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  • 1990-1994  (6)
  • 1985-1989  (7)
  • 1
    Electronic Resource
    Electronic Resource
    Early steps of the magnesium-ATPase of relaxed myofibrils. A comparison with calcium-activated myofibrils and myosin subfragment 1 (1992)
    s.l. : American Chemical Society
    Biochemistry 31 (1992), S. 8036-8042 
    Details
    ISSN: 1520-4995
    Source: ACS Legacy Archives
    Topics: Biology , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/bi00149a038
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Effect of 2,3-butanedione monoxime on myosin and myofibrillar ATPases. An example of an uncompetitive inhibitor (1992)
    s.l. : American Chemical Society
    Biochemistry 31 (1992), S. 12227-12232 
    Details
    ISSN: 1520-4995
    Source: ACS Legacy Archives
    Topics: Biology , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/bi00163a036
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    Evolutionary relationship of uptake systems for biopolymers in Escherichia coli: cross-complementation between the TonB-ExbB-ExbD and the TolA-TolQ-TolR proteins (1993)
    Oxford, UK : Blackwell Publishing Ltd
    Molecular microbiology 8 (1993), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: Escherichia coli possesses two energy-coupled import systems through which substances of low concentration and of a size too large to permit diffusion through the porins are translocated across the outer membrane. Group B colicins, ferric siderophores and vitamin B12 are taken up via the TonB-ExbB-ExbD, group A colicins via the TolA-TolQ-TolR system. Cross-complementation between the two systems was demonstrated in that tolQ tolR mutants transformed with plasmids carrying exbB exbD became sensitive to group A colicins, and exbB exbD mutants transformed with plasmid-encoded tolQ tolR became sensitive to group B colicins. TolQ-TolR interacted through TonB, and ExbB-ExbD interacted through TolA with the outer membrane receptors and colicins. Activity of ExbB ExbD via TolA was higher in cells laciting TonB, and activity of TolQ TolR via TonB was increased when TolA was missing. The very distinct TolA and TonB proteins mediate exclusive interaction with group A and group B receptors, respectively. ExbB-TolR and ExbD-TolQ mixtures showed little if any complementation of exbB exbD and tolQ tolR mutants indicating coevolution of ExbB with ExbD and TolQ with ToIR. Sequence homology and mutual functional substitution of ExbB-ExbD and TolQ-TolR suggest the evolution of the two import systems from a single import system.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2958.1993.tb01570.x
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  • 4
    Electronic Resource
    Electronic Resource
    Immune thrombocytopenia associated with hemorrhagic diathesis due to ibuprofen administration (1993)
    Springer
    Journal of molecular medicine 71 (1993), S. 413-415 
    Details
    ISSN: 1432-1440
    Keywords: Ibuprofen thrombocytopenia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Acute thrombocytopenic purpura temporally related to the oral administration of ibuprofen developed in a patient with ankylosing spondylitis. Clinical manifestations, with sudden onset occurring within t2 h of drug ingestion and rapid increase of platelet counts following discontinuation of the drug, were characteristic of an antibody-mediated immune pathomechanism. Immunological studies demonstrated IgM and IgG antibodies in the patient's serum that were capable of binding to allogenic platelets in the presence of a metabolite preparation. This finding suggested that an ibuprofen metabolite, rather then the drug itself, was the antigenic agent responsible for the immune reaction. Despite its widespread therapeutic use, ibuprofen has not been described previously as causing immune-mediated thrombocytopenia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00186632
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  • 5
    Electronic Resource
    Electronic Resource
    Myocardial infarction in young patients with Hodgkin's disease — potential pothogenic role of radiotherapy, chemotherapy, and splenectomy (1993)
    Springer
    Journal of molecular medicine 71 (1993), S. 57-64 
    Details
    ISSN: 1432-1440
    Keywords: Myocardial infarction ; Hodgkin's disease ; Radiation therapy ; Chemotherapy ; Splenectomy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Among a total of 2147 patients admitted to our hospital for acute myocardial infarction between 1978 and 1987, three young patients aged 24, 29, and 39 years had previously been treated for Hodgkin's disease. Staging laparotomy, including splenectomy, had been performed in all three patients. Two patients had both mediastinal irradiation (21 and 27 months before infarction) and chemotherapy. In the first patient, postmortem histologic examination of the coronary arteries revealed fibrotic changes, which were probably induced by radiotherapy. In our second patient, myocardial infarction developed 5 days after vinblastine treatment; early angiography showed thrombotic occlusion of the proximal right coronary artery, which was recanalized using the diagnostic Sones catheter. Subsequent angiography revealed normal coronary arteries. This is, to our knowledge, the first case of documented coronary artery thrombosis after treatment with vinca-alkaloids. In our third patient, neither mediastinal irradiation nor chemotherapy had been performed prior to myocardial infarction. However, a marked increase in platelet counts following splenectomy was observed in this patient. The role of radiotherapy, chemotherapy, and splenectomy with consecutive thrombocytosis as a third possible pathogenic factor for subsequent development of myocardial infarction is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00210966
