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  • 1
    Electronic Resource
    Electronic Resource
    Epidural anaesthesia and spinal arachnoiditis (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Anaesthesia 44 (1989), S. 0 
    Details
    ISSN: 1365-2044
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Six patients were referred to our hospital with spinal arachnoiditis after epidural anaesthesia performed one month to 3 years before the onset of symptoms. None had had previous lumbar surgery or trauma, intraspinal haemorrhage, infections or other known causative factors of arachnoiditis. All the patients were free of neurological symptoms before epidural anaesthesia and only two had transient distress in the period immediately following the procedure. The clinical signs and symptoms of spinal arachnoiditis were severe and in every case the diagnosis was confirmed by myelography. Three patients were confined to a wheelchair after 3 years of follow-up. To our knowledge, the anaesthetic procedures were performed according to standard methods. Arachnoiditis seems to be due to the epidural injection of foreign substances, and may be related to anaesthetic–vasoconstrictor solution or contaminants.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2044.1989.tb11285.x
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  • 2
    Electronic Resource
    Electronic Resource
    Spinal arteriovenous fistula (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Anaesthesia 44 (1989), S. 0 
    Details
    ISSN: 1365-2044
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: A 62-year-old male suddenly developed a severe paraparesis after epidural anaesthesia. He recovered gradually over the next few months. He had an acute relapse one year later and a selective spinal angiography showed a dural T8 arteriovenous fistula with large draining veins. Intravascular embolisation of the fistula produced immediate and sustained clinical improvement. The mechanism commonly held responsible for neurological disturbances in spinal dural arteriovenous fistulas is cord hypoxia secondary to venous hypertension. The 20-ml of local anaesthetic solution injected into a narrow spinal canal with osteophytosis may have caused further venous engorgement, cord hypoxia and acute neurological deficit.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2044.1989.tb09101.x
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  • 3
    Electronic Resource
    Electronic Resource
    A reply (1990)
    Oxford, UK : Blackwell Publishing Ltd
    Anaesthesia 45 (1990), S. 0 
    Details
    ISSN: 1365-2044
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2044.1990.tb14758.x
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  • 4
    Electronic Resource
    Electronic Resource
    The Course of Myasthenia Gravis in Patients Treated with Corticosteroids, Azathioprine, and Plasmapheresis (1987)
    Oxford, UK : Blackwell Publishing Ltd
    Annals of the New York Academy of Sciences 505 (1987), S. 0 
    Details
    ISSN: 1749-6632
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Natural Sciences in General
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1749-6632.1987.tb51320.x
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  • 5
    Electronic Resource
    Electronic Resource
    Cisplatin neuropathy in brain tumor chemotherapy (1992)
    Springer
    Neurological sciences 13 (1992), S. 311-315 
    Details
    ISSN: 1590-3478
    Keywords: Cisplatin ; peripheral nerve diseases ; chemotherapy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario Trentotto soggetti portatori di tumori cerebrali della serie gliale sono stati trattati con 5 cicli mensili di cisplatino, ciascuno di 135 mg/m2, per via endovenosa. Segni e sintomi di neuropatia periferica sono stati valutati clinicamente ed elettrofisiologicamente. Il nostro approccio differisce dai precedenti lavori riportati in letteratura in quanto offre due importanti vantaggi: i tumori cerebrali primitivi non causano mai neuropatie paraneoplastiche; non è stato usato nessun altro farmaco neurotossico all'infuori del cisplatino. Lo studio conferma l'esistenza della neuropatia periferica indotta dal cisplatino anche in questo modello altamente specifico. Abbiamo potuto osservare chiari segni clinici di neuropatia periferica nell'80% dei pazienti trattati con una dose cumulativa di cisplatino di 675 mg/m2. Dal punto di vista eletrofisiologico si è osservata una riduzione progressiva e dose-correlata nell'ampiezza dei SAP, espressione di una neuropatia sensitiva assonale e/o neuronale.
