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  • 1
    Unknown
    Albany : State University of New York Press
    SUNY series, studying the self  
    Keywords: Estime de soi chez l'adolescent. ; Identité chez l'adolescent. ; Identity (Psychology) in adolescence. ; Self-esteem in adolescence.
    Notes: Understanding early adolescent self and identity: an introduction / Thomas M. Brinthaupt & Richard P. Lipka -- Part I: Conceptual issues -- Self and identity in early adolescence: the pains and gains of knowing who and what you are / Catrin Finkenauer, Rutger C.M.E. Engels, Wim Meeus, & Annerieke Oosterwegel -- Self-esteem as a multifaceted construct / Michael H. Kernis -- Part II: School and the sense of self -- On academic identity formation in middle school settings during early adolescence: a motivational-contextual perspective / Robert W. Roeser & Shun Lau -- The ecology of middle grades school and possible selves: theory, research, and action / Peggy Clements & Edward Seidman -- Part III: Peer relationships and behavioral problems -- Self, self-esteem, conflicts, and best friendships in early adolescence / Margarita Azmitia -- Stability and change in global self-esteem and self-related affect / Françoise Alsaker & Dan Olweus -- Influence of competence and alcohol use on self-esteem: latent growth curve models using longitudinal data / Lawrence M. Scheier & Gilbert J. Botvin -- Part IV: Early adolescent interventions -- Identity in early adolescence via social change activities: experience of the adolescent social action program / Lily Dow Velarde, Randall G. Starling, & Nina B. Wallerstein -- A place to call home: youth organizations in the lives of inner city adolescents / Nancy L. Deutsch & Barton J. Hirsch -- Esteem-enhancement interventions during early adolescence / David L. DuBois, Carol Burk-Braxton, & Heather D. Tevendale
    Pages: x, 395 p.
    ISBN: 0-585-46373-5
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  • 2
    Title: Hauptsächlich Elektromagnetismus und Struktur der Materie; 2
    Author: Feynman, Richard P.
    Contributer: Leighton, Robert B. , Sands, Matthew
    Publisher: München u.a. :Oldenbourg,
    Year of publication: 1987
    Pages: 851 S.
    Series Statement: Feynman Vorlesungen über Physik 2
    Type of Medium: Book
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  • 3
    Book
    Book
    München u.a. :Oldenbourg,
    Title: Quantenmechanik; 3
    Author: Feynman, Richard P.
    Contributer: Leighton, Robert B. , Sands, Matthew
    Publisher: München u.a. :Oldenbourg,
    Year of publication: 1988
    Pages: 503 S.
    Series Statement: Feynman Vorlesungen über Physik 3
    Type of Medium: Book
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  • 4
    Book
    Book
    Basel u.a. :Birkhäuser,
    Title: Combinatorics and commutative algebra; 41
    Author: Stanley, Richard P.
    Publisher: Basel u.a. :Birkhäuser,
    Year of publication: 1996
    Pages: 164 S.
    Series Statement: Progress in mathematics 41
    Type of Medium: Book
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  • 5
    Book
    Book
    Inc., Belmont, CA :Wadsworth,
    Title: Enumerative combinatorics
    Author: Stanley, Richard P.
    Publisher: Inc., Belmont, CA :Wadsworth,
    Year of publication: 1986
    Pages: 306 S.
    Series Statement: ¬The¬ Wadsworth & Brooks/Cole Mathematics Series
    Type of Medium: Book
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  • 6
    Title: Hauptsächlich Mechanik, Strahlung und Wärme; 1
    Author: Feynman, Richard P.
    Contributer: Leighton, Robert B. , Sands, Matthew
    Publisher: München u.a. :Oldenbourg,
    Year of publication: 1987
    Pages: 748 S.
    Series Statement: Feynman Vorlesungen über Physik 1
    Type of Medium: Book
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  • 7
    Title: QED - ¬Die¬ seltsame Theorie des Lichts und der Materie; 1562
    Author: Feynman, Richard P.
    Publisher: München u.a. :Piper,
    Year of publication: 1992
    Pages: 175 S.
