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  • 1
    Electronic Resource
    Electronic Resource
    Capillary haemangiomatosis of the lungs (1978)
    Oxford, UK : Blackwell Publishing Ltd
    Histopathology 2 (1978), S. 0 
    Details
    ISSN: 1365-2559
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: A case of capillary haemangiomatosis affecting both lungs. The angiomatous growth involved particularly the intralobular fibrous septa and appeared locally aggressive as it had destroyed and obstructed the pulmonary veins and venules. Some pulmonary arteries and bronchi were affected to a lesser extent. Pulmonary venous obstruction, sometimes simulating pulmonary veno-occlusive disease, caused severe haemosiderosis and interstitial fibrosis. The clinical features and the lung biopsy erroneously suggested a diagnosis of primary pulmonary haemosiderosis. No comparable case has been found in the literature.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2559.1978.tb01734.x
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Grading of pulmonary vascular lesions-a reappraisal (1981)
    Oxford, UK : Blackwell Publishing Ltd
    Histopathology 5 (1981), S. 0 
    Details
    ISSN: 1365-2559
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Grading of pulmonary vascular alterations, introduced by Heath & Edwards (1958), has been widely used for assessment of the severity of hypertensive pulmonary vascular disease. Two factors call for a reappraisal of the grading principle: an increasing awareness of the complexity of the vascular lesions and an increasing use of lung biopsies for the pre-operative evaluation of the operability of an underlying cardiac defect. We suggest that the grading system, as well as the previously proposed variations of it, no longer fulfil the requirements of unambiguous assessment of the severity of vascular disease. The degree and extent of the various lesions, the different types of intimal fibrosis, and the eventual decrease in number and size of vessels should all be assessed, not only for arteries but also for other vessels. Careful consideration and weighing of all these features are necessary in order to form an opinion on diagnosis and prognosis. This cannot be achieved by using a single grade.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2559.1981.tb01826.x
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Mediastinal and hilar fibrosis (1981)
    Oxford, UK : Blackwell Publishing Ltd
    Histopathology 5 (1981), S. 0 
    Details
    ISSN: 1365-2559
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: A case is described of mediastinal and hilar fibrosis in a woman aged 22 years. The fibrotic mass compressed the lobar arteries as well as the veins of various lobes of both lungs. These large vessels as well as numerous smaller arteries and veins were to a large extent obstructed by organized thrombi. It seems likely that 3 years after the beginning of symptoms the fibrosing process was still active. The case provides some support for an immunopathological aetiology of this condition.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2559.1981.tb01766.x
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    Paper (German National Licenses)
    Fulltext
  • 4
    Electronic Resource
    Electronic Resource
    Pulmonary capillary haemangiomatosis diagnosed during life (1989)
    Oxford, UK : Blackwell Publishing Ltd
    Histopathology 14 (1989), S. 0 
    Details
    ISSN: 1365-2559
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2559.1989.tb02131.x
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    The ultrastructure of the various forms of pulmonary arterial intimal fibrosis (1979)
    Springer
    Virchows Archiv 382 (1979), S. 139-150 
    Details
    ISSN: 1432-2307
    Keywords: Intimal fibrosis of pulmonary arteries ; Ultrastructure of intima ; Intimal smooth muscle cells
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Intimal fibrosis of muscular pulmonary arteries may present in various forms and in varying degrees of severity according to the underlying condition. In patients with pulmonary hypertension, the type of intimal fibrosis is often significant with regard to prognosis and reversibility. For these reasons we have studied the ultrastructure of the thickened intimal layer in aged individuals, where intimal fibrosis occurs as a normal age change, and in patients with pulmonary hypertension associated with fibrosis of the lungs, mitral stenosis, chronic pulmonary thromboembolism and plexogenic pulmonary arteriopathy (either primary or secondary to congenital cardiac defects). In all these forms of intimal fibrosis, the cellular component of the subendothelial intimal layer was apparently almost exclusively the smooth muscle cell. These cells usually had a haphazard arrangement. In primary and secondary plexogenic pulmonary arteriopathy, however, there was a more regular circumferential arrangement. The ultrastructural evidence suggested that the intimal cells were derived from medial smooth muscle cells.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01102870
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    The pathology of pulmonary veno-occlusive disease (1974)
    Springer
    Virchows Archiv 364 (1974), S. 69-79 
    Details
    ISSN: 1432-2307
    Keywords: Pulmonary Veno-Occlusive Disease ; Pulmonary Venous Lesions ; Pulmonary Arterial Lesions ; Interstitial Pulmonary Fibrosis ; Interstitial Pneumonia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The histopathology of the lungs is described in 13 patients with pulmonary veno-occlusive disease. The patients were mostly children or young adults and there was no sex preference as is seen in primary pulmonary hypertension. Characteristically the small pulmonary veins and venules were narrowed or occluded by intimal fibrosis, but larger veins were also affected. These obstructive lesions almost certainly resulted from organization of thrombi. Changes in the pulmonary arteries were generally secondary to obstruction of pulmonary venous flow but there was also a high incidence of thrombotic changes in the arteries. Pulmonary parenchymal lesions commonly found included interstitial pneumonia and fibrosis, siderosis and cellular proliferation, together with increased activity of bronchial mucous glands and bronchial epithelial mucous cells. These changes may have indicated an infection, possibly of viral nature, as an aetiological factor in some of the patients. Consistent with such a cause is the common observation of an infectious febrile respiratory illness preceding the onset of symptoms due to pulmonary veno-occlusive disease.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01230858
