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  • Helicobacter pylori  (3)
  • Diffusion  (2)
  • Occupational exposure  (2)
  • Antimicrobial treatment  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Specific detection of Helicobacter pylori and non–Helicobacter pylori flora in small- and large-cell primary gastric B-cell non-hodgkin's lymphoma (1997)
    Springer
    Annals of oncology 8 (1997), S. 33-36 
    Details
    ISSN: 1569-8041
    Keywords: bacterial flora ; Helicobacter pylori ; immunohisochemistry ; MALT ; primary gastric lymphoma
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Background: Primary gastric non-Hodgkin's lymphomas possibly develop in response to local infection by Helicobacter pylori (H. pylori). We investigated the presence of H. pylori and non–H. pylori flora histologically in small- and large-cell primary gastric lymphoma using a specific staining method. Materials and methods: Specimens of 52 cases of primary gastric lymphoma(17 small cell, 35 large cell) were stained with modified Giemsa (MG) and immunohistochemically using a polyclonal antibody against H. pylori (IHC). Results: Thirty-two cases (61.5%) (small cell 76% versus large cell 53%,P 〉 0.05) showed immunoreactivity for H. pylori in the mucosa surrounding the tumor. Remarkable, there waslocalization of H. pylori in the neck of the gastric glands in 3 cases.Non– H. pylori flora was seen in 35 cases (76.3%) (small cell53% versus large cell 74%,P 〉 0.05). In 20cases, this non– H. pylori flora was mixed with H. pylori. Five cases showed no bacterial flora at all. Conclusions: (1)Using immunohistochemistry, the prevalence of gastriclymphoma cases with H. pylori (61.5%) approximates that of H. pylori in the normal population. (2) No statistical difference was found between the occurrence of H. pylori and non–H. pyloribacterial flora in small- versus large-cell lymphoma. (3) Our results suggest that H. pylori may not be the only etiologic factor in primary gastric lymphoma.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1008205510594
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Specific detection of Helicobacter pylori and non–Helicobacter pylori flora in small- and large-cell primary gastric B-cell non-hodgkin's lymphoma (1997)
    Springer
    Annals of oncology 8 (1997), S. 33-36 
    Details
    ISSN: 1569-8041
    Keywords: bacterial flora ; Helicobacter pylori ; immunohisochemistry ; MALT ; primary gastric lymphoma
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Background: Primary gastric non-Hodgkin's lymphomas possibly develop inresponse to local infection by Helicobacter pylori (H. pylori). Weinvestigated the presence of H. pylori and non–H. pylori florahistologically in small- and large-cell primary gastric lymphoma using aspecific staining method. Materials and methods: Specimens of 52 cases of primary gastric lymphoma(17 small cell, 35 large cell) were stained with modified Giemsa (MG) andimmunohistochemically using a polyclonal antibody against H. pylori (IHC). Results: Thirty-two cases (61.5%) (small cell 76% versuslarge cell 53%,P 〉 0.05) showed immunoreactivityfor H. pylori in the mucosa surrounding the tumor. Remarkable, there waslocalization of H. pylori in the neck of the gastric glands in 3 cases.Non– H. pylori flora was seen in 35 cases (76.3%) (small cell53% versus large cell 74%,P 〉 0.05). In 20cases, this non– H. pylori flora was mixed with H. pylori. Five casesshowed no bacterial flora at all. Conclusions: (1)Using immunohistochemistry, the prevalence of gastriclymphoma cases with H. pylori (61.5%) approximates that of H. pylori in the normal population. (2) No statistical difference was foundbetween the occurrence of H. pylori and non–H. pyloribacterial flora in small- versus large-cell lymphoma. (3) Our resultssuggest that H. pylori may not be the only etiologic factor in primarygastric lymphoma.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1008205510594
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    Paper (German National Licenses)
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  • 3
    Electronic Resource
    Electronic Resource
    The temperature dependency of45Ca diffusion in bovine enamel (1977)
    Springer
    Calcified tissue international 24 (1977), S. 173-177 
    Details
    ISSN: 1432-0827
    Keywords: Enamel ; Calcium ; Diffusion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine , Physics
