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  • 1
    Electronic Resource
    Electronic Resource
    Thickening of the basement membrane of cortical capillaries in Alzheimer's disease (1980)
    Springer
    Acta neuropathologica 49 (1980), S. 79-83 
    Details
    ISSN: 1432-0533
    Keywords: Alzheimer's disease ; Dementia ; Aging ; Cortical capillaries ; Basement membrane ; Electron microscopy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Data concerning width and surface of the basement membrane of cortical capillaries in three patients with Alzheimer's disease five age-matched controls, have been submitted to statistical analysis to ascertain the role of the aging process upon thickening of the basement membrane itself. The analysis showed that there was a significant difference between patients with Alzheimer's disease and controls (P〈0.01) as to both width and surface of the basement membrane, so suggesting that in these patients aging was not the only factor which thickening of the basement membrane of cortical capillaries was related to.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00692225
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Early myoclonus and quasiperiodic EEG changes in non-familial Alzheimer's disease (1979)
    Springer
    Neurological sciences 1 (1979), S. 181-187 
    Details
    ISSN: 1590-3478
    Keywords: Alzheimer's disease ; Creutzfeldt-Jakob ; Transmissible dementias
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario Una grande quantità di neuroni in degenerazione neurofibrillare e di placche senili, fu trovata nella corteccia cerebrale di una donna di 52 anni affetta da una demenza ad evoluzione subacuta, mioclonie e alterazioni elettroencefalografiche periodiche. È questo un caso sporadico di demenza presenile di Alzheimer clinicamente sovrapponibile alla malattia di Creutzfeldt-Jakob, che si presta a ipotizzare un rapporto fra quella malattia e le demenze trasmissibili.
    Notes: Abstract Large numbers of neurofibrillary tangles and neuritic plaques were found in a cerebral biopsy from a 52-year-old woman suffering from subacute dementia with early myoclonus and quasiperiodic EEG changes. This case, which appeared to be an example of non-familial Alzheimer's disease, is commented in the context of possible relation of Alzheimer's disease to transmissible dementia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02335849
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    Paper (German National Licenses)
    Fulltext
  • 3
    Electronic Resource
    Electronic Resource
    Screening of the macromolecular component of CSF from various pathologies for its interference with GABAA receptor function (1995)
    Springer
    Neurological sciences 16 (1995), S. 643-646 
    Details
    ISSN: 1590-3478
    Keywords: CSF ; Neurological diseases ; Macromolecules ; GABAA receptors
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Sommario Campioni di liquido cefalo-rachidiano di pazienti analizzati per casi di sospette malattie neurologiche sono stati separati nella componente macromolecolare (Peso Molecolare 〉3000 Daltons) ed in quella delle piccole molecole (PM〈3000 Daltons). La c omponente macromolecolare è stata testata per la sua possibile interferenza con l'accumulo di36Cl−, stimolato dal GABA, in “microsacs” della corteccia cerebrale di ratto. Per molte patologie non è stata rilevata alcuna interferenza. Fanno eccezione i pochi (2) casi di morbo di Parkinson; in entrambi i casi vi è una chiara stimolazione dell'attività GABAergica. Quest' ultima indicazione sembra degna di ulteriori indagini.
    Notes: Abstract Cerebrospinal fluid (CSF) samples from patients undergoing lumbar puncture for suspected neurological diseases were fractioned into macromolecular (MW〉3000 Daltons) and small molecule components (MW〈3000 Daltons). The macromolecular component was tested for its interference with GABA stimulation of36Cl− accumulation in rat cerebral cortex microsacs. For many pathologies, no interference was apparent; an exception was two patients with Parkinson's disease in whom there were clear signs of stimulated GABA activity. This last result seems worthy of further investigation.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02230915
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    Paper (German National Licenses)
    Fulltext
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