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  • 1
    Electronic Resource
    Electronic Resource
    Thermoregulation modulated by hypothalamic histamine in rats (1997)
    Springer
    Inflammation research 46 (1997), S. 35-36 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s000110050046
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  • 2
    Electronic Resource
    Electronic Resource
    Alpha-amylase inhibitor increases plasma 3-hydroxybutyric acid in food-restricted rats (1995)
    Springer
    Cellular and molecular life sciences 51 (1995), S. 585-588 
    Details
    ISSN: 1420-9071
    Keywords: α-amylase inhibitor ; plasma glucose ; 3-hydroxybutyric acid ; high starch diet
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract The effect on energy metabolism of delayed absorption of starch by inhibition of α-amylase was examined by considering levels of plasma glucose and 3-hydroxybutyric acid (3-OHBA) in rats. Addition of α-amylase inhibitor (αAI) to a high starch diet delayed the plasma glucose response after feeding: peak plasma glucose levels in the control group occurred 15 min after feeding, whereas in the αAI group this peak did not occur until 30 min after. The total plasma glucose response was not different between the two groups. Plasma 3-OHBA levels 1 day after food restriction increased approximately five-fold in both groups. After 3 days of food restriction, the αAI group maintained the same level of plasma 3-OHBA as after 1 day of food restriction, while the control group showed significantly decreased levels of 3-OHBA. After 3 days of food restriction, plasma insulin levels were significantly decreased in the αAI group compared with the corresponding levels of the control group and with levels before the restriction. There was no significant difference in body weight between the two groups. These findings suggest that delayed hyperglycemia due to delayed absorption of starch following αAI loading may attenuate insulin secretion, leading to altered metabolism of 3-OHBA during the delayed response to energy deficit.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02128748
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  • 3
    Electronic Resource
    Electronic Resource
    Intrahepatic cholangiocarcinoma with marked hypervascularity (1999)
    Springer
    Abdominal imaging 24 (1999), S. 66-68 
    Details
    ISSN: 1432-0509
    Keywords: Key words: Intrahepatic cholangiocarcinoma—Hypervascular.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Two rare cases of small intrahepatic cholangiocarcinoma with marked hypervascularity are reported. Dynamic computed tomographic and magnetic resonance images of the two cases revealed strong enhancement of the whole tumor on the early phase and prolonged enhancement on the late and delayed phases. In both cases, the tumors turned out to be well-differentiated tubular cholangiocarcinoma that contained a large number of tumor cells and few interstitial fibrous tissues. These results suggest that some intrahepatic cholangiocarcinoma should be differentiated from other hypervascular hepatic tumors, especially hepatocelluar carcinoma, and that prolonged enhancement of the tumor on late and delayed phases of dynamic images could be of diagnostic value.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s002619900442
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  • 4
    Electronic Resource
    Electronic Resource
    Identification of an abdominal ganglion neuron antagonizing L7-driven gill contraction in Aplysia (1999)
    Springer
    Journal of comparative physiology 185 (1999), S. 11-19 
    Details
    ISSN: 1432-1351
    Keywords: Key words Aplysia ; Gill ; Motor neuron ; L7 ; Abdominal ganglion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Gill motor neuron L7-induced longitudinal shortening of the gill in Aplysia kurodai and A. juliana was suppressed when extracellular stimuli were applied to a restricted dorsal central region of the abdominal ganglion. We found a neuron there which antagonized the L7-driven contraction. Since the contraction was suppressed when the identified neuron was activated simultaneously with L7, we refer to the newly found neuron as “Anti-L7”. Anti-L7 did not change the L7 impulse generation in the abdominal ganglion. No direct synaptic connection from L7 to Anti-L7 was detected. A fluorescent dye injected into the soma of Anti-L7 revealed that the neuron sent axonal branches to the branchial nerve. These results may show that Anti-L7 antagonizes L7 at the periphery inside the gill, rather than in the abdominal ganglion. EJPs induced by L7 were unaffected by Anti-L7. Activation of Anti-L7 alone did not induce any change in tone or membrane potential of the gill musculature. The suppressive effect of Anti-L7 lasts many seconds after the cessation of a train of Anti-L7 impulses. The results may suggest that the suppression is mediated through an inhibitory neuromodulatory mechanism without inhibition of L7 itself.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003590050361
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  • 5
    Electronic Resource
    Electronic Resource
    Functions of purified gB, gE:gI, and gH:gL, and their sialyl residues in varicella-zoster virus infection (1997)
    Springer
    Archives of virology 142 (1997), S. 2295-2301 
    Details
    ISSN: 1432-8798
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summery.  Varicella-zoster virus glycoproteins were purified by using monoclonal antibodies and analyzed for their effects on cell-free virus infection. Preinfection treatment of cells with gH:gL reduced the infection efficiency and increased the number of unadsorbed virus. Postinfection treatment of cells with gB increased the infection efficiency, but that with gE:gI reduced it. Treatment of gE:gI and gH:gL with neuraminidase (NA) abolished their inhibitory activity and the plaque formation was enhanced by NA treatment of glycoproteins and cells. Glycoproteins exhibited their diverse activities despite their common role in viral penetration, and sialyl residues were responsible for their function in cell-free virus infection.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s007050050243
