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  • 1
    Electronic Resource
    Electronic Resource
    Prediction of infusion rates of rocuronium using the bolus test dose technique (1997)
    Oxford : Blackwell Science Ltd
    Anaesthesia 52 (1997), S. 0 
    Details
    ISSN: 1365-2044
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Twenty-four patients were given a loading dose of rocuronium 1.0 mg.kg−1 intravenously followed by boluses of 20 mg (n = 19) and 10 mg (n = 24) after return of T1 of the train-of-four to 5% of control. Neuromuscular function was assessed using a Relaxograph. The time was recorded for the return of T1 to 5% after the administration of the boluses and subsequently an infusion of rocuronium was started. The aim was to maintain T1 between 3% and 7% of control for at least 40 min without a change of infusion rate. The correlations between the duration of the test doses and the infusion rates were −0.94 (10 mg) and −0.86 (20 mg). The predictive accuracy of the 10 mg bolus was assessed in a further 10 patients. At the termination of the infusion three patients had a T1% that was outside the desired range of 3–7%. A 10 mg bolus that lasts 6 min indicates a need for an infusion of at least 60 mg.h−1, 8 min (50 mg.h−1), 10 min (40 mg.h−1), 15 min (30 mg.h−1), 24 min (20 mg.h−1) and 34 min (15 mg.h−1).
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2044.1997.001-az004.x
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  • 2
    Electronic Resource
    Electronic Resource
    Odontogenic myxoma in an adolescent with tuberous sclerosis (1997)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of oral pathology & medicine 26 (1997), S. 0 
    Details
    ISSN: 1600-0714
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Tuberous sclerosis is a hamarloneoplastic syndrome characterised by early onset of convulsive seizures with mental retardation. Oral abnormalities, including enamel hypoplasia and mucosal fibromas, have been previously reported. We report here a 17-year-old girl with this disorder who had received repeated dental trauma. She presented with a swelling associated with a traumatised maxillary central incisor that suggested a diagnosis other than the Odontogenic myxoma found at operation. Odontogenic myxoma has not previously been reported in this condition; it may represent an unusual phenotypic expression of the genetic defect understood to determine tuberous sclerosis, or it may be an unrelated coincidental finding.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1600-0714.1997.tb00226.x
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    High magnetic susceptibility coatings for visualisation of optical fibres on a specialised interventional MRI system (1997)
    Springer
    European radiology 7 (1997), S. 1341-1342 
    Details
    ISSN: 1432-1084
    Keywords: Key words: Magnetic resonance imaging (MRI) ; Interventional MRI ; Fibre optics ; Laser therapy ; MR-guided therapy ; Specialised MRI systems
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract. The aim of this study was to develop methods of visualising optical fibres on MRI scans for monitoring interstitial laser therapy. Scans were performed on a specialised MRI extremity scanner at 0.17 T. Optical fibres of 0.4 mm diameter used for delivering laser energy were coated with iron particles from a superferromagnetic contrast agent. MR images of the fibres were acquired using gradient echo sequences (TR/TE = 300/10, 1 mm in-plane, 3 mm slice) and assessed for fibre visibility. Coated fibres could be resolved as lines 2 ± 1 mm wide using the gradient echo sequence. Uncoated fibres were invisible on the sequences used for in vivo therapy monitoring due to partial volume averaging. It is concluded that optical fibre visualisation by MRI may be improved by coating with ferromagnetic particles. Biocompatibility requires further assessment, but direct coating appears to be a promising method for fibre visualisation in MR-guided laser therapy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003300050300
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  • 4
    Electronic Resource
    Electronic Resource
    Percutaneous fetal access and uterine closure for fetoscopic surgery (1997)
    Springer
    Surgical endoscopy and other interventional techniques 11 (1997), S. 819 -824 
    Details
    ISSN: 1432-2218
    Keywords: Key words: Fetoscopy — Fetal surgery — Percutaneous fetal access — Uterine closure — Sheep
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Background: Maternal morbidity and preterm labor from fetal surgery might be minimized by a percutaneous technique for fetal access and uterine closure. Methods: In each of 16 ewes, we inserted three trocars percutaneously into the amniotic cavity using ultrasound and fetoscopic guidance. In six ewes, percutaneous uterine closure after the procedure was attempted. We assessed feasibility and acute complications of our technique during surgery and at autopsy. Results: We achieved percutaneous fetal access in 14 ewes and closed the uterus percutaneously in all six ewes attempted. Fetal injury was related to amnioinfusion or fixation of chorioamniotic membranes. Other complications were trocar dislodgment and damage to uterine wall and chorioamniotic membranes. The latter complication was prevented using balloon-tipped trocars. Conclusions: Percutaneous intraamniotic access and uterine closure for fetoscopic surgery can be achieved reliably with little maternal and fetal morbidity in sheep. Minor modifications are desired to apply this approach in humans.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004649900462
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  • 5
    Electronic Resource
    Electronic Resource
    Effects of Combined Glutamate and Platelet-Activating Factor Inhibition on the Outcome of Focal Cerebral Ischaemia - an Initial Screening Study (1997)
    Springer
    Metabolic brain disease 12 (1997), S. 237-249 
    Details
    ISSN: 1573-7365
    Keywords: Glutamate antagonists ; middle cerebral artery occlusion ; neuroprotection ; platelet-activating factor
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Since both glutamate excitotoxicity and inflammatory responses have been implicated in ischaemic neuronal death, we questioned whether joint inhibition of both processes would be more neuroprotective than either on its own. Therefore we assessed the effects of combined inhibition of both glutamate release (with a use-dependant sodium channel blocker, 619C89) and inflammatory processes (with a platelet-activating factor (PAF) receptor antagonist, BB-823) on the degree of motor deficit and the extent of cerebral (cortical and sub-cortical grey matter) infarction produced by middle cerebral artery occlusion (MCAO) in the rat, and compared results to appropriate single agent, vehicle and positive controls. The combination of both agents produced the greatest reduction in motor deficit, but the effect was only significant (p〈0.05) acutely (4 to 6 hours post-MCAO). The extent of cortical infarction at 24 hours post-MCAO was significantly reduced in all experimental groups compared to vehicle-controls (p〈0.05) and the greatest reduction occurred in the combination group (55%), though it was not significantly better than either of the single agent groups. Similarly the greatest reduction in sub-cortical infarction was in the combination group, but this was also not significantly better than the single agents. The results of this novel combination of pharmacological interventions suggest that inhibition of both glutamate excitotoxicity and inflammatory responses afforded an overall enhanced, if modest, neuroprotective effect, compared to inhibition of either process alone. The possible mechanisms involved are discussed, but warrant further clarification before therapeutic strategies are developed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/B:MEBR.0000007104.75056.e4
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