    Notes: Summary The diffusion of45Ca ions in sound bovine enamel at pH 7.0 was studied as a function of temperature in the range 20°–40°C. The experimental data show that the diffusion is governed by two processes with activation energies of about 0.7 and 1.3 eV respectively. These values are considerably larger than the activation energy of calcium ion diffusion in water (0.2 eV). The diffusion process with the activation energy of 1.3 eV is dependent upon the calcium concentration in solution. A model based on the complex pore structure of enamel and on interactions of diffusing ions with the enamel constituents is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02223312
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  • 4
    Electronic Resource
    Electronic Resource
    Diffusion of45Ca in bovine enamel (1977)
    Springer
    Calcified tissue international 24 (1977), S. 59-64 
    Details
    ISSN: 1432-0827
    Keywords: Enamel ; Calcium ; Exchange ; Diffusion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine , Physics
    Notes: Summary The penetration of45Ca ions into whole bovine enamel from an aqueous solution was studied as a function of the Ca content in the solution. A sectioning technique was employed to measure the penetration of45Ca into the enamel. Probably only Ca ions occupying surface positions in the hydroxyapatite crystallites participate in the exchange process at the solution-enamel interface. The fraction of Ca ions participating in the exchange was 2% of the total number of Ca ions. Two different diffusion processes occur; firstly, a process governed by a diffusion coefficient, D1 ranging from about 5×10−14 to 2×10−12 cm2/s and strongly dependent on the calcium concentration in the solutions; and secondly a process with a concentration-independent diffusion coefficient, D2, with a value of 2.8×10−12 cm2/s. It is assumed that the 2 diffusion types correspond to intra-and interprismatic transport, respectively. A model based on the pore structure of dental enamel and on surface charges on hydroxyapatite crystallites is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02223297
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    Paper (German National Licenses)
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  • 5
    Electronic Resource
    Electronic Resource
    Clinical use of selective decontamination: The concept (1990)
    Springer
    Intensive care medicine 16 (1990), S. S212 
    Details
    ISSN: 1432-1238
    Keywords: Infection ; Selective decontamination ; Immunocompromised ; Antimicrobial treatment ; Colonization resistance
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Infections can be classified according to: (1) the type of offending microorganism (virus, bacteria, fungi, parasites), (2) according to the clearance by the defence system (T cell dependent/independent) and (3) in case bacteria are the causative agents in Gram-positive and Gram-negative infections. The latter classification in Gram-positive and Gram-negative infections has appeared to have a practical consequence. Gram-negative bacteria, often involved in major infections and yeasts, appear to play practically no role in the intestinal ecological system. Consequently, it is nowadays increasingly attempted to eliminate Gram-negative bacteria and yeasts selectively from the digestive tract with antimicrobial agents. Selective suppression of Gram-positive bacteria may severely affect the ecosystem of the digestive tract. This selective suppression of Gram-negatives must be continued as long as patients are immunocompromised (locally or systemically) and is called selective decontamination of the digestive tract.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01709703
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    Paper (German National Licenses)
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  • 6
    Electronic Resource
    Electronic Resource
    Investigation of a cluster of ten cases of Hodgkin’s disease in an occupational setting (1996)
    Springer
    International archives of occupational and environmental health 68 (1996), S. 224-228 
    Details
    ISSN: 1432-1246
    Keywords: Key words Hodgkin’s disease ; Occupational exposure ; Nested case-control study.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract  The objective of this study was to identify occupational exposures that might be etiologically linked to an unusual cluster of ten cases of Hodgkin’s disease. The cases were identified within the active workforce of a large chemical manufacturing firm over a 23-year period by the medical director of the facilities. Based on comparison with regional cancer incidence rates, the standardized incidence ratio for Hodgkin’s disease was 497 (95% confidence interval: 238–915) for the period from the construction of the facilities in 1966 through early 1992. A nested case-control study was undertaken with 200 controls selected according to case-cohort sampling. Simultaneously, efforts were