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    Release of tryptophan and serotonin into the portal vein of the isolated perfused rat small intestine (1989)
    Springer
    Research in experimental medicine 189 (1989), S. 281-287 
    Details
    ISSN: 1433-8580
    Keywords: Serotonin ; Tryptophan ; Small intestine ; Rat ; Pargyline
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary To investigate the release of serotonin from intestinal enterochromaffin cells, we used an in vitro technique which allows studies excluding overlapping influences from outside the gut. The entire small intestine of rats fed a standard or tryptophan-enriched (3% of total) diet was totally isolated by ligatures with the exception of the superior mesentric artery and portal vein that supply and drain the intestine. Simultaneously to the vascular perfusion (Krebs-Ringer bicarbonate buffer, 0,4% human albumin, 5 mM glucose, 0.6 mM glutamine) the gut lumen was infused (buffer or 0.1 N HCL). Acidification of the gut lumen resulted in an increment of venously released tryptophan and serotonin. After feeding tryptophan-enriched food the release of tryptophan was increased. However, the total amount of released serotonin after tryptophan diet did not differ as compared to that after standard diet. Addition of a monoamino-oxidase inhibitor (pargyline) to the arterial perfusate enhanced the released amount of serotonin 3-fold in the portal venous effluent (at a concentration of 1 mM but not 0.1 mM). Recovery studies done by arterial infusions of serotonin (1 µM, 10µM) and evaluation of the amounts venously released revealed a high loss of infused serotonin (40%–70%). Our data suggest gut-born serotonin to more likely play a paracrine role than a role as a classical hormone.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01852260
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  • 7
    Electronic Resource
    Electronic Resource
    The magnetic hyperfine field of lutetium in iron by NMR/ON (1985)
    Springer
    Hyperfine interactions 22 (1985), S. 167-171 
    Details
    ISSN: 1572-9540
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract Nuclear magnetic resonance of oriented177Lu in iron has been found with a sample prepared by on-line implantation of177Lu in iron at T〈0.2 K. The broad resonance, FWHM=20.5 (1.3) MHz, has a centre frequency of νL=355.06 (51) MHz at zero external field. With the g-factor of177Lu g=0.637 (3) from literature the magnetic hyperfine field of Lu in Fe is derived as Bhf=−73.12(36) T. Static nuclear orientation data are not compatible with a two site model where the nuclei which are oriented experience the hyperfine interaction found in NMR/ON. A fraction with a lower hyperfine field is necessary to explain the data.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02063988
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    Paper (German National Licenses)
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  • 8
    Electronic Resource
    Electronic Resource
    NMR/ON of206Bi implanted at T 〈 0.2 K and at room temperature into nickel (1985)
    Springer
    Hyperfine interactions 22 (1985), S. 173-176 
    Details
    ISSN: 1572-9540
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract NMR/ON has been observed for206BiNi with samples prepared by implantation at T〈0.2 K and at room temperature. The integrated destruction of anisotropy for the sample implanted at 290 K is only about half of that for the on-line implanted sample but the resonance line is less broad and has a narrow component with centre frequency νL= 223.62(10) MHz for zero external field. With the known g-factor of206Bi we derive from this frequency the hyperfine field of BiNi as Bhf=38.3(4) T.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02063989
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  • 9
    Electronic Resource
    Electronic Resource
    The magnetic hyperfine field of bromine in iron by NMR/ON (1986)
    Springer
    The European physical journal 64 (1986), S. 353-356 
    Details
    ISSN: 1434-6036
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract Nuclear magnetic resonance of82Br oriented at low temperature in iron has been observed with a sample prepared by ion implantation atT〈0.2 K. The asymmetric resonance signal can be decomposed in a broad background signal and a narrow line of FWHM=1.8 (4) MHz which can be attributed to82Br in undisturbed substitutional sites of Fe. From the center frequency of this narrow line ν (B ext=0)=201.86(13) MHz we derive the magnetic hyperfine field asB hf(BrFe)=81.38(6) T. This value is considerably larger than the result of theoretical calculations.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01303605
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  • 10
    Electronic Resource
    Electronic Resource
    Nuclear magnetic resonance of175Hf oriented in iron (1986)
    Springer
    The European physical journal 63 (1986), S. 241-246 
    Details
    ISSN: 1434-6036
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract Nuclear magnetic resonance of175Hf oriented at low temperature in iron has been observed with a sample prepared by ion implantation. The centre frequency of the broad resonance line isv L (B ext = 0)=138.53(36)MHz. Possible origins of the large inhomogeneous line width of FWHM=11.0(1.1) MHz are discussed. A comparison with model calculations for combined magnetic and electric hyperfine interaction indicates that the centre frequency may be interpreted as the magnetic interaction frequency for175Hf in unperturbed substitutional sites of the host iron. With theg-factor of175Hf from literature the magnetic hyperfine field of Hf in Fe is derived asB hf=−64.9(9.3) T fitting well into systematics.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01309244
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