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  • 1
    Electronic Resource
    Electronic Resource
    CLINICAL EXPERIENCES WITH CHLORPROPAMIDE, WITH SPECIAL REFERENCE TO COMPARISON WITH PREVIOUS EXPERIENCE WITH CARBUTAMIDE AND TOLBUTAMIDE (1959)
    Oxford, UK : Blackwell Publishing Ltd
    Annals of the New York Academy of Sciences 74 (1959), S. 0 
    Details
    ISSN: 1749-6632
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Natural Sciences in General
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1749-6632.1959.tb39600.x
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  • 2
    Electronic Resource
    Electronic Resource
    Transition metal complexes as ligands, Dioxodithithiotungstate ion WO2S22- (1974)
    s.l. : American Chemical Society
    Inorganic chemistry 13 (1974), S. 1001-1002 
    Details
    ISSN: 1520-510X
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ic50134a049
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  • 3
    Electronic Resource
    Electronic Resource
    Laminar neuropathology in Alzheimer's disease by a modified Gallyas impregnation
    Amsterdam : Elsevier
    Psychiatry Research 29 (1989), S. 463-465 
    Details
    ISSN: 0165-1781
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0165-1781(89)90124-8
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  • 4
    Electronic Resource
    Electronic Resource
    Altered P300 topography due to functional and structural disturbances in the limbic system in dementia and psychoses and to pharmacological conditions
    Amsterdam : Elsevier
    Psychiatry Research 29 (1989), S. 391-393 
    Details
    ISSN: 0165-1781
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0165-1781(89)90099-1
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  • 5
    Electronic Resource
    Electronic Resource
    Pulmonary and systemic embolism after deliberate intravenous fluorocarbon administration (1980)
    Springer
    Virchows Archiv 386 (1980), S. 331-341 
    Details
    ISSN: 1432-2307
    Keywords: Fluorocarbon ; Extracorporeal circulation (EEC) ; Pulmonary ; systemic embolism ; Rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary After intravenous injection of 0.1 ml Fluorocarbon (FC) into the caudal vein of rats clear droplets which are reminiscent of gas emboli appear in the pulmonary and cerebral arteries. These droplets cannot be stained with Azan, haematoxylin-eosin, Nile blue sulfate, Sudan black B, and Sudan III in Paraplast embedded or frozen sections. Gas chromatography of affected lung tissue reveals a high concentration of FC. The clear droplets are the histological correlates of FC emboli which lead to haemorrhagic lung infarction and ischaemic brain infarcts. After intralienal injection FC induces haemorrhagic infarcts of the spleen near the injection site and massive embolization of the intrahepatic portal veins with consequent liver cell necrosis. FC droplets are phagocytosed by hepatic sinusoidal lining cells. Due to the absence of a specific method for identifying FC embolization of renal vessies is difficult to assess.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00427302
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  • 6
    Electronic Resource
    Electronic Resource
    Cortical and striatal neurone number in Huntington's disease (1994)
    Springer
    Acta neuropathologica 88 (1994), S. 320-333 
    Details
    ISSN: 1432-0533
    Keywords: Key words Huntington's disease ; Human cerebral cortex ; Striatum ; Neurone number ; Stereology
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The total cortical and striatal neurone and glial numbers were estimated in five cases of Huntington's disease (three males, two females) and five age- and sex-matched control cases. Serial 500-μm-thick gallocyanin-stained frontal sections through the left hemisphere were analysed using Cavalieri's principle for volume and the optical disector for cell density estimations. The average cortical neurone number of five controls (mean age 53±13 years, range 36 – 72 years) was 5.97×109±320×106, the average number of small striatal neurones was 82×106±15.8×106. The left striatum (caudatum, putamen, and accumbens) contained a mean of 273×106±53×106 glial cells (oligodendrocytes, astrocytes and unclassifiable glial profiles). The mean cortical neurone number in Huntington's disease patients (mean age 49±14 years, range 36 – 75 years) was diminished by about 33  % to 3.99×109±218×106 nerve cells (P≤ 0.012, Mann-Whitney U-test). The mean number of small striatal neurones decreased tremendously to 9.72 × 106± 3.64×106 ( – 88  %). The decrease in total glial cells was less pronounced (193 × 106±26 × 106) but the mean glial index, the numerical ratio of glial cells per neurone, increased from 3.35 to 22.59 in Huntington's disease. Qualitatively, neuronal loss was most pronounced in supragranular layers of primary sensory areas (Brodmann's areae 3,1,2; area 17, area 41). Layer IIIc pyramidal cells were preferentially lost in association areas of the temporal, frontal, and parietal lobes, whereas spared layer IV granule cells formed a conspicuous band between layer III and V in these fields. Methodological issues are discussed in context with previous investigations and similarities and differences of laminar and lobar nerve cell loss in Huntington's disease are compared with nerve cell degeneration in other neuropsychiatric diseases.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00310376
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  • 7
    Electronic Resource
    Electronic Resource
    Nerve cell loss in the thalamic centromedian-parafascicular complex in patients with Huntington's disease (1996)
    Springer
    Acta neuropathologica 91 (1996), S. 161-168 
    Details
    ISSN: 1432-0533
    Keywords: Key words Huntington's disease ; Human brain ; Thalamus ; Nuclei centromedianus-parafascicularis ; Neurone number
