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  • 11
    Electronic Resource
    Electronic Resource
    Latex allergy in spina bifida patients – prevention by primary prophylaxis (1998)
    Oxford, UK : Blackwell Publishing Ltd
    Allergy 53 (1998), S. 0 
    Details
    ISSN: 1398-9995
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Background The effect of latex prophylaxis has not been investigated in spina bifida children, a high-risk group for latex allergy. As repeated operations have been identified as a major cause of latex sensitization, we wanted to find out whether primary latex prophylaxis during surgery could prevent latex allergy in children with spina bifida. Methods In December 1995, we established latex-free surgery and anesthesia for all patients with spina bifida regardless of their sensitization to latex. Twelve children born after that date (mean age 1.2 years, mean ntmiber of operations 3.3, range 1 -7) were tested for specific IgE against latex until December 1997 (ImmunoCap, Pharmacia, Uppsala, Sweden) and compared with eight children born before December 1995 (mean age 1.3 years, mean number of operations 3.6, range 1–8), in whom a test for latex IgE had been done before the age of 2 years. Results Before we established primary prophylaxis, three of seven children with spina bifida (38%) were sensitized to latex until the age of 2 years. After the establishment of a latex-free operating theater for spina bifida patients, none of the 12 patients were sensitized to latex despite up to seven operations in each child. Conclusions Primary latex prophylaxis during surgery can prevent latex sensitization in young spina bifida patients.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1398-9995.1998.tb03958.x
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  • 12
    Electronic Resource
    Electronic Resource
    Schwere arterielle Hypertonie als Manifestation einer Nierenarterienstenose bei Williams-Beuren-Syndrom (1998)
    Springer
    Monatsschrift Kinderheilkunde 146 (1998), S. 862-864 
    Details
    ISSN: 1433-0474
    Keywords: Schlüsselwörter Williams Beuren-Syndrom ; Nierenarterienstenose ; Renovaskuläre Hypertonie ; Key words Williams-Beuren-syndrom ; Renovascular hypertension ; Renal artery stenosis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Summary Patients with Williams-Beuren-Syndrome characterized by the combination of typical morphology, supravalvular aortic stenosis and mental retardation. The typical cardiac malformation, the supravalvular aortic stenosis may be absent. Progressive peripheral vascular lesions as renal artery stenosis, coronary vascular abnormalities and cerebrovascular ischemia were not commonly observed in the past. Case report: We present a 12 year old girl with Williams-Beuren-Syndrome without typical signs of cardiac involvement who developed renovascular hypertension after 9 years of follow up due to stenosis of a renal artery. It was successfully treated by aortorenal bypass. Discussion: In the absence of typical cardiac involvement Patients with Williams-Beuren Syndrome can develop progressive vascular lesions as renal artery stenosis, coronary artery stenosis or cerebro-vascular lesions. Therefore irrespective of cardiac involvement patients with Willliams-Beuren syndrome should be followed up with special regards to this complication.
