S.l.] : American Institute of Physics (AIP)
    Journal of Applied Physics 65 (1989), S. 553-560 
    Details
    ISSN: 1089-7550
    Source: AIP Digital Archive
    Topics: Physics
    Notes: Closed-ampoule Zn diffusion in InP results in a net acceptor concentration that is much smaller than the Zn concentration. After subsequent annealing of InP in an atmosphere without Zn, the Zn and net acceptor concentrations have become almost identical, due to a decreased Zn concentration and an increased net acceptor concentration. The annealing treatment gives rise to a decreased contact resistivity. The diffusion depth has not changed. Annealing with a SiN cap on the InP surface does not have this effect on the concentrations. These annealing effects also take place in InGaAsP on InP layers. The results can be explained quantitatively by assuming that Zn is incorporated as both substitutional acceptors and interstitial donors and that only the interstitial Zn is driven out by the annealing, owing to its large diffusion coefficient. Profiles calculated with this interstitial-substitutional model can be fitted to experimental profiles assuming Zn to diffuse as singly ionized interstitial donors. This model can also describe earlier reported results on Zn diffusion in n-type InP for which a profile cutoff is found at a depth where the acceptor concentration equals the background donor concentration and in which the acceptor solubility is higher than in undoped InP.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.343140
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  • 2
    Electronic Resource
    Electronic Resource
    Bismuth abundance anomaly in a Hg—Mn star (1982)
    [s.l.] : Nature Publishing Group
    Nature 299 (1982), S. 535-536 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] The IUE spectra were processed at the University College London Starlink node using the standard analysis programs STAK and TRAK. In Fig. 1 we compare the region of the Bi II resonance line at 1,436.83 A in SWP images of three stars: HR7775, i CrB (Hg-Mn) and v Cap (normal B9.5 V). The spectra of ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/299535a0
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  • 3
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    Stolen Picture, The (1904)
    London : Periodicals Archive Online (PAO)
    Temple bar. 129 (1904:Jan./June) 211 
    Details
    ISSN: 1751-9063
    Topics: General, Interdisciplinary
    URL: http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pao:&rft_dat=xri:pao:article:8741-1904-129-00-000048
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  • 4
    Electronic Resource
    Electronic Resource
    Morphological changes in IgM paraproteinaemic neuropathy (1990)
    Springer
    Acta neuropathologica 80 (1990), S. 77-84 
    Details
    ISSN: 1432-0533
    Keywords: Peripheral nerve ; IgM paraprotein ; Myelin ; Axonal atrophy ; Demyelination
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Sural nerve biopsies were examined from two patients with neuropathy associated with IgM kappa [anti-myelin-associated glycoprotein (MAG)] paraproteinaemia. Both nerves had a moderate loss of myelinated fibres. The pathology in one was of a chronic primary demyelinating type, in the other it was associated with axonal atrophy. Widened myelin (WM) was seen in both nerves affecting over 80% and 50% of myelinated fibres, respectively. The WM was associated with deposition of material which sometimes appeared granular but could also display a highly organised pattern, an appearance not previously described in these neuropathies. Granular material was also identified at the external surface of the Schwann cells of myelinated, but not of unmyelinated, fibres. WM was seen not only at the outer lamellae (a commonly observed site) but also at terminal myelin loops at the paranode, at Schmidt Lanterman incisures and at the inner and outer mesaxon. Material was also seen on the inner (adaxonal) Schwann cell suface. These are all sites associated with the presence of MAG. Other pathological features are described, including evidence of impairment of remyelination, abnormal Schwann cell/axon specialisations and the presence of tomaculous bodies. The implications of these findings are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294225
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  • 5
    Electronic Resource
    Electronic Resource
    Uncompacted lamellae as a feature of tomaculous neuropathy (1991)
    Springer
    Acta neuropathologica 83 (1991), S. 87-91 
    Details
    ISSN: 1432-0533
    Keywords: Tomaculous neuropathy ; Uncompacted myelin ; Tomaculous bodies
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Clinical findings suggested an inherited tendency to pressure palsies, but in this case without a family history. This was confirmed pathologically by the identification of a tomaculous neuropathy showing some atypical features including the presence of uncompacted lamellae in a high proportion of fibres.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294435
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  • 6
    Electronic Resource
    Electronic Resource
    An unusual demyelinating neuropathy in a patient with Waardenburg's syndrome (1992)
    Springer
    Acta neuropathologica 83 (1992), S. 670-674 
    Details
    ISSN: 1432-0533
    Keywords: Waardenburg's syndrome type II ; Hirschsprung's disease ; Demyelination ; Hereditary motor and sensory neuropathy ; Myelin sheath folding