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  • 6
    Electronic Resource
    Electronic Resource
    Lymphokine activated killer cells in murine coxsackievirus B3 myocarditis (1997)
    Springer
    Basic research in cardiology 92 (1997), S. 402-409 
    Details
    ISSN: 1435-1803
    Keywords: Myocarditis ; Coxsackievirus B3 ; Interleukine-2 ; Lymphokine activated killer cell ; Cytotoxic reaction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The purpose of the present study was to determine whether lymphokine activated killer (LAK) cells were involved in the development of coxsackievirus B3 (CB3) myocarditis in both the acute viremic (Experiment I) and the subacute aviremic (Experiment II) stages. To induce LAK cells, recombinant human interleukin-2 (IL-2) was administered to CB3-infected mice subcutaneously daily, starting on day 0 in Experiment I and on day 7 in Experiment II for 7 days, respectively. The treated groups were compared to infected controls. Splenic lymphocytes of IL-2 treated mice were further cultured in vitro in IL-2 containing medium for 7 days, and LAK cell activity, i.e., cytotoxic activity of the lymphocytes against EL-4 tumor cells and against cultured fetal myocytes, was assayed by51Cr-release method. In Experiment I, histologic scores, myocardial virus titers, and LAK cell activity did not differ significantly between IL-2 treated and untreated groups. In contrast, in Experiment II, there were more cellular infiltration associated with severe necrosis and higher LAK cell activity against EL-4 cells and cultured myocytes in IL-2 treated than in untreated groups. The presence of LAK cells was demonstrated in the subacute stage of murine CB3 myocarditis. Thus, the behavior of LAK cell activity may vary with the course of myocarditis, and enhanced LAK cell activity may be involved in the development of the disease.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00796214
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  • 7
    Electronic Resource
    Electronic Resource
    Lymphokine activated killer cells in murine coxsackievirus B3 myocarditis (1997)
    Springer
    Basic research in cardiology 92 (1997), S. 402-409 
    Details
    ISSN: 1435-1803
    Keywords: Key words Myocarditis – Coxsackievirus B3 – Interleukine-2 – Lymphokine activated killer cell – Cytotoxic reaction
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The purpose of the present study was to determine whether lymphokine activated killer (LAK) cells were involved in the development of coxsackievirus B3 (CB3) myocarditis in both the acute viremic (Experiment I) and the subacute aviremic (Experiment II) stages. To induce LAK cells, recombinant human interleukin-2 (IL-2) was administered to CB3-infected mice subcutaneously daily, starting on day 0 in Experiment I and on day 7 in Experiment II for 7 days, respectively. The treated groups were compared to infected controls. Splenic lymphocytes of IL-2 treated mice were further cultured in vitro in IL-2 containing medium for 7 days, and LAK cell activity, i.e., cytotoxic activity of the lymphocytes against EL-4 tumor cells and against cultured fetal myocytes, was assayed by 51Cr-release method. In Experiment I, histologic scores, myocardial virus titers, and LAK cell activity did not differ significantly between IL-2 treated and untreated groups. In contrast, in Experiment II, there were more cellular infiltration associated with severe necrosis and higher LAK cell activity against EL-4 cells and cultured myocytes in IL-2 treated than in untreated groups. The presence of LAK cells was demonstrated in the subacute stage of murine CB3 myocarditis. Thus, the behavior of LAK cell activity may vary with the course of myocarditis, and enhanced LAK cell activity may be involved in the development of the disease.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00796214
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  • 8
    Electronic Resource
    Electronic Resource
    Further measurement of the 7Be(p,γ)8B cross section at low energies with the coulomb dissociation of 8B (1998)
    Springer
    The European physical journal 3 (1998), S. 213-215 
    Details
    ISSN: 1434-601X
    Keywords: PACS: 25.40.Lw Radiative capture – 35.60.-t Reactions induced by unstable nuclei – 25.70.De Coulomb excitation – 25.70.Mn Projectile and target fragmentation – 26.65.+t Solar neutrinos
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract: We have measured the dissociation of 8B in the Coulomb field of 208Pb at Ein=51.9 MeV/nucleon and extracted the cross section of the 7Be(p,γ)8B reaction at 0.4 ≤ Erel≤ 3 MeV, which is of importance for the 8B solar-neutrino production rate. The extracted astrophysical S17 factors are consistent with our earlier Coulomb dissociation measurement, and agree with the values deduced from the direct capture measurements by Filippone et al., Vaughn et al. and Hammache et al. The S factor at zero energy was extracted to be 18.9±1.8 eV-b with the help of theoretical energy-dependence.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s100500050170
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  • 9
    Electronic Resource
    Electronic Resource
    New α-decaying neutron deficient isotopes197Rn and200Fr (1995)
    Springer
    The European physical journal 352 (1995), S. 7-8 
    Details
    ISSN: 1434-601X
    Keywords: 23.60.+e ; 25.70.−z ; 27.80.+w
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract New neutron-deficient isotopes,197Rn,197mRn, and200Fr have been produced and identified on the basis of genetic correlations in the166Er(36Ar,5n)197Rn (Elab=186, 200 MeV), and169Tm(36Ar,5n)200Fr (Elab=186 MeV) reactions. The evaporation residues were separated from the beam by using a gas-filled recoil separator and implanted onto a position-sensitive solid-state detector. The α-decay energies (half-lives) of197Rn,197mRn and200Fr have been determined to be 7261±30 keV (51 −15 +35 ms), 7370±30 keV (18 −5 +9 ms), and 7500±3O keV, (570 −140 +270 ms), respectively.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01292753
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