Library

feed icon rss

Your email was sent successfully. Check your inbox.

An error occurred while sending the email. Please try again.

Proceed reservation?

Export
  • 1
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    FEBS Letters 184 (1985), S. 52-55 
    ISSN: 0014-5793
    Keywords: Myeloperoxidase ; Raman spectrum
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Physics
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 2
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    FEBS Letters 202 (1986), S. 245-250 
    ISSN: 0014-5793
    Keywords: (Bovine spleen) ; Heme protein ; Myeloperoxidase
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Physics
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 3
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    FEBS Letters 214 (1987), S. 111-116 
    ISSN: 0014-5793
    Keywords: (Bovine spleen) ; Heme-protein ; Myeloperoxidase ; NMR
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Physics
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 4
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Protein Structure and Molecular 873 (1986), S. 62-72 
    ISSN: 0167-4838
    Keywords: (Bovine spleen) ; Circular dichroism ; Magnetic circular dichroism ; Myeloperoxidase ; Spleen green hemeprotein
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 5
    Electronic Resource
    Electronic Resource
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Protein Structure and Molecular 1076 (1991), S. 317-320 
    ISSN: 0167-4838
    Keywords: Cyanide complex ; EPR ; Enzyme catalysis ; Halide ; Myeloperoxidase
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 6
    Electronic Resource
    Electronic Resource
    Springer
    Journal of neurology 216 (1977), S. 79-89 
    ISSN: 1432-1459
    Keywords: Normal muscle spindle ; Histochemistry ; Intrafusal fiber types ; Fusimotor endings ; Fusisensory endings
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Es wurden normale Skeletmuskelspindeln von Menschen histochemisch untersucht. 1. Histochemisch wurden vier Muskelfasertypen der intrafusalen Fasern — Bag I fiber, Bag II fiber, Chain I fiber und Chain II fiber — von ATPase-Aktivität klassifiziert. Bei der NADH-Tetrazolium-Reduktase wurden zwei Muskelfasertypen an intrafusalen Fasern — Bag I fiber und Bag II fiber — beobachtet. 2. Drei Typen der motorischen Endplatte — en plaque Endplatte, en grappe Endplatte und diffuse Endplatte — wurden an Muskelspindeln von Cholinesterase klassifiziert. 3. Zwei Typen der sensorischen Nervenenden — primär-sensorisches Nervenende und sekundärsensorisches Nervenende — wurden an Muskelspindeln mit NADH-Tetrazolium-Reduktase und elektronmiktroskopisch beobachtet.
    Notes: Summary Normal muscle spindles of human skeletal muscle were studied histochemically. 1) Four histochemical types of intrafusal muscle fibers were classified by ATPase stain: Bag I fiber, Bag II fiber, Chain I fiber and Chain II fiber. Moreover, two types of nuclear bag fibers were classified by NADH Tetrazolium Reductase stain and PAS stain: Bag I fiber and Bag II fiber. 2) Three kinds of fusimotor endings were verified by the cholinesterase technic: en plaque, en grappe and diffuse endings. 3) Two kinds of fusisensory endings were verified by NADH TR stain and also electron-microscopically: primary and secondary sensory endings.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 7
    ISSN: 1420-908X
    Keywords: Cartilage ; Arthritis ; Nitric oxide ; Interleukin-1
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract To determine the role of nitric oxide (NO) in the inhibition of aggrecan synthesis, we measured levels of NO produced by bovine chondrocytes from different layers of articular cartilage in the presence of interleukin-1 (IL-1). Chondrocytes from the superficial layer showed a large increase in NO synthesis in response to IL-1. Although chondrocytes from the deep layer also produced NO in response to IL-1, the amount was less than that from the superficial layer. Enhanced NO production evoked by IL-1 was accompanied by a significant inhibition of aggrecan synthesis. These data suggest that chondrocytes in both superficial and deep layer of articular cartilage inhibit aggrecan synthesis with IL-1 via NO production. In addition, superficial layer cells respond to lower amounts of IL-1 with respect to NO-production and inhibition of proteoglycan synthesis.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 8
