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  • 1
    Electronic Resource
    Electronic Resource
    High-performance resin-bonded magnets produced from zinc metal-coated Sm2(Fe0.9Co0.1)17Nx fine powders (1999)
    Woodbury, NY : American Institute of Physics (AIP)
    Applied Physics Letters 75 (1999), S. 1601-1603 
    Details
    ISSN: 1077-3118
    Source: AIP Digital Archive
    Topics: Physics
    Notes: Fine powders of Sm2(Fe0.9Co0.1)17Nx (x=∼3) with particle sizes (d) around 1 μm as coated with zinc metal produced via the photodecomposition of diethylzinc [Zn(C2H5)2], which still provided high remanence (Br) and coercivity (Hcj) values of ∼1.43 T and ∼0.85 MAm−1, were molded to compression-type resin bonded Zn/Sm2(Fe0.9Co0.1)17Nx magnets with density values of ∼6.33 g cm−3. By optimizing the preparation conditions such as grinding, surface coating, and molding for them, the highest maximum energy product of (BH)max=186 kJm−3 for Hcj=0.73 MAm−1 was recorded among all kinds of the Sm–Fe–N based magnets reported to date. Furthermore, the excellent aging behavior of the bonded Zn/Sm2(Fe0.9Co0.1)17Nx magnets was observed after standing in air at ∼393 K and the thermal coefficient for permanent magnet was evaluated to be α(Br)=−0.04% K−1. © 1999 American Institute of Physics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.124767
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  • 2
    Electronic Resource
    Electronic Resource
    Rubber from Guayule (1947)
    s.l. : American Chemical Society
    Industrial & engineering chemistry 39 (1947), S. 1477-1485 
    Details
    ISSN: 1520-5045
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology , Process Engineering, Biotechnology, Nutrition Technology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ie50455a018
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  • 3
    Electronic Resource
    Electronic Resource
    ROLE OF CEREBRAL ATP-SENSITIVE K+ CHANNELS IN ARTERIAL PRESSURE REGULATION DURING ACUTE CEREBRAL ISCHAEMIA IN SHR AND WKY RATS (1995)
    Oxford, UK : Blackwell Publishing Ltd
    Clinical and experimental pharmacology and physiology 22 (1995), S. 0 
    Details
    ISSN: 1440-1681
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: 1. ATP-sensitive K+ channels (KATP) are activated either by decreased intracellular ATP content or ATP/ADP ratio during ischaemia. We examined the role of a cerebral KATP in arterial pressure regulation during acute cerebral ischaemia using SHR and WKY rats. Thirteen week old male SHR or WKY rats were anaesthetized with urethane, and arterial pressure and heart rate were recorded under an artificial ventilation.2. Intracerebroventricular (i.c.v.) injections of glibenclamide, a specific inhibitor of KATP, elicited dose-dependent vasopressor responses in WKY with bilateral ligation of carotid arteries, whereas it caused smaller vasopressor responses in SHR than WKY.3. Systemic administration of AVP V1 receptor antagonist, OPC-21268, abolished the vasopressor responses of i.c.v. injections of glibenclamide in WKY but not in SHR.4. Intracerebroventricular injections of glibenclamide caused both the increase in plasma concentration of AVP and the decrease in pituitary AVP content in WKY with bilateral ligation of carotid arteries, whereas it elicited no significant change in plasma and pituitary concentration of AVP in SHR with bilateral ligation of carotid arteries.5. Cerebral KATP may play a role in the protection of excess hypertension by inhibiting AVP release from the pituitary glands during acute ischaemia in WKY, but this mechanism might not work in SHR during acute cerebral ischaemia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1440-1681.1995.tb02975.x
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  • 4
    Electronic Resource
    Electronic Resource
    Subcellular and developmental changes in distribution of aconitase isoforms in pumpkin cotyledons (1995)
    Springer
    Planta 195 (1995), S. 464-468 
    Details
    ISSN: 1432-2048
    Keywords: Aconitase ; Cucurbita ; Glyoxylate cycle ; Glyoxysome ; Mitochondrion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract The subcellular localization and developmental changes of aconitase (EC 4.2.1.3) isoforms were studied in pumpkin tissues by anion-exchange chromatography. In etiolated pumpkin cotyledons three aconitase isoforms (denoted Aco I, II, and III, respectively) were found, and the first to be eluted (Aco I) represented the major part of the total aconitase activity in the tissue. Aconitase I gradually disappeared during the greening of cotyledons. In hypocotyls, roots and mature leaves Aco I was barely detectable, though the two other isoforms were present. In pumpkin mitochondria purified by differential or density gradient centrifugation, a high amount of Aco II was present, whereas the other isoforms were detected in low quantities. Since aconitase has never been clearly detected in glyoxysomal fractions, we conclude that the first and the third aconitase isoforms are cytosolic, while the second one represents the mitochondrial aconitase. Moreover, the first aconitase isoform is present in appreciable amounts only in etiolated cotyledons, so that it should represent the aconitase isoform involved in the glyoxylate cycle.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00202606
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  • 5
    Electronic Resource
    Electronic Resource
    Increase in natural killer cell activity following living-related liver transplantation (1998)
    Springer
    Transplant international 11 (1998), S. S185 
    Details
    ISSN: 1432-2277
    Keywords: Key words NK activity ; Living-related liver transplantation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract We monitored the serial changes of natural killer cell (NK) activity in eight recipients of living-related liver transplantation. The HLA types of all eight patients were haplotypically identical with those of their donors. Tacrolimus and methylprednisolone were used for immunosuppression. The NK activity before transplantation was 24.1 ± 20.2 % which is surprisingly low when compared with the value for normal individuals (67.7 ± 13.2 %, P 〈 0.01) or a liver dysfunction group (49.4 ± 21.9 %, P 〈 0.05). Serial changes in NK activity revealed a minimum of 6.1 ± 3.6 % 1 week after transplantation, gradually increasing to 49.2 ± 12.5 % at 2 months after transplantation. These results suggest that the diseased liver might play an important role in the suppression of NK activity.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001470050457
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  • 6
    Electronic Resource
    Electronic Resource
    Presenilin mutations associated with Alzheimer disease cause defective intracellular trafficking of β-catenin ,a component of the presenilin protein complex (1999)
    [s.l.] : Nature America Inc.