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We present clinical and laboratory data from a patient with Waardenburg's syndrome type II comprising iris heterochromia and deafness, complicated by Hirschsprung's disease — a known association -and an unusual demyelinating peripheral neuropathy — a unique association. The neuropathy is characterised by excessive focal folding of myelin sheaths. It is our view that, although both disorders could represent the consequences of neural crest embryopathy, it is more likely that they are associated by chance.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00299420
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  • 7
    Electronic Resource
    Electronic Resource
    Myelin changes in leprous neuropathy (1987)
    Springer
    Acta neuropathologica 74 (1987), S. 75-80 
    Details
    ISSN: 1432-0533
    Keywords: Leprosy neuropathy ; Peripheral nerve ; Myelin ; Myelin periodicity ; Artefact
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Myelin changes were observed in fibres of nerves from cases of leprosy. The myelin had a ‘loosened’ appearance caused by increased and irregular separation of the intraperiod line. ‘Loosening’ might affect all, or only some, of the lamellae forming a myelin sheath. There was a pathcy distribution of fibres with abnormal myelin, and they were seen only in nerves showing other marked pathological changes including the presence of oedema. The appearances are suggestive of intramyelinic oedema which may be related to the presence of endoneurial oedema.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688341
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  • 8
    Electronic Resource
    Electronic Resource
    Cytomembranous inclusions in the peripheral nerves in AIDS (1989)
    Springer
    Acta neuropathologica 79 (1989), S. 336-339 
    Details
    ISSN: 1432-0533
    Keywords: AIDS ; Cytomembranous inclusions ; Tubuloreticular inclusions ; Ultrastructure ; Peripheral nerve
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We report finding tubuloreticular inclusions (TRI) in the endothelial cells of endo- and epineurial vessels in the sural nerve of 11 patients with AIDS. Six patients had a painful peripheral neuropathy, one a non-painful sensory neuropathy, one an acute inflammatory demyelinating polyradiculoneuropathy and one a thalidomide-related neuropathy. Two patients had no clinical evidence of neuropathy. The TRI are not specific to one neuropathy and are unlikely to contribute to the pathogenesis of peripheral nerve syndromes in AIDS.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00294672
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  • 9
    Electronic Resource
    Electronic Resource
    Axonal atrophy in the painful peripheral neuropathy in AIDS (1990)
    Springer
    Acta neuropathologica 81 (1990), S. 198-203 
    Details
    ISSN: 1432-0533
    Keywords: AIDS ; Pain ; Neuropathy ; Axonal atrophy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary We compared the sural nerves from patients with AIDS; four with the painful peripheral neuropathy (PPN), three patients with non-painful distal symmetrical peripheral neuropathy (DSPN), one without clinical peripheral nerve involvement and two human immunodeficiency virus negative controls. Fibre diameter distributions showed a slight reduction in larger fibres in all patients with AIDS compared with controls. No significant difference was found in the relative frequency distribution of the lamellae counts between any of the groups. The relative frequency distribution of axonal area of myelinated fibres did not differ between controls, non-pailful DSPN or the patient without peripheral nerve involvement; however, all patients with PPN showed marked reduction in the number of axons of myelinated fibres of larger area which was significant when compared to each patient from the other groups (P〈0.0001). This indicates that the reduction in larger fibres in PPN is mostly due to axonal atrophy rather than selective fibre loss. Axonal atrophy is associated with painful peripheral neuropathy in AIDS but not with those without pain. The possible role of axonal atrophy as a pathological substrate for pain is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00334508
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  • 10
    Electronic Resource
    Electronic Resource
    The pathology of allyl chloride neurotoxicity in mice (1981)
    Springer
    Acta neuropathologica 55 (1981), S. 125-133 
    Details
    ISSN: 1432-0533
    Keywords: Allyl chloride ; Neurotoxicity ; Axonopathy ; Peripheral nerves
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Allyl chloride is known to produce a neuropathy in man after occupational exposure to its vapour. The present study describes the neuropathy which develops in mice given allyl chloride by mouth. Mice were dosed three times weekly with 300 or 500 mg/kg allyl chloride for periods from 2–17 weeks. Functional disability was observed in some animals. Apart from evidence of focal kidney damage in 70% of dosed mice, pathological changes were restricted to the nervous system. Nerve fibre degeneration was found in many peripheral nerves and in roots, tending to be more marked distally and to affect more motor than sensory nerves. Degenerated fibres were also found in dorsal, ventral and lateral columns of the spinal cord. Males were more severely affected than females. Increased numbers of filaments were an early axonal change, occurring multifocally and apparently preceding axonal degeneration. No neuronal death was observed, but occasional anterior horn and dorsal root ganglion cells showed some morphological changes. Vacuolated lesions mainly due to swelling of astrocytes and their processes were found in the ventral horn in cervical and lumbar regions of spinal cord. Animals appeared to become tolerant to allyl chloride after continuous dosing. This neuropathy appears to be a centralperipheral distal type of axonopathy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00699237
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