    Electronic Resource
    Electronic Resource
    Springer
    Journal of neurology 219 (1978), S. 261-271 
    ISSN: 1432-1459
    Keywords: Fusimotor endings ; Acetylcholinesterase ; Parkinsonism ; Motor neuron disease ; Myasthenia ; Muscle spindles
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung In Serienschnitten wurden die motorischen Endplatten der intrafusalen Muskelfasern in Muskelspindeln von Patienten mit Parkinsonismus, myatrophischer Lateralsklerose und Myasthenia gravis mittels Acetylcholinesterasereaktion untersucht. Beim Parkinsonismus erschienen die diffusen Endigungen, von welchen angenommen wird, daß sie die Endigungen des Gammafasersystemes darstellen, nennenswert verbreitert, während die en-plaque- und en-grappe-Endigungen atrophisch erschienen. In den Fällen von myatrophischer Lateralsklerose erwiesen sich en-plaque- und en-grappe-Endigungen, von welchen angenommen wird, daß sie zu den Betaneuronen und den dynamischen Gammafasern gehören, als ausgesprochen atrophisch. Die diffusen Endigungen allerdings waren normal. Bei Myasthenia gravis und dem myasthenischen Syndrom von Eaton-Lambert zeigten en-plaque- und engrappe-Endigungen Atrophien, während einzig die diffusen Endigungen ausgespart blieben. Es wird die Bedeutung dieser Veränderungen diskutiert.
    Notes: Summary Pathological changes of the fusimotor endings in parkinsonism, motor neuron disease and myasthenia were examined by the acetylcholinesterase technic on serial sections. In parkinsonism, the diffuse endings, which are thought to be supplied by the static gamma nerve fibers, showed remarkable enlargement, while en plaque and en grappe endings were atrophic. In motor neuron disease, en plaque and en grappe endings, which are thought to be innervated by the beta nerve fibers and dynamic gamma nerve fibers respectively, revealed marked atrophy. However the diffuse endings were normal. In myasthenia gravis and myasthenic syndrome (Eaton-Lambert syndrome), en plaque and en grappe endings were atrophic, though only the diffuse endings were spared. The significance of these changes in the fusimotor endings is discussed.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
  • 9
    Electronic Resource
    Electronic Resource
    Springer
    Urological research 28 (2000), S. 141-146 
    ISSN: 1434-0879
    Keywords: Key words Kidney ; Nitric oxide ; Ischemia-reperfusion injury ; Rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract In this study we attempted to clarify the release of nitric oxide (NO) and its role in the ischemia-reperfusion rat kidney. After right nephrectomy, male Wistar rats were divided into four groups: one sham operated and three groups who underwent ischemia (30 min) and reperfusion of the left renal artery. Thirty minutes prior to ischemia-reperfusion, two groups were injected intraperitoneally with 10 and 30 mg/kg of NG-nitro-l-arginine methylester (L-NAME). Real-time monitoring of blood flow and NO release in the rat kidney was measured with a laser Doppler flowmeter and an NO-selective electrode, respectively. Serum creatinine and blood urea nitrogen (BUN) levels were measured 1 and 7 days after the induction of ischemia-reperfusion. Clamping of the renal artery decreased blood flow to 1–5% of the basal level measured before clamping. After removal of the clip, the blood flow of the 30 mg/kg L-NAME rats was significantly lower than that of the controls. Immediately following the clipping of the renal artery, NO release rapidly increased. After removing the clip, NO release immediately returned to three-quarters of the basal level. Serum creatinine and BUN levels of the ischemia-reperfusion rats were slightly but not significantly higher and those of 30 mg L-NAME rats were significantly higher than those of the control or ischemia-reperfusion rats 1 day and 7 days after ischemia-reperfusion. Our data suggest that NO acts as a cytoprotective agent in ischemia-reperfusion injury of the rat kidney.
    Type of Medium: Electronic Resource
    Library Location Call Number Volume/Issue/Year Availability
    BibTip Others were also interested in ...
Close ⊗
This website uses cookies and the analysis tool Matomo. More information can be found here...