    Nature medicine 5 (1999), S. 164-169 
    Details
    ISSN: 1546-170X
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Medicine
    Notes: [Auszug] The presenilin proteins are components of high–molecular–weight protein complexes in the endoplasmic reticulum and Golgi apparatus that also contain β-catenin. We report here that presenilin mutations associated with familial Alzheimer disease (but not the non–pathogenic ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/5526
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  • 7
    Electronic Resource
    Electronic Resource
    Mercury in the Kuroshio and Oyashio regions and the Japan Sea (1975)
    [s.l.] : Nature Publishing Group
    Nature 258 (1975), S. 224-225 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] One source of error is to be found in the use of polyethylene bottles-we found that nanogram amounts of mercury were leached out of the plastic by the samples, whose mercury concentrations increased by factors of 2 to 100, even if the bottles had been washed out with nitric or hydrochloric acid ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/258224a0
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  • 8
    Electronic Resource
    Electronic Resource
    Brain hormone contained in the silkworm body ofBombyx mori (1975)
    Springer
    Cellular and molecular life sciences 31 (1975), S. 1105-1107 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The present report is concerned with the comparison of the brain hormone titers contained in the head and thorax-abdomen of both sexes of 3 races of silkworms,Bombyx mori during and post imaginal differentiation and also with the effects of debraining and castration on the brain hormone titer in the body.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02326985
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  • 9
    Electronic Resource
    Electronic Resource
    Hyperresponse in calcium-induced insulin release from electrically permeabilized pancreatic islets of diabetic GK rats and its defective augmentation by glucose (1995)
    Springer
    Diabetologia 38 (1995), S. 772-778 
    Details
    ISSN: 1432-0428
    Keywords: Key words Insulin release ; intracellular calcium ; exocytosis ; GK rat ; permeabilized islets.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In spontaneously diabetic GK rats, insulin secretion from pancreatic beta cells in response to glucose is selectively impaired, probably due to deficient intracellular metabolism of glucose and impaired closure of KATP channels during glucose stimulation. By using electrically permeabilized islets of GK rats, we explored the functional modulations in exocytotic steps distal to the rise in [Ca2 + ]i in the diabetic condition. At 30 nmol/l Ca2 + (basal conditions) insulin release was similar between GK and non-diabetic control Wistar rats. In response to 3.0 μmol/l Ca2 + (maximum stimulatory conditions), insulin release was significantly augmented in permeabilized GK islets (p 〈 0.01). Raising glucose concentrations from 2.8 to 16.7 mmol/l further augmented insulin release induced by 3.0 μmol/l Ca2 + from permeabilized control islets(p 〈 0.001), but had no effect on that from permeabilized GK islets. The stimulatory effect of glucose on insulin release from permeabilized control islets was partly inhibited by 2,4-dinitrophenol, an inhibitor of mitochondrial oxidative phosphorylation (p 〈 0.01). The hyperresponse to Ca2 + in GK islets may play a physiologically compensatory role on the putative functional impairment both in [Ca2 + ]i rise and energy state in response to glucose in diabetic β cells, and may explain the relative preservation of insulin release induced by non-glucose depolarizing stimuli, such as arginine, from pancreatic islets in non-insulin-dependent diabetes mellitus. [Diabetologia (1995) 38: 772–778]
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001250050351
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  • 10
    Electronic Resource
    Electronic Resource
    Genetic contribution of the BAT2 gene microsatellite polymorphism to the age-at-onset of insulin-dependent diabetes mellitus (1999)
    Springer
    Human genetics 〈Berlin〉 105 (1999), S. 197-199 
    Details
    ISSN: 1432-1203
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract. The BAT2 gene lies within the class III region of the major histocompatibility complex. We investigated the frequency of the BAT2 microsatellite alleles (BAT2) in 74 young-onset insulin-dependent diabetes mellitus (IDDM) patients, 51 adult-onset IDDM patients, and 85 normal control subjects, and assessed the associations among these BAT2 alleles, TNFa microsatellite alleles (TNFa), and HLA-DRB1 alleles. The frequency of the BAT2.9 allele was significantly increased in the young-onset IDDM patients (12.8 vs 4.1%, Pc=0.04896), whereas the frequency of BAT2.12 allele was significantly decreased in young-onset IDDM patients (0.0 vs 11.8%, Pc=0.00002) compared with control subjects. The BAT2.9 allele was strongly associated with TNFa9 in the young-onset IDDM patients, although no association was found between the BAT2.9 and HLA-DRB1 alleles. The BAT2.12 allele was strongly associated with TNFa13, and with DRB1*1502 in control subjects. These results suggest that the BAT2 microsatellite polymorphism is associated with the age-at-onset of IDDM and possibly with the inflammatory process of pancreatic β-cell destruction during the development of IDDM. However, this association is not independent of TNFa polymorphisms.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004399900100
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