    Notes: Abstract 38 patients with glial brain tumors received 135 mg/m2 cisplatin intravenously every month for 5 courses. Signs and symptoms of peripheral neuropathy were evaluated clinically and electrophysiologically. This approach differs from methods previously reported in that it offers two major advantages: primary brain tumors do not cause paraneoplastic neuropathy; no neurotoxic drugs other than cisplatin were employed. Our study confirmed in this highly specific clinical model the existence of cisplatin peripheral neuropathy: we observed definite clinical signs or symptoms of neuropathy in 80% of patients receiving a cumulative cisplatin dosage of 675 mg/m2; there was a progressive dose-related decrease in SAP amplitudes, expression of a sensory axonopathy or neuronopathy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02223095
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  • 6
    Electronic Resource
    Electronic Resource
    Neurological examination in patients recovering from general anesthesia (1992)
    Springer
    Neurological sciences 13 (1992), S. 749-753 
    Details
    ISSN: 1590-3478
    Keywords: General anesthesia ; recovery ; neurological examination ; post-hyperventilation apnea
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario Abbiamo sottoposto a valutazione neurologica fino a 3 ore dal termine dell'erogazione dei farmaci anestetici (Isoflurane-N20-02 per 25 pazienti, Alotano-N20-02 per 38, Fentanyl-Droperidolo-N20-02 per 23) 86 pazienti operati per asportazione di disco intervertebrale lombare erniato. Al tempo 0 la risposta idiomuscolare da percussione del muscolo estensore del carpo era presente in tutti i case mentre erano sempre assenti i riflessi osteotendinei. L'iperreflessia si è presentata con frequenza analoga a quella del brivido, anche se più precocemente. Non siamo stati comunque in grado di evidenziare una correlazione tra i due fenomeni. Non c'è stata relazione alcuna tra temperatura rettale e brivido. La positività alle prove di post-hyperventilationapnoea a 120 minuti in 35 pazienti, nonostante la quasi totalità della popolazione fosse in grado di collaborare ed eseguire correttamente prove di calcolo (100–7), suggerisce una persistenza di anomalie neurologische, quali l'inadeguato “drive” respiratorio, in pazienti considerati “svegli”.
    Notes: Abstract We performed serial neurological evaluations on 86 patients who underwent surgery for herniated lumbar disk during the first 3 hours after the end of anesthesia (isoflurane-N20-02 for 25 patients, halothane-N20-02 for 38, fentanyl-droperidol-N20-02 for 23). At time 0, the idiomuscular response to percussion of the extensor carpi muscle was present in every patient, while tendon reflexes were always absent. Hyperreflexia was as frequent as shivering, but it took place earlier; we could determine no correlation between these two phenomena. There was no correlation between shivering and rectal temperature of the patients. Although almost all the patients were cooperative and could correctly calculate 100–7, the post-hyperventilation-apnea test was positive in 35 patients at time 120 minutes: this suggests that many patients considered “awake” still exhibit neurological abnormalities, such as inadequate respiratory drive.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02229160
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  • 7
    Electronic Resource
    Electronic Resource
    Combined central and peripheral acute demyelination (1993)
    Springer
    Neurological sciences 14 (1993), S. 83-86 
    Details
    ISSN: 1590-3478
    Keywords: Polyradiculoneuritis ; multiple sclerosis ; myelin sheath ; demyelinating diseases ; peripheral neuropathy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario In una paziente portatrice di sclerosi multipla una poussée di demielinizzazione centrale si associa ad una polineuropatia demielinizzante infiammatoria acuta. Il contemporaneo coinvolgimento del sistema nervoso centrale e periferico da parte di una malattia demielinizzante è stato riportato sporadicamente nell'uomo, ma può essere sperimentalmente indotto nell'animale e può essere sostenuto da un fattore patogenetico comune.
