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  • 1
    Electronic Resource
    Electronic Resource
    Springer
    Surgical and radiologic anatomy 4 (1982), S. 249-250 
    ISSN: 1279-8517
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Springer
    Cellular and molecular life sciences 41 (1985), S. 415-417 
    ISSN: 1420-9071
    Keywords: Facial nerve ; hemifacial spasm ; synkinesis ; intraoperative recordings
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary We show evidence that the motonucleus of the facial nerve is involved in producing the synkinesis in patients with hemifacial spasm. These results were obtained by recording from the intracranial portion of the facial nerve and from the orbicularis oculi muscle in patients operated upon for hemifacial spasm during electrical stimulation of the mandibular branch of the facial nerve. Also, the electromyographic response from the same muscle was recorded when the facial nerve was electrically stimulated at a location near the brainstem. The results show that it is unlikely that the symptoms of patients with hemifacial spasm can be explained on the basis of ephaptic transmission at the site of lesion of the facial nerve.
    Type of Medium: Electronic Resource
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  • 3
    Electronic Resource
    Electronic Resource
    Springer
    Experimental brain research 68 (1987), S. 411-416 
    ISSN: 1432-1106
    Keywords: Trigeminal organization ; Cornea ; HRP
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Experiments were carried out in cats to learn the location of sensory axons from the cornea in the trigeminal nerve root just prior to its entry into the brainstem. HRP injected into the cornea labelled these axons and indicated they were not restricted to the ophthalmic division of the nerve root as had been indicated from previous studies. These findings, if representative of other branches of this nerve, offer a partial explanation for the variable preservation of function following transection of an entire division of the trigeminal nerve root in cases of trigeminal neuralgia.
    Type of Medium: Electronic Resource
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  • 4
    ISSN: 0942-0940
    Keywords: Disabling positional vertigo ; microvascular decompression
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Two-hundred seven patients who were operated on consecutively between January 1983 and December 1990 to relieve disabling positional vertigo (DPV) using the microvascular decompression (MVD) procedure were studied. Selection of the patients for MVD operations was based on both case history and the results of otoneurological tests. Of the 177 patients with unilateral symptoms, 8 were excluded because of previous vestibular nerve section, and 6 did not return for follow-up; of the remaining 163 patients, 129 (79%) were free of symptoms or markedly improved following MVD, and none became worse. Thirty patients had symptoms and signs of bilateral DPV, and of these 1 was excluded because of previous vestibular nerve section and 3 because of multiple operations. Of the remaining 26 patients, 20 (77%) were free of symptoms or markedly improved following MVD. Eleven of these patients had more than 2 operations. The follow-up time was an average of 38 months, ranging from 3 months to 10 years. The cure rate (about 80%) of MVD for DPV is similar to that reported for MVD for trigeminal neuralgia and hemifacial spasm. The cure rate of MVD for DPV was not related to gender or to the duration of the symptoms. Following a total of 254 operations that these 207 patients underwent, 4 patients (1.6%) lost hearing and 4 (1.6%) suffered marked hearing loss. Three patients suffered temporary deficits of other cranial nerves. There were no other complications to these operations.
    Type of Medium: Electronic Resource
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  • 5
    ISSN: 0942-0940
    Keywords: Acoustic neuroma ; cochlear nerve ; hearing preservation ; nerve injury
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Pathophysiological mechanisms responsible for intraoperative and postoperative hearing deficits associated with cerebellopontine (CP) angle operations were explored experimentally in dogs. The CP angle operative manipulations performed were the same as those experienced by human patients, and auditory evoked potentials were monitored intraoperatively. As a result of the operative manipulations, petechial or confluent hemorrhages occurred at the compressed portions of the cochlear nerve, and intravascular clots were often observed. Disintegration of the nerve fibers was verified by ultrastructural examination. Moreover, rupture of the microvasculature within the cochlear nerve occurred at locations remote from the operative site, due to stretching of the nerve trunk. The Obersteiner-Redlich zone, the Schwann-glial junction of the cochlear nerve, was a locus minoris resistentiae in CP angle surgery; the vasa nervorum easily bled at this zone and the peripheral and central myelins easily separated at their junctional zones (“central” avulsion injury). Intracochlear hemorrhages were identified as the most probable cause of the sudden loss of all components of the auditory evoked potentials, a frequent predictor of postoperative hearing loss, although rupture, occlusion, or vasospasm of the main trunk of the internal auditory artery have also been implicated as possible causes of such hearing losses. The results of this study show that hearing preservation is highly dependent on preserving not only the nerve at the operative site but also the remote O-R zone and intracochlear structures.
    Type of Medium: Electronic Resource
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  • 6
    Electronic Resource
    Electronic Resource
    Springer
    Acta neurochirurgica 134 (1995), S. 21-26 
    ISSN: 0942-0940
    Keywords: Spasmodic torticollis ; microvascular decompression ; spinal accessory nerve ; movement disorders
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Twenty patients with spasmodic torticollis (ST) were treated by microvascular decompression (MVD) of the spinal accessory nerves, the upper cervical nerve roots and the brainstem. Thirteen were female and seven male. Median age was 47 years (range 39 to 70 years). Median duration of symptoms was 5 years (range 4 months to 17 years). Ten had right horizontal; nine, left horizontal; and one, retrocollis ST. Twenty-two operations were performed on twenty patients, suboccipital craniectomy and C1 laminectomy in 18 and retromastoid craniectomy in 4 operations. The most common compressing blood vessels were the vertebral artery and/or the posterior inferior cerebellar artery. No nerve section was performed. Three patients died of unrelated conditions, 3, 5 1/2, and 6 years postoperatively, respectively. Minimum follow-up period in the rest of the cases is 5 years (range 5 to 10 years). Thirteen (65%) were cured, four (20%) improved with minimal spasm, one (5%) improved with moderate spasm, and two (10%) improved minimally or unchanged. In most cases the cure or improvement was noticed gradually over 6 months to two years following the operation. There was no operative mortality. Postoperative morbidities included transient cerebrospinal fluid leakage through the surgical incision in one case and an apparent multiple small vessel stroke involving periventricular white matter in one reoperation case with full recovery. MVD for ST is a nondestructive benign procedure with high probability of cure or significant improvement.
    Type of Medium: Electronic Resource
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  • 7
    ISSN: 0942-0940
    Keywords: Spasmodic torticollis ; spinal accessory nerve ; microvascular decompression ; hemifacial spasm ; Sternocleidomastoid muscle
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Electromyographic (EMG) recordings from patients undergoing microvascular decompression (MVD) operations to relieve spasmodic torticollis were studied. When EMG potentials were recorded from the sternocleidomastoid muscle in response to electrical stimulation of the spinal accessory nerve (SAN) at the neck, an abnormal (delayed) response was seen in 9 of 12 patients who had unilateral symptoms. In 5 patients with bilateral symptoms, no such delayed response was seen. We assume that this abnormal muscle response depends on an abnormal cross-transmission. Neural conduction time measurements, using electrical stimulation of the intracranial portion of the SAN, indicated that the location of this cross-transmission was more central than the vascular compression of the SAN. We hypothesize that this location might be in the motonucleus of the SAN. Similarities between these abnormal EMG findings in patients with spasmodic torticollis and those reported earlier in patients with hemifacial spasm (HFS) are presented.
    Type of Medium: Electronic Resource
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