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  • 1
    Electronic Resource
    Electronic Resource
    Oxford, UK : Munksgaard International Publishers
    Scandinavian journal of medicine & science in sports 12 (2002), S. 0 
    ISSN: 1600-0838
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine , Sports Science
    Notes: Context: Exercise is widely perceived to be beneficial for glycemic control and weight loss in patients with type 2 diabetes. However, clinical trials on the effects of exercise in patients with type 2 diabetes have had small sample sizes and conflicting results. Objective: To systematically review and quantify the effect of exercise on glycosylated hemoglobin (HbA(1c)) and body mass in patients with type 2 diabetes. Data sources: Database searches of MEDLINE, EMBASE, Sport Discuss, Health Star, Dissertation Abstracts and the Cochrane Controlled Trials Register for the period up to and including December 2000. Additional data sources included bibliographies of textbooks and articles identified by the database searches. Study selection: We selected studies that evaluated the effects of exercise interventions (duration ≥ = 8 weeks) in adults with type 2 diabetes. Fourteen (11 randomized and three non-randomized) controlled trials were included. Studies that included drug cointerventions were excluded. Data extraction: Two reviewers independently extracted baseline and post-intervention means and SDs for the intervention and control groups. The characteristics of the exercise interventions and the methodological quality of the trials were also extracted. Data synthesis: Twelve aerobic training studies [mean (SD), 3.4 (0.9) times/week for 18 (15) weeks] and two resistance training studies [mean (SD), 10 (0.7) exercises, 2.5 (0.7) sets, 13 (0.7) repetitions, 2.5 (0.4) times/week for 15 (10) weeks] were included in the analyses. The weighted mean post-intervention HbA(1c) was lower in the exercise groups compared with the control groups (7.65% vs 8.31%; weighted mean difference, 0.66%; P 〈 0.001). The difference in post-intervention body mass between exercise groups and control groups was not significant (83.02 kg vs 82.48 kg; weighted mean difference, 0.54; P= 0.76). Conclusion: Exercise training reduces HbA(1c) by an amount that should decrease the risk of diabetic complications, but no significantly greater change in body mass was found when exercise groups were compared with control groups.
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Springer
    European journal of applied physiology 76 (1997), S. 116-121 
    ISSN: 1439-6327
    Keywords: Key words Warm-water immersion ; Esophageal temperature ; Cutaneous vasodilation ; Thermoregulation ; Heat loss
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract We demonstrated previously that esophageal temperature (T es) remains elevated by ≈0.5°C for at least 65 min after intense exercise. Following exercise, average skin temperature (T avg) and skin blood flow returned rapidly to pre-exercise values even though T es remained elevated, indicating that the T es threshold for vasodilation is elevated during this period. The present study evaluates the hypothesis that the threshold for sweating is also increased following intense exercise. Four males and three females were immersed in water (water temperature, T w = 42°C) until onset of sweating (Immersion 1), followed by recovery in air (air temperature, T a = 24°C). At a T a of 24°C, 15 min of cycle ergometry (70% VO2max) (Exercise) was then followed by 30 min of recovery. Subjects were then immersed again (T w = 42°C) until onset of sweating (Immersion 2). Baseline T es and T skavg were 37.0 (0.1)°C and 32.3 (0.3)°C, respectively. Because the T skavg at the onset of sweating was different during Exercise [30.9 (0.3)°C] than during Immersion 1 and Immersion 2 [36.8 (0.2)°C and 36.4 (0.2)°C, respectively] a corrected core temperature, T es (calculated), was calculated at a single designated skin temperature, T sk(designated), as follows: T es(calculated) = T es + [β/(1−β)][T skavg−T sk(designated)]. The T sk(designated) was set at 36.5°C (mean of Immersion 1 and Immersion 2 conditions) and β represents the fractional contribution of T skavg to the sweating response (β for sweating = 0.1). While T es(calculated) at the onset of sweating was significantly lower during exercise [36.7 (0.2)°C] than during Immersion 1 [37.1 (0.1)°C], the threshold of sweating during Immersion 2 [37.3 (0.1)°C] was greater than during both Exercise and Immersion 1 (P 〈 0.05). We conclude that intense exercise decreases the sweating threshold during exercise itself, but elicits a subsequent short-term increase in the resting sweating threshold.
    Type of Medium: Electronic Resource
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  • 3
    Electronic Resource
    Electronic Resource
    Springer
    European journal of applied physiology 79 (1999), S. 495-499 
    ISSN: 1439-6327
    Keywords: Key words Esophageal temperature ; Thermoregulation ; Heat loss
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The purpose of this study was to evaluate the effect of exercise on the subsequent post-exercise thresholds for vasoconstriction and shivering measured during water immersion. On 2 separate days, seven subjects (six males and one female) were immersed in water (37.5°C) that was subsequently cooled at a constant rate of ≈6.5°C · h−1 until the thresholds for vasoconstriction and shivering were clearly established. Water temperature was then increased to 37.5°C. Subjects remained immersed for ≈20 min, after which they exited the water, were towel-dried and sat in room air (22°C) until both esophageal temperature and mean skin temperature (T¯ sk) returned to near-baseline values. Subjects then either performed 15 min of cycle ergometry (at 65% maximal oxygen consumption) followed by 30 min of recovery (Exercise), or remained seated with no exercise for 45 min (Control). Subjects were then cooled again. The core temperature thresholds for both vasoconstriction and shivering increased significantly by 0.2°C Post-Exercise (P 〈 0.05). Because the T¯ sk at the onset of vasoconstriction and shivering was different during Pre- and Post-Exercise Cooling, we compensated mathematically for changes in skin temperatures using the established linear cutaneous contribution of skin to the control of vasoconstriction and shivering (20%). The calculated core temperature threshold (at a designated skin temperature of 32.0°C) for vasoconstriction increased significantly from 37.1 (0.3)°C to 37.5 ( 0.3)°C post-exercise (P 〈 0.05). Likewise, the shivering threshold increased from 36.2 (0.3)°C to 36.5 (0.3)°C post-exercise (P 〈 0.05). In contrast to the post-exercise increase in cold thermal response thresholds, sequential measurements demonstrated a time-dependent similarity in the Pre- and Post-Control thresholds for vasoconstriction and shivering. These data indicate that exercise has a prolonged effect on the post-exercise thresholds for both cold thermoregulatory responses.
    Type of Medium: Electronic Resource
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