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  • 1
    Electronic Resource
    Electronic Resource
    Design of Hybrid Sandwich Panel with Aluminum Foam Core and Carbon Fiber Reinforced Plastic Face Sheets under Three-Point Bending (2006)
    s.l. ; Stafa-Zurich, Switzerland
    Solid state phenomena Vol. 111 (Apr. 2006), p. 63-66 
    Details
    ISSN: 1662-9779
    Source: Scientific.Net: Materials Science & Technology / Trans Tech Publications Archiv 1984-2008
    Topics: Physics
    Notes: Aluminum foams are very popular material for structural applications because of its attractive combination of properties. Structural performance of those foams can be enhanced by bonding them between strong and stiff face sheets such as carbon fiber reinforced plastics (CFRP). The response of hybrid sandwich panels comprising aluminum foam core and CFRP face sheets were investigated under three-point bending and measured response is verified with finite element numerical simulations. Core indentation and core shear, failure modes are identified. Experimentally measured elastic stiffness and failure load of thee tested beamswere found to be in good agreement with the numerical simulation and analytical predictions
    Type of Medium: Electronic Resource
    URL: http://www.tib-hannover.de/fulltexts/2011/0528/02/22/transtech_doi~10.4028%252Fwww.scientific.net%252FSSP.111.63.pdf
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  • 2
    Electronic Resource
    Electronic Resource
    Phenotypic characterization of Zimbabwean isolates of Pasteurella multocida
    Amsterdam : Elsevier
    Veterinary Microbiology 38 (1994), S. 351-357 
    Details
    ISSN: 0378-1135
    Keywords: Characterization ; Crocodile ; Dog ; Pasteurella multocida
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/0378-1135(94)90152-X
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  • 3
    Electronic Resource
    Electronic Resource
    Action of nitroimidazoles on algae I Anacytis nidulans
    Amsterdam : Elsevier
    Mutation Research/Environmental Mutagenesis and Related Subjects 53 (1978), S. 263-264 
    Details
    ISSN: 0165-1161
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0165-1161(78)90339-4
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  • 4
    Electronic Resource
    Electronic Resource
    Quaternary liquid-liquid equilibrium studies on hydrocarbon-solvent systems (1992)
    s.l. : American Chemical Society
    Journal of chemical & engineering data 37 (1992), S. 104-106 
    Details
    ISSN: 1520-5134
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology , Process Engineering, Biotechnology, Nutrition Technology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/je00005a027
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  • 5
    Electronic Resource
    Electronic Resource
    Conceptual design of a cellular filter to remove trace viral contaminants in blood (1990)
    s.l. : American Chemical Society
    Biotechnology progress 6 (1990), S. 104-113 
    Details
    ISSN: 1520-6033
    Source: ACS Legacy Archives
    Topics: Process Engineering, Biotechnology, Nutrition Technology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/bp00002a003
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  • 6
    Electronic Resource
    Electronic Resource
    Angiotensin II Type 1 Receptor mRNA Levels in the Brains of Normotensive and Spontaneously Hypertensive Rats (1993)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 60 (1993), S. 0 
    Details
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Abstract: The type 1 angiotensin II (All) receptor (AT1-R) has been implicated in the physiological actions mediated by All in the brain. In view of the reported hyperactivity of the brain All system in the spontaneously hypertensive rat (SHR), we compared the expression of AT,-R mRNAs in the brains of normotensive [Wistar Kyoto (WKY)] and SHR animals. Northern blot analysis showed about three- and ∼20-fold increases in the levels of AT1-R mRNAs from the hypothalamus and brainstem areas, respectively, of the SHR compared with the WKY rat brain. This was attributable to greater levels of both AT,1A- and AT,1B-R mRNA subtypes in these areas from the SHR. These observations suggest that increased All receptor levels in SHR brain may, in part, be a result of increased expression of the AT1-R gene.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1471-4159.1993.tb13426.x
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  • 7
    Electronic Resource
    Electronic Resource
    Metabolism of Angiotensin Peptides by Neuronal and Glial Cultures from Rat Brain (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 52 (1989), S. 0 
    Details
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Abstract: The degradation pattern and rate of [Ile5]-Angio-tensin (Ang) I, II, and III were studied in neuron-enriched and glia-enriched cells in primary cultures from rat brain. Metabolites were separated by HPLC, and their identities were evaluated by comparison of their retention times with those of synthetic Ang peptide fragments and by analysis of their amino acid composition. Major metabolites were identified as des-Asp1-[Ile5]-Ang I, des-Asp1-[Ile5]-Ang II, [Ile5]-Ang II (3–8) hexapeptide, [Ile5]-Ang II (4–8) pentapeptide, and [Ile5]-Ang II (5–8) tetrapeptide. Glia-enriched cells degraded [Ile5]-Ang I and [Ile5]-Ang III significantly faster than neuron-enriched cells, whereas no difference between the two types of cells was found in the degradation rate of [Ile5]-Ang II. Although the half-lives of [Ile5]-Ang I and [Ile5]-Ang III in neuron-enriched cells from normotensive Wistar-Kyoto (WKY) rats and spontaneously hypertensive rats (SHR) were not significantly different, neuron-enriched cultures from WKY rats metabolized [Ile5]-Ang II about 2.6 times faster than neuron-enriched cells derived from SHR.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1471-4159.1989.tb02534.x
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  • 8
    Electronic Resource