initiated to confirm and characterize each case more fully. Occupational exposures were identified and categorized using process, work history, medical record, and industrial hygiene data. Tissue slides were available for eight cases and a second review confirmed the diagnosis of Hodgkin’s disease. For one case, a final diagnosis of large-cell anaplastic lymphoma was determined after histology review. Among 214 different chemical agents studied, eight were identified to which three or more of the cases had been exposed prior to the date of their initial diagnosis. Exposure odds ratios were statistically elevated for five of these agents; dose-response evaluations for two of the agents, ethylene oxide and benzene, failed to provide additional support for a causal relationship. In conclusion, although several statistical associations were identified, no substance emerged as a likely candidate for explaining the observed Hodgkin’s disease cluster.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00381432
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    Paper (German National Licenses)
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  • 7
    Electronic Resource
    Electronic Resource
    Investigation of a cluster of ten cases of Hodgkin's disease in an occupational setting (1996)
    Springer
    International archives of occupational and environmental health 68 (1996), S. 224-228 
    Details
    ISSN: 1432-1246
    Keywords: Hodgkin's disease ; Occupational exposure ; Nested case-control study
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The objective of this study was to identify occupational exposures that might be etiologically linked to an unusual cluster of ten cases of Hodgkin's disease. The cases were identified within the active workforce of a large chemical manufacturing firm over a 23-year period by the medical director of the facilities. Based on comparison with regional cancer incidence rates, the standardized incidence ratio for Hodgkin's disease was 497 (95% confidence interval: 238–915) for the period from the construction of the facilities in 1966 through early 1992. A nested case-control study was undertaken with 200 controls selected according to case-cohort sampling. Simultaneously, efforts were initiated to confirm and characterize each case more fully. Occupational exposures were identified and categorized using process, work history, medical record, and industrial hygiene data. Tissue slides were available for eight cases and a second review confirmed the diagnosis of Hodgkin's disease. For one case, a final diagnosis of large-cell anaplastic lymphoma was determined after histology review. Among 214 different chemical agents studied, eight were identified to which three or more of the cases had been exposed prior to the date of their initial diagnosis. Exposure odds ratios were statistically elevated for five of these agents; dose-response evaluations for two of the agents, ethylene oxide and benzene, failed to provide additional support for a causal relationship. In conclusion, although several statistical associations were identified, no substance emerged as a likely candidate for explaining the observed Hodgkin's disease cluster.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00381432
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    Paper (German National Licenses)
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  • 8
    Electronic Resource
    Electronic Resource
    The prevalence of Helicobacter pylori serum antibodies in children with recurrent abdominal pain (1992)
    Springer
    European journal of pediatrics 151 (1992), S. 799-801 
    Details
    ISSN: 1432-1076
    Keywords: Recurrent abdominal pain ; Children ; Helicobacter pylori ; Serum antibodies
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract As part of a large, prospective study we investigated the prevalenceHelicobacter pylori serum antibodies in children with recurrent abdominal pain (RAP). All patients suffered from recurrent bouts of abdominal pain for at least 6 months and ranged in age from 6 to 12 years.H. pylori antibodies were detected using an enzyme-linked immunosorbent assay. The prevalence ofH. pylori antibodies in the RAP group was compared to that of a control group which consisted predominantly of pre-operative children. None of the control group suffered or had suffered from RAP. Antibodies toH. pylori were found in 7 of 82 (8.5%) RAP patients and in 2 of 39 (5.1%) control children. The latter difference is not significant and suggests that RAP is only rarely caused in children byH. pylori infection.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01957927
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