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  • Articles: DFG German National Licenses  (6)
  • 1985-1989  (6)
Source
  • Articles: DFG German National Licenses  (6)
Material
Years
Year
  • 1
    Electronic Resource
    Electronic Resource
    HISTOCOMPATIBILITY AND THE GENETICS OF TUMOUR RESISTANCE (1986)
    Oxford, UK : Blackwell Publishing Ltd
    International journal of immunogenetics 13 (1986), S. 0 
    Details
    ISSN: 1744-313X
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1744-313X.1986.tb01085.x
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    HILLIARD FESTENSTEIN (1989)
    Oxford, UK : Blackwell Publishing Ltd
    International journal of immunogenetics 16 (1989), S. 0 
    Details
    ISSN: 1744-313X
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1744-313X.1989.tb00470.x
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Qa-12—-A NOVEL DETERMINANT OF ACTIVATED T AND B LYMPHOCYTES (1989)
    Oxford, UK : Blackwell Publishing Ltd
    International journal of immunogenetics 16 (1989), S. 0 
    Details
    ISSN: 1744-313X
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: Investigation of the antigenic phenotype of activated lymphocytes using the broadly cross-reactive mAb 6.3.4 revealed two phenotypic alterations as compared with the resting lymphocytes. The Qa specificity Qa-m208 disappears after lectin activation of Qa-m208-positive T lymphocytes. Analysis of Q7 and Q9 transfectants expressing the Qa-2 polypeptides shows that Qa-m208 is an epitope of the Qa-2 antigen. Because the Qa-2 antigens are still expressed on T lymphoblasts which have lost Qa-m208, changes of the Qa-2 molecules occur and result in the loss of certain epitopes.The second phenotypic change that we observed is the appearance of a novel specificity, Qa-12. Its expression is induced by lymphocyte activation and it is expressed on lymphoblasts of both T and B cell origin. The presence of this novel non-ubiquitous antigenic specificity is determined by the Tla region.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1744-313X.1989.tb00472.x
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Recombinant congenic strains — A new tool for analyzing genetic traits determined by more than one gene (1986)
    Springer
    Immunogenetics 24 (1986), S. 416-422 
    Details
    ISSN: 1432-1211
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00377961
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Corticosteroid-induced cleft palate:Cis interaction of MHC genes and hybrid resistance (1985)
    Springer
    Immunogenetics 22 (1985), S. 183-188 
    Details
    ISSN: 1432-1211
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00563516
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    H-2 class I antigen expression on mouse teratocarcinoma cell lines (1985)
    Springer
    Immunogenetics 22 (1985), S. 543-552 
    Details
    ISSN: 1432-1211
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Immunity against PCC3 teratocarcinoma cells (129, H-2 b) was induced in allogeneic (C3H, H-2 k) mice by preimmunization with L cells (C3H, H-2 k) expressing cosmid-introduced K b or D b genes, but not with nontransfected L cells. In addition, the growth of PCC3 cells in sublethally irradiated (C3H × B6-H-2 bm1)F1 and (C3H × B6-H-2 bm13 )F1 mice bearing the K bm1 and D bm13 mutations, respectively, was either prevented, stopped, or delayed in comparison with the (C3H × B6)F1 (k × b) mice, which failed to reject the PCC3 cells. The teratocarcinoma line OC15S was exceptional because it reacted specifically with Kb- and Db-specific (but not Ib-specific) alloantisera, and because Kb- and Db-specific antibodies could be absorbed by OC15S cells. The subpopulation of OC15S cells bearing the ECMA-7 antigen characteristic for embryonic carcinoma (EC) cells was isolated by the fluorescence-activated cell sorter and was shown to react specifically with Kb- and Db-specific antisera. These experiments show that teratocarcinoma cells express antigens similar or identical to the K-and D-region products of differentiated cells. The lack of expression of class I antigens is thus neither a condition nor a consequence of the pluripotentiality of the EC cells. The exact nature of the major histocompatibility complex antigens on EC cells has yet to be established using the methods of molecular biology and biochemistry.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00430302
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    Paper (German National Licenses)
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