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  • Triiodothyronine  (2)
  • gastric damage  (2)
  • Adenosine  (1)
  • Dietary lipid  (1)
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  • 1
    Digitale Medien
    Digitale Medien
    The gastric cytoprotective action of adenosine and prostaglandin E2 in rabbits (1994)
    Springer
    Inflammation research 42 (1994), S. 146-148 
    Details
    ISSN: 1420-908X
    Schlagwort(e): Adenosine ; Prostaglandin E2 ; Gastric gland ; Damage
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract The direct protective action of adenosine and prostaglandin E2 (PGE2) was examined in an isolated gastric gland preparation in rabbits. Ethanol, (8%, v/v) incubation markedly increased the release of lactate dehydrogenase (LDH) and number of non-viable glands in the preparation. Both effects were prevented by PGE2 preincubation in a concentration (10−6, 1.4×10−5 or 2.8×10−5 M)-dependent manner. The protective action was smaller in adenosine-treated groups, and yet the highest concentration (10−4 M) of the compound also significantly inhibited the cytotoxic effects of ethanol. These findings indicate that both adenosine and PGE2 possess cytoprotective action on gastric glands in rabbits, but the former compound exerts its action beyond physiological concentrations. It is concluded that endogenous PGE2, but not adenosine may act as an ulcer modulator in the stomach.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF01983481
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  • 2
    Digitale Medien
    Digitale Medien
    Effect of dietary cholecalciferol deficiency on plasma thyroid hormone concentrations in rainbow trout,Salmo gairdneri (Pisces, Salmonidae) (1980)
    Springer
    Environmental biology of fishes 5 (1980), S. 167-173 
    Details
    ISSN: 1573-5133
    Schlagwort(e): Vitamin D ; Thyroid gland ; Growth ; Endocrines ; Trout ; Calcium ; Thyroxine ; Triiodothyronine
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Biologie
    Notizen: Synopsis Diets deficient in vitamin D effected a significant increase in plasma triiodothyronine (T3) concentration in raibow trout (Salmo gairdneri); different levels of dietary calcium exerted no effect on plasma T3 levels. These effects of vitamin D deficiency on plasma T3 levels appeared to be reversible, vitamin D supplementation after a period of vitamin D deficiency lowered T3 levels. Vitamin D3, vitamin D2 and the metabolites 25(OH)-D3 and 1, 25(OH)2D3 were all effective in lowering plasma T3 levels; vitamin D3 appeared to be more effective than vitamin D2. There appeared to be a correlation between weight gain and plasma T3 concentration in the groups fed different types and levels of vitamin D supplementation suggesting that the increased T3 levels may be a compensatory increase to the reduced weight gain of the vitamin D deficient fish. Plasma T4 levels were not affected by dietary vitamin D deficiency.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF02391624
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  • 3
    Digitale Medien
    Digitale Medien
    Further studies on the effect of diet on serum thyroid hormone concentrations and thyroid histology in rainbow trout,Salmo gairdneri (Pisces, Salmonidae) (1980)
    Springer
    Environmental biology of fishes 5 (1980), S. 175-179 
    Details
    ISSN: 1573-5133
    Schlagwort(e): Keywords ; Dietary protein ; Dietary lipid ; Endocrinology ; Vitamin C ; Thyroxine ; Triiodothyronine
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Biologie
    Notizen: Synopsis In a 3 × 2 factorial experiment examining the effects of combinations of ambient temperature (18°, 15°, 9° C) and dietary protein content (35% and 45%) on thyroid activity inSalmo gairdneri, although there was an apparent increase in activity of the thyroid in cold-adapted trout, assessed by histological appearance of the gland, there were no significant changes in serum thyroid hormone titers. In a second experiment examining the effects of combinations of ambient temperature (15°, 12.5°, 10°C) with dietary lipid content (6% and 16%) there was a similar apparent increase in thyroid activity in cold-adapted fish which was accompanied, in fish fed the higher lipid diet, with an increase in serum thyroxine (T4) and triiodothyronine (T3) levels. Trout fed an ascorbic acid-free diet (experiment 3) had lower serum T3 levels than in those given an ascorbic acid supplemented diet (1280 mg·kg-1). In experiments 2 and 3 serum thyroid hormone concentrations were approximately inversely proportional to ambient temperature and concomitant weight gain, but no such correlation was evident in experiment 1 suggesting that the changes in hormone levels in experiments 2 and 3 were not ipso facto related to differences in either ambient temperature or weight gain but rather to the specific metabolic changes imposed by the dietary lipids or ascorbic acid deficiency.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF02391625