    Notes: Zusammenfassung Patienten mit Williams-Beuren-Syndrom werden durch die klassischen Symptome der auffälligen Morphologie des Gesichts, der supravalvulären Aortenstenose und der mentalen Retardierung beschrieben. Die klassische kardiale Beteiligung in Form der supravalvulären Aortenstenose kann bei Patienten mit Williams-Beuren-Syndrom fehlen. Unabhängig davon können progressive periphere Gefäßveränderungen bestehen. Diese können z. B. als Nierenarterienstenose, Koronaranomalie oder zerebrovaskuläre Ischämie symptomatisch werden. Fallbericht: Wir berichten über 1 Patientin mit Williams-Beuren-Syndrom ohne die kardiale Veränderung der supravalvulären Aortenstenose, die im Alter von 10 Jahren eine schwere arterielle Hypertonie infolge einer einseitigen Nierenarterienstenose entwickelte. Die Anlage eines aortorenoarterialen Bypasses war eine effektive Behandlung. Diskussion: Auch beim Fehlen der klassischen Symptomen des Williams-Beuren-Syndroms müssen progrediente Gefäßveränderungen rechtzeitig erkannt und als bedeutsame Komplikationen des Krankheitsverlaufs bedacht werden.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001120050336
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  • 13
    Electronic Resource
    Electronic Resource
    In vivo activity of asialo-erythropoietin in combination with asialo-glycoproteins (1982)
    Springer
    Annals of hematology 44 (1982), S. 173-175 
    Details
    ISSN: 1432-0584
    Keywords: Asialo-glycophorin ; Asialo-erythropoietin-activation in vivo
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary In vitro active asialo-erythropoietin has no effect on hemesynthesis in vivo. Asialo-glycophorin induces a very low 59Fe-uptake rate in heme in exhypoxic polycythemic mice. The combination of asialo-erythropoietin and asialo-glycophorin or asialo-fetuin induced an activation comparable to the activation by native erythropoietin. Obviously the combination of asialo-erythropoietin and tested glycoproteins influence the activity of erythropoiesis-stimulating capacity of asialo-erythropoietin.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00320764
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  • 14
    Electronic Resource
    Electronic Resource
    Onkologie (1999)
    Springer
    Monatsschrift Kinderheilkunde 147 (1999), S. 911-911 
    Details
    ISSN: 1433-0474
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001120050514
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  • 15
    Electronic Resource
    Electronic Resource
    Hypersensitivitätssyndrom durch Antikonvulsiva (1997)
    Springer
    Monatsschrift Kinderheilkunde 145 (1997), S. 109-113 
    Details
    ISSN: 1433-0474
    Keywords: Schlüsselwörter Carbamazepin ; Hypersensitivitätssyndrom ; Fieber ; Exanthem ; Lymphadenopathie ; Key words Carbamazepine ; Hypersensitivity syndrome ; Fever ; Skin rash ; Lymphadenopathy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Summary 2 weeks after initiation of carbamazepine therapy a 3 year old boy presented with skin rash, fever, lymphadenopathy, hepatosplenomegaly and nephritis. After exclusion of infectious diseases our differential diagnosis focussed on carbamazepine induced hypersensitivity syndrome. We found a positive lymphocyte transformation assay and patch test to carbamazepine, indicating a specific response, but no anti-carbamazepine antibody, no circulating immune complexes and no decrease in complement components. The hypersensitivity syndrome refers to a severe hypersensitivity response due to a idiosyncratic reaction. The origin is unknown. The similarity to other multisystem disorders is discussed. Discussion: Not only aromatic antiepileptic agents (carbamazepine, phenobarbital, phenytoin) sulfonamides but as well allopurinol and gold salts are associated with the syndrome. Withdrawal of the drug and treatment with corticosteroids usually lead to complete recovery.
    Notes: Zusammenfassung Wir berichten über einen 3 jährigen Jungen, der 2 Wochen nach Beginn einer antiepileptischen Behandlung an einem Carbamazepin-induzierten Hypersensitivitätssyndrom, bestehend aus therapieresistentem Fieber, polymorphem Exanthem, oropharyngealen Läsionen, generalisierter Lymphadenopathie, Hepatosplenomegalie und Nephritis, erkrankte. Wir fanden positive Lymphozytenstimulations- und Epikutantests auf Carbamazepin als Ausdruck einer spezifischen Überempfindlichkeit. Spezifische Anti-Carbamazepin-Antikörper, zirkulierende Immunkomplexe oder eine Komplementerniedrigung waren nicht nachweisbar. Es handelt sich um eine Überempfindlichkeitsreaktion unbekannter Ätiologie im Sinn einer Idiosynkrasie. Nach Ausschluß infektiöser Ursachen wird die Differentialdiagnose zu anderen Systemerkrankungen erörtert. Diskussion: Neben aromatischen Antikonvulsiva (Carbamazepin, Phenobarbital, Phenytoin) sind Sulfonamide und Goldsalze mögliche Auslöser eines Hypersensitivitätssyndroms. Absetzen des Induktors und systemische Kortikosteroide führen in der Regel zur vollständigen Remission.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001120050110
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