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The centromedian-parafascicular complex represents a nodal point in the neuronal loop comprising striatum – globulus pallidus – thalamus – striatum. Striatal neurone degeneration is a hallmark in Huntington's disease and we were interested in estimating total neurone and glial number in this thalamic nuclear complex. Serial 500-μm-thick gallocyanin-stained frontal sections of the left hemisphere from six cases of Huntington's disease patients (three females, three males) and six age- and sex-matched controls were investigated applying Cavalieri's principle and the optical disector. Mean neurone number in the controls was 646,952 ± 129,668 cells versus 291,763 ± 60,122 in Huntington's disease patients (Mann-Whitney U-test, P 〈 0.001). Total glial cell number (astrocytes, oligodendrocytes, microglia, and unclassifiable glial profiles) was higher in controls with 9,544,191 ± 3,028,944 versus 6,961,989 ± 2,241,543 in Huntington's disease patients (Mann-Whitney U-test, P 〈 0.021). Considerable increase of fibrous astroglia within the centromedian-parafascicular complex could be observed after Gallyas' impregnation. Most probably this cell type enhanced the numerical ratio between glial number and neurone number (glial index: Huntington's disease patients = 24.4 ± 8.1; controls = 15.0 ± 5.2; Mann-Whitney U-test, P 〈 0.013). The neurone number in the centromedian-parafascicular complex correlated negatively, although statistically not significantly, with the striatal neurone number. This lack of correlation between an 80% neuronal loss in the striatum and a 55% neurone loss in the centromedian-parafascicular complex points to viable neuronal circuits connecting the centromedian-parafascicular complex with cortical and subcortical regions that are less affected in Huntington's disease.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004010050408
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  • 8
    Electronic Resource
    Electronic Resource
    Quantitative anatomical studies on the postnatal development of the cerebellum of the albino rat (1977)
    Springer
    Anatomy and embryology 151 (1977), S. 201-218 
    Details
    ISSN: 1432-0568
    Keywords: Cerebellum ; Albino rat ; Ontogeny ; Quantitative anatomy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The quantitative postnatal changes of the cerebella of 65 Wistar rats aged 2–120 days have been examined. The cerebellar volume increases in two phases: The first phase lasts from birth to the seventh postnatal week. The second phase begins ten weeks post partum and lasts for a longer period than the first phase. The cerebellar surface increases continuously from birth to the end of the seventh week. The volume of the external granular layer is maximal when the organ grows rapidly. The external granular layer has nearly disappeared 24 days after birth; the volume of the internal granular layer is maximal at this time. Later on, the volume and the width of the internal granular layer decrease. Myelinization of the cerebellar fibers and growth of the molecular layer run parallel to this decrease. The second late, but protracted growth of the cerebellum, ten weeks after birth, is due to an increase of the molecular and medullary layer. These findigns are in good accord with histological, histochemical, and ultrastructural observations of other authors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00297481
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  • 9
    Electronic Resource
    Electronic Resource
    Postnatal quantitative changes in the cerebellar uvula of albino rats (1978)
    Springer
    Anatomy and embryology 154 (1978), S. 285-304 
    Details
    ISSN: 1432-0568
    Keywords: Cerebellum ; Albino rat ; Postnatal growth ; Quantitative investigations
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The postnatal quantitative changes in cell diameter, cell density and total number of granule cells in the sublobule IXa of female rats from 6 to 760 days old were examined. There occurs and initial rapid increase in cell density from 1232.00±91.92 granule cells per 10-3 mm3 to 2995.50±322.07 granule cells per 10-3 mm3 and from 3 135 316±233 937 to more than 24 millions of granule cells between the 6th and 25th postnatal day. After the middle of the 3rd postnatal week, cell density and total number of granule cells decrease. The diameter of the granule cells reaches a maximum at the 6th day post partum and decreases continuously with progressing age. The possible mechanisms of these quantitative changes are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00345658
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  • 10
    Electronic Resource
    Electronic Resource
    Regional differences in the distribution of lipofuscin in Purkinje cell perikarya (1981)
    Springer
    Anatomy and embryology 161 (1981), S. 453-464 
    Details
    ISSN: 1432-0568
    Keywords: Lipofuscin ; Purkinje cells ; Pigmentarchitectonics ; Senile rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The distribution of lipofuscin in the perikarya of Purkinje cells of vermal and hemispheric lobules has been determined quantitatively in 7 rats, 30–38 months old, by the point-counting method. On the basis of morphologically and statistically significant differences a pigmentarchitectonics of the cerebellar cortex is established. The Purkinje cells of lobule VIa (Larsell 1952) are extremely lipofuscin-rich. The Purkinje cells of the hemispheres, lobules V, VI b+c and VII contain considerable amounts of a finely granular lipofuscin, the Purkinje cells of lobules I–III and VIII–IX a a globular type of lipofuscin. The Purkinje cells of sublobule XI d c and X are lipofuscin-poor cells. Three types of lipofuscin have been identified in the light microscope.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00316054
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