    Electronic Resource
    Phorbol Ester-Induced Upregulation of Angiotensin II Receptors in Neuronal Cultures Is Potentiated by a Calcium Ionophore (1988)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 51 (1988), S. 0 
    Details
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Previous studies have suggested that protein ki-nase C is important in the regulation of angiotensin II receptors in neuronal cultures, because the C-kinase agonists, phorbol esters, are able to increase the number of these receptors. In the present study, we have further investigated the role of protein kinase C in angiotensin II receptor regulation. This enzyme is calcium dependent, and so we investigated the effects of A23187, a calcium ionophore, on phorbol ester-stimulated and basal angiotensin II receptor regulation. A23187, at concentrations that increased 45Ca2+ influx, caused a dose-dependent potentiation of phorbol-12-myristate-13-acetate (TPA)-stimulated upregulation of angiotensin II receptors. This potentiation by A23187 was a further increase in angiotensin II receptor number and was abolished in calcium-free medium. In the absence of TPA, A23187 caused a decrease in angiotensin II receptor number, an effect not observed in calcium-free medium. The results suggest at least two pathways for angiotensin II receptor regulation in neuronal cells: (a) by calcium-dependent protein kinase C and (b) via an influx of calcium into the cell.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1471-4159.1988.tb04849.x
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  • 9
    Electronic Resource
    Electronic Resource
    Protein Kinase C Agonists Increase the Expression of Angiotensin II Receptors in Neuronal Cultures (1987)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 48 (1987), S. 0 
    Details
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Abstract: Previous studies have shown that norepinephrine is important in the regulation of central angiotensin II receptors, an effect mediated by α1-adrenergic receptors. Because α1-adrenergic stimulation leads to inositol phospholipid hydrolysis and activation of protein kinase C, we have examined a possible role of this enzyme in the regulation of central angiotensin II (Ang II) receptors. In the present study, we have examined the effects of protein kinase C activators, phorbol esters, on the expression of Ang II receptors in neuronal cultures prepared from 1-day-old rat brains. The active phorbol ester phorbol- 12-myristate-13-acetate (TPA) caused time- and concentration-dependent increases in the specific binding of [125I]Ang II to its receptors in neuronal cultures of normotensive and spontaneously hypertensive rat brains. The stimulatory effect of TPA on Ang II receptors was apparent within 15 min and reached a maximum between 1 and 2 h. Ang II specific binding had returned to control levels by 24 h. Various phorbol esters increased [125I]Ang II binding in accordance with their order of potency in stimulating protein kinase C activity. Saturation and Scatchard analysis revealed that the phorbol ester-induced increase in [125I]Ang II binding was due to an increase in the number of Ang II receptors. These observations indicate that activation of protein kinase C results in an increased expression of Ang II receptors in neuronal cultures from both normotensive and spontaneously hypertensive rat brains. The results suggest a possible role of phosphorylation in Ang II receptor expression in neuronal cultures.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1471-4159.1987.tb05760.x
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  • 10
    Electronic Resource
    Electronic Resource
    α1-Adrenergic Receptors in Neuronal Cultures from Rat Brain: Increased Expression in the Spontaneously Hypertensive Rat (1986)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 47 (1986), S. 0 
    Details
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Abstract Neuronal cells in primary culture from 1-day-old brains of normotensive, Wistar-Kyoto strain (WKY) and spontaneously hypertensive (SH) rats have been utilized to study the expression of α1-adrenergic receptors. Binding of a selective α1 antagonist, [125I]2-[β-(4-hydroxy-3-iodophenyl)-ethylaminomethyl]-tetralone ([125I]HEAT) to neuronal membranes prepared from primary brain cultures of WKY and SH rats was 75–80% specific, rapid, and time-dependent although the binding was 1.5–2 times higher in neuronal membranes from SH rat brain cultures. Kinetic analysis of the association and dissociation data demonstrated no significant differences between rat strains. Competition-inhibition experiments provided IC50 values for various antagonists and agonists in the following order: prazosin 〈 phentolamine 〈 yohimbine 〈 phenylephrine 〈 norepinephrine 〈 propranolol, suggesting that [125I]-HEAT bound selectively to α1-adrenergic receptors. Scatchard analysis of the binding data provided straight lines for both strains of rats, indicating the presence of a homogeneous population of binding sites. It also showed that the increase in the binding in neuronal cells from SH rat brains over those from normotensive WKY controls was a result of an increase in the number of α1-adrenergic receptors. Incubation of neuronal cultures from both strains of rats with phenylephrine, an α1-adrenergic agonist, caused a time- and dose-dependent decrease in the binding of [125I]HEAT. This decrease was due to a decrease in the number of α1-adrenergic receptors. In conclusion, we demonstrated that the expression of α1-adrenergic receptors in neuronal cultures of SH rat brain was increased compared with neuronal cultures from normotensive controls. We suggest that the elevated number of these receptors in neurons from the brains of SH rats is an intrinsic property of these cells and therefore provides an etiological basis for the predisposition of these animals to the hypertensive state.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1471-4159.1986.tb00739.x
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