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    Paper (German National Licenses)
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  • 7
    Electronic Resource
    Electronic Resource
    Pulmonary venous changes in chronic hypoxia (1976)
    Springer
    Virchows Archiv 372 (1976), S. 51-56 
    Details
    ISSN: 1432-2307
    Keywords: Pulmonary venous changes ; Chronic hypoxia ; High altitude ; Intimai smooth muscle cells ; Venoconstriction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Lung tissue from 14 normal residents of high altitude regions, 10 patients with chronic bronchitis and emphysema, and 1 patient with Pickwickian syndrome was studied with regard to the occurrence of pulmonary vascular changes. In addition to the well-known pulmonary arterial alterations, lesions in small pulmonary veins were found in the great majority of the cases. These changes, consisting of medial hypertrophy and arterialization and of bundles of smooth muscle cells within the venous intima, have not been described before in man. These findings suggest that alveolar hypoxia acts not only on small pulmonary arteries and arterioles but also on veins of small caliber, probably by inducing venoconstriction.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00429716
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    Paper (German National Licenses)
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  • 8
    Electronic Resource
    Electronic Resource
    Longitudinal smooth muscle in pulmonary arteries (1984)
    Springer
    Virchows Archiv 404 (1984), S. 265-274 
    Details
    ISSN: 1432-2307
    Keywords: Pulmonary arteries ; Intimal thickening ; Intimal smooth muscle ; Congenital heart disease
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In lung biopsy specimens of 19 patients with congenital heart disease and pulmonary hypertension, in addition to the common features of plexogenic arteriopathy, longitudinal smooth muscle cells were found in small pulmonary arteries. These cells were arranged in bundles or layers, particularly in the intima but sometimes within the media or adventitia of the arteries. They often caused severe narrowing of the lumen. Corrective surgery of the cardiac defect was performed in 14 patients. The results suggested that even when these changes are wide-spread and severe, they do not stand in the way of a favourable postoperative course. In one patient who underwent a banding procedure of the pulmonary artery, virtually complete regression of the smooth muscle layers could be demonstrated in a second biopsy, taken 5 years later during a corrective operation.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00694892
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    Paper (German National Licenses)
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  • 9
    Electronic Resource
    Electronic Resource
    Development and regression of pulmonary arterial lesions after experimental air embolism (1985)
    Springer
    Virchows Archiv 406 (1985), S. 203-212 
    Details
    ISSN: 1432-2307
    Keywords: Pulmonary artery ; Air embolism ; Intima ; Oedema ; Electron microscopy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Repeated systemic venous air embolism produces pulmonary vascular lesions, the nature of which is still a subject of controversy. We investigated the pulmonary arterial lesions produced by repeated air embolism in rabbits, both at light and electron microscopic level. We found that they form a remarkable histopathological entity, consisting of initial pronounced vasoconstriction, combined with severe intimal inflammatory changes. Within 4 days after the last injection of air, peculiar sheet-like structures consisting of oedematous tissue and lined by endothelium, projected into the lumen. These structures probably resulted from the shearing stress of the blood, streaming over the severely oedematous intima. They subsequently became thinner and disappeared after two weeks. Various types of blood-borne and mesenchymal cells were present in the thickened intima and within the sheets. The origin of the latter cells remained undecided. They may originate from medial smooth muscle cells penetrating the internal elastic lamina as well as by transition from blood-borne cells into mesenchymal cells, or both.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00737086
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    Paper (German National Licenses)
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  • 10
    Electronic Resource
    Electronic Resource
    Cor triatriatum masked by primary pulmonary hypertension (1985)
    Springer
    Pediatric cardiology 6 (1985), S. 161-164 
    Details
    ISSN: 1432-1971
    Keywords: Cor triatriatum ; Primary pulmonary hypertension ; Echocardiography
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In a 17-month-old boy with clinical signs of right heart failure, the diagnosis of primary pulmonary hypertension was made, based on cardiac catheterization findings with high pulmonary arterial pressure and normal pulmonary wedge pressure although two-dimensional echocardiography demonstrated a left atrial membrane. Postmortem examination confirmed a left atrial membrane (cor triatriatum), and the microscopic examination revealed the histological pattern of primary pulmonary hypertension. Due to reduced pulmonary blood flow, pulmonary venous obstruction was masked and could not be assessed by measuring pulmonary wedge pressure.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02336557
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    Paper (German National Licenses)
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