    Series Statement: Serie Piper 1562
    Type of Medium: Book
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  • 8
    Electronic Resource
    Electronic Resource
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 55 (1990), S. 0 
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Adenosine transport by rat and guinea pig synaptosomes was studied to establish the basis for the marked differences in the potency of some transport inhibitors in these species. An analysis of transport kinetics in the presence and absence of nitrobenzylthioinosine (NBTI) using synaptosomes derived from several areas of rat and guinea pig brain indicated that at least three systems contributed to adenosine uptake, the Km values of which were ˜0.4, 3, and 15 μM in both species. In both species, the system with the Km of 3 μM was potently (IC50 of ˜0.3 nM) and selectively inhibited by NBTI. This NBTI-sensitive system accounted for a greater proportion of the total uptake in the guinea pig than in the rat and was inhibited by dipyridamole, mioflazine, and related compounds more potently in the guinea pig. Preliminary experiments with other species indicate that adenosine transport in the mouse is similar to that in the rat, whereas in the dog and rabbit, it is more like that in the guinea pig. In the rat, none of the systems appeared to require Na+, but the two systems possessing the higher affinities for adenosine were inhibited by veratridine- and K+-induced depolarization. The transport systems were active over a broad pH range, with maximal activity between pH 6.5 and 7.0. Our results are consistent with the possibility that adenosine transport systems may be differentiated into uptake and release systems.
    Type of Medium: Electronic Resource
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  • 9
    Electronic Resource
    Electronic Resource
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 61 (1993), S. 0 
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Abstract: Nuclear magnetic resonance (NMR) was used to study the metabolic pathways involved in the conversion of glucose to glutamate, γ-aminobutyrate (GABA), glutamine, and aspartate. d-[1-13C]Glucose was administered to rats intraperitoneally, and 6, 15, 30, or 45 min later the rats were killed and extracts from the forebrain were prepared for 13C-NMR analysis and amino acid analysis. The absolute amount of 13C present within each carbon-atom pool was determined for C-2, C-3, and C-4 of glutamate, glutamine, and GABA, for C-2 and C-3 of aspartate, and for C-3 of lactate. The natural abundance 13C present in extracts from control rats was also determined for each of these compounds and for N-acetylaspartate and taurine. The pattern of labeling within glutamate and GABA indicates that these amino acids were synthesized primarily within compartments in which glucose was metabolized to pyruvate, followed by decarboxylation to acetyl-CoA for entry into the tricarboxylic acid cycle. In contrast, the labeling pattern for glutamine and aspartate indicates that appreciable amounts of these amino acids were synthesized within a compartment in which glucose was metabolized to pyruvate, followed by carboxylation to oxaloacetate. These results are consistent with the concept that pyruvate carboxylase and glutamine synthetase are glia-specific enzymes, and that this partially accounts for the unusual metabolic compartmentation in CNS tissues. The results of our study also support the concept that there are several pools of glutamate, with different metabolic turnover rates. Our results also are consistent with the concept that glutamine and/or a tricarboxylic acid cycle intermediate is supplied by astrocytes to neurons for replenishing the neurotransmitter pool of GABA. However, a similar role for astrocytes in replenishing the transmitter pool of glutamate was not substantiated, possibly due to difficulties in quantitating satellite peaks arising from 13C-13C coupling.
    Type of Medium: Electronic Resource
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  • 10
    Electronic Resource
    Electronic Resource
    Oxford, UK : Blackwell Publishing Ltd
    Journal of neurochemistry 49 (1987), S. 0 
    ISSN: 1471-4159
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Several ions commonly used as substitutes for Na+ or Cl- were found to inhibit directly the high-affinity uptake of norepinephrine, dopamine, serotonin, and γ-aminobutyric acid, but not glutamate or glutamine. When Na+ was partially replaced by any of several different cations or sucrose the uptake of all neurotransmitters studied except that of serotonin was reduced more than could be accounted for by just the inhibitory effect of the cation substitute. In contrast, when Cl was partially replaced by any of several anions only the uptake of dopamine was reduced more than could be accounted for by the inhibitory effect of the anion substitute. These results suggest that for most neurotransmitters the electrochemical potential for Na+, but not for Cl, contributes to the uptake driving force. When either Na+ or Cl was totally replaced by an ion substitute or by sucrose the high-affinity uptake was virtually abolished, an exception being that glutamate uptake was not affected when isethionate was substituted for Cl. The lack of uptake in the absence of either Na+ or Cl may reflect a specific role for these ions in either increasing the affinity between the substrate and the carrier, or facilitating the translocation process. Alternatively, the transport carriers may undergo a nonspecific conformational change to an inactive form in the absence of Na+ or Cl. A partial substitution of Na+ with Li+ or sucrose differentially affected the kinetics of uptake in that replacement with Li+, but not sucrose, usually resulted in a marked increase in the Km values. The results of this study emphasize the importance of taking into consideration the effects of ions used as substitutes in experiments undertaken to elucidate the roles of Na+ and Cl in the high-affinity uptake of monoamine and amino acid neurotransmitters.
    Type of Medium: Electronic Resource
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