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  • 4
    Digitale Medien
    Digitale Medien
    Portal hypertension (1993)
    Springer
    Digestive diseases and sciences 38 (1993), S. 2203-2208 
    Details
    ISSN: 1573-2568
    Schlagwort(e): portal hypertension ; gastric function ; ethanol ; gastric damage
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract The time-course effects of portal hypertension on gastric secretory function, mucosal blood flow, vascular permeability, and ethanol-induced gastric mucosal damage were examined in anesthetized rats. Partial ligation of the portal vein effectively produced portal hypertension one to three days later but the raised pressure returned to normal on the sixth day after ligation. This time-course effect coincided with reduced pepsin secretion and mucosal blood flow and also with potentiated ethanol-induced mucosal damage during the first to third days. These effects started to tail off on the sixth day. However, gastric acid output was significantly reduced on the third day, and this was strongest on the sixth day after operation. Portal vein ligation also reduced basal vascular permeability, which was markedly potentiated after ethanol treatment. It is concluded that: (1) portal vein blood pressure changes are a time-dependent process following ligation; (2) changes in gastric mucosal blood flow (GMBF) and lesion formation are closely related to portal hypertension; (3) gastric mucosal injury is associated with vascular damage, as evidenced by increased in vascular permeability; and (4) pepsin but not acid secretion is closely related to the state of the GMBF.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF01299896
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  • 5
    Digitale Medien
    Digitale Medien
    Role of dorsal motor nucleus of vagus in gastric function and mucosal damage induced by ethanol in rats (1995)
    Springer
    Digestive diseases and sciences 40 (1995), S. 2312-2316 
    Details
    ISSN: 1573-2568
    Schlagwort(e): dorsal motor nucleus of vagus ; ethanol ; gastric mucosal blood flow ; gastric acid secretion ; gastric damage
    Quelle: Springer Online Journal Archives 1860-2000
    Thema: Medizin
    Notizen: Abstract Experimental evidence indicates that the autonomic nervous system, especially the cholinergic pathway, modulates the mucosal defensive mechanism and affects mucosal damage in the stomach. The present study investigated the role of the dorsal motor nucleus of vagus (DMV) in gastric function and its influences on ethanol-induced mucosal damage in pentobarbitone-anesthetized rats. Electrolytic lesion of the DMV as compared with sham operation and lesions of other brain areas, eg, nucleus reticular gigantocellularis and cuneate nucleus, reduced the basal gastric mucosal blood flow (GMBF) and also the blood flow after ethanol administration. The same operation did not affect the acid secretion either in the basal state or during the ethanol treatment period. Lesions at the caudal half of the DMV produced a bigger depression of GMBF when compared with lesion at the rostral half. In the sham-operated rats, ethanol induced severe hemorrhagic lesions in the gastric glandular mucosa, and this was significantly potentiated by lesions at the DMV, especially in the caudal half. The present findings indicate that acute DMV damage at the caudal half markedly affects the GMBF but not the acid secretion. The action on GMBF may contribute to the aggravation of ethanol-induced gastric damage in rats. These data reinforce the idea that the central vagal pathway, especially the caudal half of the DMV, plays a significant role in the modulation of GMBF, which in turn affects the integrity of gastric mucosal barrier.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1007/BF02063230
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