    Notes: Abstract We describe a patient with multiple sclerosis who had a bout of central demyelination associated with an acute inflammatory demyelinating polyneuropathy. The contemporary involvement of central and peripheral nervous system due to a demyelinating disease has been reported anecdotically in humans, and can be induced experimentally in animals. It may be sustained by a common pathogenetic factor.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02339047
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  • 8
    Electronic Resource
    Electronic Resource
    Carbamazepine for paroxysmal distonia due to spinal cord lesions (1993)
    Springer
    Neurological sciences 14 (1993), S. 185-187 
    Details
    ISSN: 1590-3478
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02335754
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  • 9
    Electronic Resource
    Electronic Resource
    Homozygous hypertrophic hereditary motor and sensory neuropathies (1994)
    Springer
    Neurological sciences 15 (1994), S. 5-14 
    Details
    ISSN: 1590-3478
    Keywords: hereditary motor and sensory neuropathy ; Charcot-Marie-Tooth disease ; Dejerine-Sottas disease ; demyelination ; hypomyelination ; 17p11.2 duplication
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario Abbiamo paragonato 25 pazienti affetti da neuropatia ereditaria sensitivo-motoria (HMSN) di tipo I ad ereditarietà autosomica dominante con 7 pazienti affetti da neuropatia ipertrofica ad ereditarietà non dominante. Tutti i pazienti con ereditarietà autosomica dominante sono risultati portatori della duplicazione 17p11.2, dimostrando così che questa è largamente presente nelle famiglie HMSN I. Il secondo gruppo comprendeva: due fratelli affetti da una neuropatia ipertrofica di gravità nettamente diversa, nati da genitori sani non consanguinei; due sorelle con un fenotipo HMSN I nate da genitori sani, cugini di primo grado; due fratelli con un fenotipo HMSN III nati da genitori non consanguinei affetti da HMSN II; un bimbo con un classico fenotipo HMSN III, nato da genitori sani, non consanguinei. L'analisi del DNA ha dimostrato l'assenza della duplicazione sia nei pazienti del secondo gruppo che nei loro genitori. I nostri dati dimostrano che: l'HMSN III è eterogenea e comprende pazienti omozigoti per differenti geni neuropatogeni; è opportuno tenere distinti i pazienti con neuropatia ipertrofica recessiva da quelli portatori di una HMSN Ia dominante poiché è probabile che le due malattie siano dovute a difetti genici diversi.
    Notes: Abstract We compared 25 autosomal dominant hereditary motor and sensory neuropathy (HMSN) type I patients with 7 subjects affected by hypertrophic HMSN with non-dominant inheritance. All the autosomal dominant HMSN I cases carried the chromosome 17p11.2 duplication, providing evidence that it is widely represented in HMSN I families. The second group included: Two siblings born to unrelated, unaffected parents and suffering from hypertrophic HMSN of strikingly different severity; two sisters with HMSN I phenotype, born to first-cousin unaffected parents; two brothers with HMSN III phenotype born to unrelated parents both showing HMSN II phenotype; a child with classic HMSN III phenotype, born to unrelated, unaffected parents. The 17p11.2 duplication was not found in any of the patients of the second series or in their parents. Our data provide further evidence that: HMSN III is heterogeneous and encompasses the homozygous expressions of different neuropathic genes; it is advisable to separate autosomal recessive hypertrophic HMSN from dominant HMSN Ia, because they appear to be due to different DNA mutations.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02343492
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  • 10
    Electronic Resource
    Electronic Resource
    The last days of Alessandro Manzoni (1995)
    Springer
    Neurological sciences 16 (1995), S. 199-202 
    Details
    ISSN: 1590-3478
    Keywords: history of neurology ; subdural hematoma
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario Alessandro Manzoni è morto il 22 maggio 1873 a 88 anni compiuti. La mattina del 6 gennaio 1873, mentre si recava alla Messa nella chiesa milanese di San Fedele, era caduto sulla gradinata e aveva battuto la fronte sugli scalini. Dalle cronache dell'epoca risulta che l'incidente, nella sua apparente banalità, è risultato fatale al vecchio scrittore. Sembra probabile che la morte sia stata causata da un ematoma subdurale cronico.
    Notes: Abstract Alessandro Manzoni died on 22 May 1873 at the age of 88. On the morning of 6 January 1873, on his way to Mass in the Milanese church of San Fedele, he fell and hit his head on the church steps. From the newspaper accounts of the day, it appears that this apparently banal accident was fatal for the old writer. It seems likely that his death was caused by a chronic subdural hematoma.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02282988
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