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  • 1
    Electronic Resource
    Electronic Resource
    Microstructures of 18R martensite induced by deformation and thermomechanical cycles in CuZnAl shape memory alloy (1991)
    Springer
    Journal of materials science 26 (1991), S. 5527-5533 
    Details
    ISSN: 1573-4803
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract Microstructures of martensite in a Cu-26.4Zn-4.8Al shape memory alloy after deformation and thermomechanical cycles are reported. By detailed transmission electron microscopy investigation, it is clearly established that the microstructure induced by deformation is different from that induced by thermomechanical cycles, and the effects on the thermoelastic martensite transformation are also not the same at all. The microstructure of martensite induced by deformation is mainly deformation twins when the deformation exceeds the range of recoverable strain of the shape memory alloy. The main microstructure characteristic of martensite which has undergone thermomechanical cycles is regularly distributed dislocations, which are mainly aligned but sometimes tangled. Micro-twins similar to those found in deformed martensite and crossed bands also exist inside the martensite of samples after thermomechanical cycles, but the amounts are too small to be regarded as important microstructure features.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02403954
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Microstructures of 18R martensite induced by deformation and thermomechanical cycles in CuZnAl shape memory alloy (1991)
    Springer
    Journal of materials science 26 (1991), S. 5527-5533 
    Details
    ISSN: 1573-4803
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract Microstructures of martensite in a Cu-26.4Zn-4.8Al shape memory alloy after deformation and thermomechanical cycles are reported. By detailed transmission electron microscopy investigation, it is clearly established that the microstructure induced by deformation is different from that induced by thermomechanical cycles, and the effects on the thermoelastic martensite transformation are also not the same at all. The microstructure of martensite induced by deformation is mainly deformation twins when the deformation exceeds the range of recoverable strain of the shape memory alloy. The main microstructure characteristic of martensite which has undergone thermomechanical cycles is regularly distributed dislocations, which are mainly aligned but sometimes tangled. Micro-twins similar to those found in deformed martensite and crossed bands also exist inside the martensite of samples after thermomechanical cycles, but the amounts are too small to be regarded as important microstructure features.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00553655
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Influence of heat treatment on the transformation hysteresis of CuAlNiMnTi shape-memory alloys (1993)
    Springer
    Journal of materials science 28 (1993), S. 6433-6436 
    Details
    ISSN: 1573-4803
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract The electrical resistance method, metallograph analysis, TEM observation and X-ray diffraction analysis have been employed to investigate the influence of heat treatment on the transformation hysteresis (Af-Ms) of CuAlNiMnTi shape-memory alloys (SMAs). It was found that when the quenching specimens were subjected to ageing treatment, the transformation temperature was decreased and the transformation hysteresis was initially increased, but then tended towards stability; when the quenched specimens were subjected to moderate-temperature treatment at 600
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01352208
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    Paper (German National Licenses)
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  • 4
    Electronic Resource
    Electronic Resource
    Effects of dietary restriction on insulin resistance in obese mice (2000)
    Springer
    Age 23 (2000), S. 95-101 
    Details
    ISSN: 1574-4647
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract In many cases, development of insulin resistance has been linked to obesity and may contribute to mechanism of aging. The role of diet, irrespective of degree of obesity, in modulating insulin resistance and development of age degeneration disease remains uncertain. Lowered blood glucose levels are commonly associated with diet restriction (DR), which is an intervention shown to successfully retard aging and age associated disease. The effects of DR on blood glucose and insulin resistance were measured in yellow obese (Avy/A), lean black (a/a) mice and in another common inbred strain (B6C3F1) (at three different ages). The yellow obese mice become diabetic as a result of an insulin receptor defect which is not clearly understood. Insulin responses and radioinsulin binding were assayed in yellow obese and lean black mice fed either ad libitum (AL) or DR diets (YAL, BAL, YDR and YAL, respectively) at four different circadian intervals. The B6C3F1 controls were fed either AL (CAL) or DR (CDR) and measures were made at six circadian stages and three different ages. Within 23 days, DR produced a significant loss in body weight and a time-dependent 22–55% reduction in basal blood glucose levels in the yellow obese mice. Additionally, exogenous insulin produced circadian stage dependent (at the time of food intake) reductions in blood glucose in the YDR animals that were not present in YAL animals. 125I-Insulin binding in liver was increased nearly 2-fold in YDR and BDR mice during the time of day that animals were active and eating. 125I-Insulin binding was two-fold-higher in CDR mice at 4, 12 and 〉24 months of age. Binding decreased as a function of age in both the CAL and CDR animals. However, even in the 〉24 month group the CDR animals were found to have levels of binding that were as high as those found in younger CAL liver. The mechanism of action appears to be through resolution of insulin resistance by modulating an insulin receptor defect.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s11357-000-0010-0
    Library Location Call Number Volume/Issue/Year Availability
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    Paper (German National Licenses)
    Fulltext
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